Page last updated: 2024-08-24

goralatide and Cirrhosis

goralatide has been researched along with Cirrhosis in 37 studies

Research

Studies (37)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	16 (43.24)	29.6817
2010's	15 (40.54)	24.3611
2020's	6 (16.22)	2.80

Authors

Authors	Studies
Athari, SS; Cai, J; Chen, Q; Mehrabi Nasab, E	1
Jia, W; Wang, W; Zhang, C	1
Deng, HJ; Gong, HB; Gong, RB; Miao, ZY; Tang, XY; Zhang, CM	1
Kanasaki, K	1
Gao, X; Ge, X; Jin, F; Li, S; Li, Y; Mao, N; Wei, Z; Xu, H; Yang, F; Yang, Y; Zhang, M; Zhang, Y	1
Gao, R; Hu, Q; Kanasaki, K; Kitada, M; Koya, D; Li, J; Li, S; Liu, H; Srivastava, SP; Wu, G	1
Carretero, OA; Kumar, N; Liao, TD; Maheshwari, M; Monu, SR; Peterson, EL; Romero, CA	1
Attwood, KM; Dahal, S; Nguyen, J; Pokharel, S; Sexton, S; Sharma, UC; Singh, AK; Sonkawade, SD; Spernyak, JA; van Berlo, JH	1
Carretero, OA; Kumar, N; Liao, TD; Nakagawa, P; Peterson, EL; Romero, CA; Worou, ME	1
Brolin, TJ; Carretero, OA; Chun, B; Fogo, AB; Kazi, N; Kon, V; Ma, LJ; Myöhänen, T; Orhan, D; Potthoff, SA; Rhaleb, NE; Yang, HC; Zuo, Y	1
Bhardwaj, N; Danilov, SM; Douglas, RG; Garcia, JG; Hogarth, KD; Nesterovitch, AB; Popova, IA; Schwager, SL; Schwartz, DE; Sturrock, ED; Wade, MS	1
Ishigaki, Y; Kanasaki, K; Kanasaki, M; Kitada, M; Koya, D; Nagai, T; Nakamura, Y; Shi, S; Srivastava, SP	1
Kanasaki, K; Kanasaki, M; Koya, D; Nagai, T; Nitta, K	1
Chan, GC; Huang, XR; Lan, HY; Lin, M; Tang, SC; Wong, DW; Wu, HJ; Yiu, WH	1
Borkum, MS; Engel, ME; Mayosi, BM; Mnguni, AT	1
He, J; Ishigaki, Y; Kanasaki, K; Kanasaki, M; Kitada, M; Koya, D; Nagai, T; Nakamura, Y; Shi, S; Srivastava, SP	1
Chan, GCW; Chan, KW; Huang, XR; Lai, KN; Lan, HY; Tang, SCW; Wu, HJ; Yiu, WH; Zou, A	1
Basha, H; Carretero, OA; Janic, B; Liao, TD; Masjoan-Juncos, JX; Nakagawa, P; Peterson, EL; Romero, CA; Worou, ME	1
Carretero, OA; D'Ambrosio, MA; Liao, TD; Lin, CX; Rhaleb, NE; Yang, XP	1
André, S; Carretero, OA; D'Ambrosio, M; Gabius, HJ; Liao, TD; Liu, YH; Peng, H; Rhaleb, NE; Sharma, U	1
Fogo, AB; Ma, LJ	1
Bombardi, C; Castoldi, G; Corradi, B; di Gioia, CR; Leopizzi, M; Mancini, M; Perego, C; Perego, L; Perlini, S; Stella, A; Zerbini, G	1
Feng, H; Li, Q; Sun, Y; Wang, R; Wei, Z; Yan, J; Yang, F; Zhang, L; Zhang, X	1
Jia, X; Liu, R; Mu, S; Wang, M; Xie, R	1
Merali, S; Sherwi, N; Wong, K	1
Haneda, M; Isono, M; Kanasaki, K; Kashiwagi, A; Koya, D; Sugimoto, T	1
Brigstock, DR; Carretero, OA; Oja-Tebbe, N; Peng, H; Rhaleb, NE	1
Carretero, OA; Cingolani, O; Liu, YH; Rhaleb, NE; Xu, J; Yang, F; Yang, XP	1
Carretero, OA; Cingolani, OH; Liu, YH; Rhaleb, NE; Villanueva, M; Yang, XP	1
Brigstock, DR; Carretero, OA; Cavasin, MA; Peng, H; Rasoul, S; Rhaleb, NE; Sanchez-Mendoza, A; Zhuo, J	1
Bohnemeier, H; Cleutjens, JP; Crijns, HJ; Paul, M; Pinto, YM; Pokharel, S; Schunkert, H; Sharma, UC; Tian, XL; van Geel, PP	1
Haneda, M; Inomata, N; Kanasaki, K; Kato, Y; Kojima, R; Koya, D; Omata, M; Sho, R; Taniguchi, H	1
Carretero, OA; Liao, TD; Peng, H; Peterson, EL; Rhaleb, NE	1
Carretero, OA; Cavasin, MA; Liao, TD; Yang, JJ; Yang, XP	1
