Compounds > goralatide
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goralatide and Cardiac Hypertrophy

goralatide has been researched along with Cardiac Hypertrophy in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (66.67)29.6817
2010's1 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Carretero, OA; Dai, X; González, GE; Leung, P; Liao, TD; Liu, Y; Nakagawa, P; Rhaleb, NE; Yang, XP1
André, S; Carretero, OA; D'Ambrosio, M; Gabius, HJ; Liao, TD; Liu, YH; Peng, H; Rhaleb, NE; Sharma, U1
Carretero, OA; Harding, P; Peng, H; Pokharel, S; Rasoul, S; Rhaleb, NE; Sharma, U1

Other Studies

3 other study(ies) available for goralatide and Cardiac Hypertrophy

ArticleYear
N-acetyl-seryl-aspartyl-lysyl-proline reduces cardiac collagen cross-linking and inflammation in angiotensin II-induced hypertensive rats.
    Clinical science (London, England : 1979), 2014, Jan-01, Volume: 126, Issue:1

    Topics: Amino Acid Oxidoreductases; Angiotensin II; Animals; Body Weight; Cardiomegaly; CD4-Positive T-Lymphocytes; Collagen; Extracellular Matrix; Hypertension; Inflammation; Lung; Male; NF-kappa B; Oligopeptides; Organ Size; Protein-Lysine 6-Oxidase; Rats; Rats, Inbred Lew; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Ventricular Function, Left; Ventricular Remodeling

2014
N-acetyl-seryl-aspartyl-lysyl-proline prevents cardiac remodeling and dysfunction induced by galectin-3, a mammalian adhesion/growth-regulatory lectin.
    American journal of physiology. Heart and circulatory physiology, 2009, Volume: 296, Issue:2

    Topics: Animals; Anti-Inflammatory Agents; Blood Pressure; Body Weight; Cardiac Output; Cardiomegaly; Cardiotonic Agents; Collagen; Disease Models, Animal; Echocardiography, Doppler; Fibrosis; Galectin 3; Heart Rate; Hemodynamics; Inflammation; Infusions, Parenteral; Isoproterenol; Macrophages; Male; Mast Cells; Myocardial Contraction; Myocardium; Oligopeptides; Phosphorylation; Rats; Rats, Sprague-Dawley; Signal Transduction; Smad3 Protein; Stroke Volume; Time Factors; Transforming Growth Factor beta; Ventricular Function, Left; Ventricular Remodeling

2009
Novel anti-inflammatory mechanisms of N-Acetyl-Ser-Asp-Lys-Pro in hypertension-induced target organ damage.
    American journal of physiology. Heart and circulatory physiology, 2008, Volume: 294, Issue:3

    Topics: Angiotensin II; Animals; Anti-Inflammatory Agents; Azo Compounds; Blotting, Western; Cardiomegaly; Cell Separation; Collagen; Fibrosis; Galectin 3; Heart Rate; Hypertension; In Vitro Techniques; Inflammation; Kidney; Male; Mice; Mice, Inbred C57BL; Myocardium; Neutrophil Infiltration; Oligopeptides; Stem Cells; Tumor Necrosis Factor-alpha

2008