glyoxylic acid has been researched along with Chagas Disease in 1 studies
glyoxylic acid: RN given refers to parent cpd
glyoxylic acid : A 2-oxo monocarboxylic acid that is acetic acid bearing an oxo group at the alpha carbon atom.
Chagas Disease: Infection with the protozoan parasite TRYPANOSOMA CRUZI, a form of TRYPANOSOMIASIS endemic in Central and South America. It is named after the Brazilian physician Carlos Chagas, who discovered the parasite. Infection by the parasite (positive serologic result only) is distinguished from the clinical manifestations that develop years later, such as destruction of PARASYMPATHETIC GANGLIA; CHAGAS CARDIOMYOPATHY; and dysfunction of the ESOPHAGUS or COLON.
Excerpt | Relevance | Reference |
---|---|---|
"The acute phase of the experimental Chagas' disease in rats induces extensive lesion of the heart sympathetic nerve terminals." | 1.29 | Trypanosoma cruzi infection in rats induced early lesion of the heart noradrenergic nerve terminals by a complement-independent mechanism. ( Carvalho, EM; de Oliveira, DA; Machado, CR; Magalhaes, MJ; Ramalho-Pinto, FJ, 1994) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (100.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Machado, CR | 1 |
de Oliveira, DA | 1 |
Magalhaes, MJ | 1 |
Carvalho, EM | 1 |
Ramalho-Pinto, FJ | 1 |
1 other study available for glyoxylic acid and Chagas Disease
Article | Year |
---|---|
Trypanosoma cruzi infection in rats induced early lesion of the heart noradrenergic nerve terminals by a complement-independent mechanism.
Topics: Animals; Chagas Disease; Complement System Proteins; Elapid Venoms; Erythrocytes; Female; Fluorescen | 1994 |