Carretero, OA; Harding, P; Peng, H; Pokharel, S; Rasoul, S; Rhaleb, NE; Sharma, U	1
Ito, K; Iwamoto, N; Muraki, T; Nitta, K; Shiraga, H; Xano, HJ; Yoshioka, T	1
Carretero, OA; Harding, P; LaPointe, MC; Peng, H; Rhaleb, NE; Tayeh, M	1

Reviews

7 review(s) available for goralatide and Cirrhosis

Article	Year
The Role of Tβ4-POP-Ac-SDKP Axis in Organ Fibrosis. International journal of molecular sciences, 2022, Oct-31, Volume: 23, Issue:21 Topics: Fibrosis; Humans; Oligopeptides; Prolyl Oligopeptidases; Thymosin	2022
[Meta-analysis of Ac-SDKP inhibition of Pulmonary fibrosis in animal models]. Zhonghua lao dong wei sheng zhi ye bing za zhi = Zhonghua laodong weisheng zhiyebing zazhi = Chinese journal of industrial hygiene and occupational diseases, 2023, Apr-20, Volume: 41, Issue:4 Topics: Animals; Disease Models, Animal; Fibrosis; Proline; Pulmonary Fibrosis; Rats; Rats, Wistar	2023
N-acetyl-seryl-aspartyl-lysyl-proline is a valuable endogenous antifibrotic peptide for kidney fibrosis in diabetes: An update and translational aspects. Journal of diabetes investigation, 2020, Volume: 11, Issue:3 Topics: Animals; Diabetes Complications; Fibrosis; Humans; Kidney; Kidney Diseases; Oligopeptides; Peptidyl-Dipeptidase A; Signal Transduction	2020
The biological significance of angiotensin-converting enzyme inhibition to combat kidney fibrosis. Clinical and experimental nephrology, 2015, Volume: 19, Issue:1 Topics: Angiotensin-Converting Enzyme Inhibitors; Fibrosis; Humans; Kidney Diseases; Oligopeptides	2015
The Effects of Angiotensin Converting Enzyme Inhibitors (ACE-I) on Human N-Acetyl-Seryl-Aspartyl-Lysyl-Proline (Ac-SDKP) Levels: A Systematic Review and Meta-Analysis. PloS one, 2015, Volume: 10, Issue:12 Topics: Angiotensin-Converting Enzyme Inhibitors; Fibrosis; Humans; Myocardium; Oligopeptides; Patient Selection; Pericarditis, Tuberculous; Prospective Studies; Renal Insufficiency	2015
PAI-1 and kidney fibrosis. Frontiers in bioscience (Landmark edition), 2009, 01-01, Volume: 14, Issue:6 Topics: Angiotensins; Animals; Chronic Disease; Disease Models, Animal; Fibrosis; Humans; Kidney Diseases; Mice; Oligopeptides; Organ Specificity; Plasminogen Activator Inhibitor 1; Renin-Angiotensin System; Thymosin; Transforming Growth Factor beta1	2009
Personalizing biomarker strategies in heart failure with galectin-3. Future cardiology, 2012, Volume: 8, Issue:6 Topics: Biomarkers; Fibrosis; Galectin 3; Heart Failure; Humans; Oligopeptides; Prognosis; Translational Research, Biomedical; Ventricular Remodeling	2012

Other Studies

30 other study(ies) available for goralatide and Cirrhosis

Article	Year
Immunomodulatory effect of N-acetyl-seryl-aspartyl-proline and vasoactive intestinal peptide on chronic obstructive pulmonary disease pathophysiology. Fundamental & clinical pharmacology, 2022, Volume: 36, Issue:6 Topics: Animals; Anti-Inflammatory Agents; Cytokines; Fibrosis; Inflammation; Mice; Oligopeptides; Pulmonary Disease, Chronic Obstructive; Superoxide Dismutase; Vasoactive Intestinal Peptide	2022
N-Acetyl-Seryl-Asparyl-Lysyl-Proline regulates lung renin angiotensin system to inhibit epithelial-mesenchymal transition in silicotic mice. Toxicology and applied pharmacology, 2020, 12-01, Volume: 408 Topics: Angiotensin I; Angiotensin II; Angiotensin II Type 1 Receptor Blockers; Angiotensin-Converting Enzyme 2; Angiotensin-Converting Enzyme Inhibitors; Animals; Captopril; Cell Line; Epithelial-Mesenchymal Transition; Fibrosis; Losartan; Lung; Male; Mice, Inbred C57BL; Oligopeptides; Peptide Fragments; Peptidyl-Dipeptidase A; Renin-Angiotensin System; Silicosis	2020
Endothelial FGFR1 (Fibroblast Growth Factor Receptor 1) Deficiency Contributes Differential Fibrogenic Effects in Kidney and Heart of Diabetic Mice. Hypertension (Dallas, Tex. : 1979), 2020, Volume: 76, Issue:6 Topics: Animals; Cell Line; Diabetes Mellitus, Experimental; Endothelium; Epithelial Cells; Epithelial-Mesenchymal Transition; Fibrosis; Humans; Kidney; Kidney Tubules, Proximal; Mice, Inbred C57BL; Mice, Knockout; Myocardium; Oligopeptides; Receptor, Fibroblast Growth Factor, Type 1	2020
Renal Protective Effects of N-Acetyl-Seryl-Aspartyl-Lysyl-Proline (Ac-SDKP) in Obese Rats on a High-Salt Diet. American journal of hypertension, 2018, 07-16, Volume: 31, Issue:8 Topics: Albuminuria; Animals; Anti-Inflammatory Agents; Antihypertensive Agents; Blood Pressure; Disease Models, Animal; Fibrosis; Glomerulonephritis; Hypertension; Kidney; Macrophages; Male; Obesity; Oligopeptides; Rats, Zucker; Sodium Chloride, Dietary	2018
A Small Peptide Ac-SDKP Inhibits Radiation-Induced Cardiomyopathy. Circulation. Heart failure, 2018, Volume: 11, Issue:8 Topics: Animals; Apoptosis; Cardiomyopathies; Cardiotoxicity; Collagen Type I; Collagen Type III; Disease Models, Animal; Extracellular Matrix; Female; Fibrosis; Galectin 3; Macrophages; Magnetic Resonance Imaging; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Myocytes, Cardiac; Oligopeptides; Radiation Injuries; Radiation-Protective Agents; Rats, Sprague-Dawley; RAW 264.7 Cells; Transforming Growth Factor beta1; Ventricular Function, Left	2018
Renal release of N-acetyl-seryl-aspartyl-lysyl-proline is part of an antifibrotic peptidergic system in the kidney. American journal of physiology. Renal physiology, 2019, 01-01, Volume: 316, Issue:1 Topics: Animals; Disease Models, Animal; Fibrosis; Kidney Diseases; Kidney Medulla; Male; Metalloendopeptidases; Nephrons; Oligopeptides; Prolyl Oligopeptidases; Rats, Sprague-Dawley; Serine Endopeptidases; Signal Transduction; Thymosin	2019
Thymosin β4 and its degradation product, Ac-SDKP, are novel reparative factors in renal fibrosis. Kidney international, 2013, Volume: 84, Issue:6 Topics: Animals; Collagen; Disease Models, Animal; Fibronectins; Fibrosis; Kidney; Kidney Diseases; Macrophages; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Myofibroblasts; Oligopeptides; Plasminogen Activator Inhibitor 1; Prolyl Oligopeptidases; Serine Endopeptidases; Serine Proteinase Inhibitors; Thymosin; Time Factors; Ureteral Obstruction; Urological Agents	2013
A novel angiotensin I-converting enzyme mutation (S333W) impairs N-domain enzymatic cleavage of the anti-fibrotic peptide, AcSDKP. PloS one, 2014, Volume: 9, Issue:2 Topics: Amino Acid Sequence; Animals; Cell Line; CHO Cells; Cricetulus; Fibrosis; Humans; Kinetics; Molecular Sequence Data; Mutation; Oligopeptides; Peptides; Peptidyl-Dipeptidase A; Sequence Alignment	2014
N-acetyl-seryl-aspartyl-lysyl-proline inhibits diabetes-associated kidney fibrosis and endothelial-mesenchymal transition. BioMed research international, 2014, Volume: 2014 Topics: Animals; Cytokines; Diabetes Mellitus, Experimental; Diabetic Nephropathies; Epithelial-Mesenchymal Transition; Fibrosis; Growth Inhibitors; Male; Mice; MicroRNAs; Oligopeptides; Receptors, Fibroblast Growth Factor	2014
N-Acetyl-seryl-aspartyl-lysyl-proline Alleviates Renal Fibrosis Induced by Unilateral Ureteric Obstruction in BALB/C Mice. Mediators of inflammation, 2015, Volume: 2015 Topics: Actins; Angiotensin-Converting Enzyme Inhibitors; Animals; Captopril; Chemokine CCL2; Collagen Type IV; Fibrosis; Immunohistochemistry; Inflammation; Kidney Diseases; Lymphocytes; Macrophages; Male; Mice; Mice, Inbred BALB C; Muscle, Smooth; Oligopeptides; Real-Time Polymerase Chain Reaction; Ureteral Obstruction	2015
Effect of Antifibrotic MicroRNAs Crosstalk on the Action of N-acetyl-seryl-aspartyl-lysyl-proline in Diabetes-related Kidney Fibrosis. Scientific reports, 2016, 07-18, Volume: 6 Topics: Animals; Diabetes Mellitus; Dipeptidyl Peptidase 4; Dipeptidyl-Peptidase IV Inhibitors; Endothelial Cells; Fibroblast Growth Factor 1; Fibrosis; Humans; Interferon-gamma; Kidney; Mice; MicroRNAs; Microvessels; Models, Biological; Oligopeptides; Phosphorylation; Signal Transduction; Transforming Growth Factor beta; Up-Regulation	2016
N-acetyl-seryl-aspartyl-lysyl-proline mediates the anti-fibrotic properties of captopril in unilateral ureteric obstructed BALB/C mice. Nephrology (Carlton, Vic.), 2018, Volume: 23, Issue:4 Topics: Angiotensin-Converting Enzyme Inhibitors; Animals; Captopril; Disease Models, Animal; Extracellular Matrix Proteins; Fibrosis; Indoles; Kidney; Kidney Diseases; Male; Mice, Inbred BALB C; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Oligopeptides; Peptidyl-Dipeptidase A; Prolyl Oligopeptidases; Serine Endopeptidases; Serine Proteinase Inhibitors; Signal Transduction; Thiazolidines; Transforming Growth Factor beta1; Ureteral Obstruction	2018
Effects of N-acetyl-seryl-asparyl-lysyl-proline on blood pressure, renal damage, and mortality in systemic lupus erythematosus. Physiological reports, 2017, Volume: 5, Issue:2 Topics: Albuminuria; Animals; Blood Pressure; Female; Fibrosis; Hypertension; Kidney; Kidney Diseases; Lupus Erythematosus, Systemic; Mice; Oligopeptides; Survival Analysis; T-Lymphocytes	2017
Prevention of aortic fibrosis by N-acetyl-seryl-aspartyl-lysyl-proline in angiotensin II-induced hypertension. American journal of physiology. Heart and circulatory physiology, 2008, Volume: 295, Issue:3 Topics: Angiotensin II; Animals; Aorta; Collagen; Elastin; Enzyme Activation; Fibrosis; Heart Diseases; Hypertension; Immunohistochemistry; Intercellular Adhesion Molecule-1; Lipid Metabolism; Male; Neutrophil Infiltration; Oligopeptides; Oxidation-Reduction; Phosphorylation; Protein Kinase C; Rats; Rats, Sprague-Dawley; Smad2 Protein; Transforming Growth Factor beta1; Vasoconstrictor Agents	2008
N-acetyl-seryl-aspartyl-lysyl-proline prevents cardiac remodeling and dysfunction induced by galectin-3, a mammalian adhesion/growth-regulatory lectin. American journal of physiology. Heart and circulatory physiology, 2009, Volume: 296, Issue:2 Topics: Animals; Anti-Inflammatory Agents; Blood Pressure; Body Weight; Cardiac Output; Cardiomegaly; Cardiotonic Agents; Collagen; Disease Models, Animal; Echocardiography, Doppler; Fibrosis; Galectin 3; Heart Rate; Hemodynamics; Inflammation; Infusions, Parenteral; Isoproterenol; Macrophages; Male; Mast Cells; Myocardial Contraction; Myocardium; Oligopeptides; Phosphorylation; Rats; Rats, Sprague-Dawley; Signal Transduction; Smad3 Protein; Stroke Volume; Time Factors; Transforming Growth Factor beta; Ventricular Function, Left; Ventricular Remodeling	2009
Prevention of myocardial fibrosis by N-acetyl-seryl-aspartyl-lysyl-proline in diabetic rats. Clinical science (London, England : 1979), 2009, Oct-26, Volume: 118, Issue:3 Topics: Angiotensin-Converting Enzyme Inhibitors; Animals; Cardiomyopathies; Diabetes Complications; Diabetes Mellitus, Experimental; Fibrosis; Heart Ventricles; Hypoglycemic Agents; Insulin; Male; Myocardium; Oligopeptides; Ramipril; Rats; Rats, Sprague-Dawley; Smad Proteins; Transforming Growth Factor beta1	2009
New anti-fibrotic mechanisms of n-acetyl-seryl-aspartyl-lysyl-proline in silicon dioxide-induced silicosis. Life sciences, 2010, Aug-14, Volume: 87, Issue:7-8 Topics: Animals; Cell Migration Inhibition; Cell Proliferation; Chemokine CCL2; Collagen; Fibroblasts; Fibrosis; Gene Expression Regulation; JNK Mitogen-Activated Protein Kinases; Lung; Macrophages; Male; Oligopeptides; Protein Transport; Rats; Rats, Wistar; Silicon Dioxide; Silicosis; Transforming Growth Factor beta1	2010
N-acetyl-seryl-aspartyl-lysyl-proline attenuates renal inflammation and tubulointerstitial fibrosis in rats. International journal of molecular medicine, 2010, Volume: 26, Issue:6 Topics: Animals; Biomarkers; Disease Models, Animal; Fibrosis; Histocytochemistry; Intercellular Signaling Peptides and Proteins; Kidney Tubules; Macrophages; Male; Nephritis; Oligopeptides; Rats; Rats, Wistar	2010
N-Acetyl-seryl-aspartyl-lysyl-proline inhibits TGF-beta-mediated plasminogen activator inhibitor-1 expression via inhibition of Smad pathway in human mesangial cells. Journal of the American Society of Nephrology : JASN, 2003, Volume: 14, Issue:4 Topics: Cell Culture Techniques; DNA-Binding Proteins; Fibrosis; Glomerular Mesangium; Growth Inhibitors; Humans; Oligopeptides; Plasminogen Activator Inhibitor 1; RNA, Messenger; Signal Transduction; Smad Proteins; Smad7 Protein; Trans-Activators; Transforming Growth Factor beta	2003
Ac-SDKP reverses cardiac fibrosis in rats with renovascular hypertension. Hypertension (Dallas, Tex. : 1979), 2003, Volume: 42, Issue:6 Topics: Angiotensins; Animals; Blood Pressure; Body Weight; Bradykinin; Collagen; Connective Tissue Growth Factor; Fibrosis; Heart Ventricles; Hypertension, Renovascular; Immediate-Early Proteins; Intercellular Signaling Peptides and Proteins; Male; Myocardium; Oligopeptides; Organ Size; Rats; Rats, Sprague-Dawley; Transforming Growth Factor beta	2003
Ac-SDKP reverses inflammation and fibrosis in rats with heart failure after myocardial infarction. Hypertension (Dallas, Tex. : 1979), 2004, Volume: 43, Issue:2 Topics: Animals; Anti-Inflammatory Agents; Blood Pressure; Cardiac Output, Low; Cell Movement; Collagen; Fibrosis; Heart; Heart Rate; Inflammation; Macrophages; Male; Myocardial Infarction; Myocardium; Oligopeptides; Rats; Rats, Inbred Lew; Transforming Growth Factor beta	2004
Reduction of cardiac fibrosis decreases systolic performance without affecting diastolic function in hypertensive rats. Hypertension (Dallas, Tex. : 1979), 2004, Volume: 43, Issue:5 Topics: Animals; Capillaries; Cell Size; Collagen; Diastole; Fibrosis; Heart Ventricles; Hydroxyproline; Hypertension; Hypertrophy, Left Ventricular; Myocardium; Oligopeptides; Pressure; Rats; Rats, Inbred SHR; Rats, Inbred WKY; Systole	2004
Antifibrotic effect of Ac-SDKP and angiotensin-converting enzyme inhibition in hypertension. Journal of hypertension, 2004, Volume: 22, Issue:3 Topics: Angiotensin-Converting Enzyme Inhibitors; Animals; Blood Pressure; Captopril; Cell Division; Collagen; Connective Tissue Growth Factor; Drug Therapy, Combination; Fibrosis; Growth Inhibitors; Heart Rate; Hypertension; Hypertrophy, Left Ventricular; Immediate-Early Proteins; Intercellular Signaling Peptides and Proteins; Kidney; Macrophages; Male; Mast Cells; Monocytes; Myocardium; Myocytes, Cardiac; Oligopeptides; Rats; Rats, Sprague-Dawley; Transforming Growth Factor beta	2004
Increased myocardial collagen content in transgenic rats overexpressing cardiac angiotensin-converting enzyme is related to enhanced breakdown of N-acetyl-Ser-Asp-Lys-Pro and increased phosphorylation of Smad2/3. Circulation, 2004, Nov-09, Volume: 110, Issue:19 Topics: Activin Receptors, Type I; Angiotensin II; Animals; Animals, Genetically Modified; Binding, Competitive; Collagen; DNA-Binding Proteins; DNA, Complementary; Fibrosis; Humans; Hypertrophy, Left Ventricular; Myocardium; Oligopeptides; Peptidyl-Dipeptidase A; Phosphorylation; Protein Processing, Post-Translational; Protein Serine-Threonine Kinases; Rats; Rats, Sprague-Dawley; Receptor, Transforming Growth Factor-beta Type I; Receptors, Transforming Growth Factor beta; Smad2 Protein; Smad3 Protein; Substrate Specificity; Trans-Activators; Transforming Growth Factor beta; Transforming Growth Factor beta1	2004
N-acetyl-seryl-aspartyl-lysyl-proline ameliorates the progression of renal dysfunction and fibrosis in WKY rats with established anti-glomerular basement membrane nephritis. Journal of the American Society of Nephrology : JASN, 2006, Volume: 17, Issue:3 Topics: Analysis of Variance; Animals; Anti-Glomerular Basement Membrane Disease; Biopsy, Needle; Blood Chemical Analysis; Blotting, Western; Disease Models, Animal; Disease Progression; Dose-Response Relationship, Drug; Drug Administration Schedule; Fibrosis; Immunohistochemistry; Injections, Subcutaneous; Kidney Function Tests; Male; Oligopeptides; Probability; Random Allocation; Rats; Rats, Inbred WKY; Reverse Transcriptase Polymerase Chain Reaction; Sensitivity and Specificity; Statistics, Nonparametric; Transforming Growth Factor beta; Urinalysis	2006
Role of N-acetyl-seryl-aspartyl-lysyl-proline in the antifibrotic and anti-inflammatory effects of the angiotensin-converting enzyme inhibitor captopril in hypertension. Hypertension (Dallas, Tex. : 1979), 2007, Volume: 49, Issue:3 Topics: Angiotensin-Converting Enzyme Inhibitors; Animals; Antibodies, Monoclonal; Captopril; Disease Models, Animal; Enzyme Activation; Fibrosis; Heart; Hypertension; Inflammation; Kidney; Male; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Oligopeptides; Rats	2007
Decreased endogenous levels of Ac-SDKP promote organ fibrosis. Hypertension (Dallas, Tex. : 1979), 2007, Volume: 50, Issue:1 Topics: Angiotensin II; Animals; Arginine Vasopressin; Collagen; Fibrosis; Glomerular Mesangium; Glomerulosclerosis, Focal Segmental; Kidney; Male; Myocardium; Oligopeptides; Prolyl Oligopeptidases; Protease Inhibitors; Rats; Rats, Sprague-Dawley; Serine Endopeptidases; Substance P; Time Factors	2007
Novel anti-inflammatory mechanisms of N-Acetyl-Ser-Asp-Lys-Pro in hypertension-induced target organ damage. American journal of physiology. Heart and circulatory physiology, 2008, Volume: 294, Issue:3 Topics: Angiotensin II; Animals; Anti-Inflammatory Agents; Azo Compounds; Blotting, Western; Cardiomegaly; Cell Separation; Collagen; Fibrosis; Galectin 3; Heart Rate; Hypertension; In Vitro Techniques; Inflammation; Kidney; Male; Mice; Mice, Inbred C57BL; Myocardium; Neutrophil Infiltration; Oligopeptides; Stem Cells; Tumor Necrosis Factor-alpha	2008
Acetyl-seryl-aspartyl-lysyl-proline is a novel natural cell cycle regulator of renal cells. Life sciences, 2000, Mar-03, Volume: 66, Issue:15 Topics: Animals; Cell Cycle; Cell Division; Cell Line; Fibroblasts; Fibrosis; Flow Cytometry; Growth Inhibitors; Kidney; Kidney Tubules; L-Lactate Dehydrogenase; LLC-PK1 Cells; Oligopeptides; Swine	2000
Effect of N-acetyl-seryl-aspartyl-lysyl-proline on DNA and collagen synthesis in rat cardiac fibroblasts. Hypertension (Dallas, Tex. : 1979), 2001, Volume: 37, Issue:3 Topics: Angiotensin II; Angiotensin Receptor Antagonists; Angiotensin-Converting Enzyme Inhibitors; Animals; Cell Division; Cells, Cultured; Collagen; DNA; Dose-Response Relationship, Drug; Endothelin-1; Enzyme Activation; Enzyme Inhibitors; Fibrosis; Gene Expression Regulation; Humans; Hypertension; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinases; Myocardium; Oligopeptides; Peptidyl-Dipeptidase A; Rats; Rats, Sprague-Dawley; Receptor, Angiotensin, Type 1; Receptor, Angiotensin, Type 2; Receptors, Angiotensin; Renin-Angiotensin System	2001