Page last updated: 2024-08-21

glycyrrhizic acid and Hyperplasia

glycyrrhizic acid has been researched along with Hyperplasia in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's3 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Afnan, Q; Bhardwaj, SC; Bhushan, S; Kaiser, PJ; Nazir, LA; Rafiq, RA; Sandhir, R; Tasduq, SA1
Hisatsune, A; Isohama, Y; Katsuki, H; Miyata, T; Nishimoto, Y; Yokomizo, K1
Chen, J; Chen, S; Jiang, H; Xu, C; Xu, L; Yang, J; Zhang, J1

Other Studies

3 other study(ies) available for glycyrrhizic acid and Hyperplasia

ArticleYear
Glycyrrhizic acid prevents ultraviolet-B-induced photodamage: a role for mitogen-activated protein kinases, nuclear factor kappa B and mitochondrial apoptotic pathway.
    Experimental dermatology, 2016, Volume: 25, Issue:6

    Topics: Animals; Anti-Inflammatory Agents; Apoptosis; Cell Line; Dermatitis; Drug Evaluation, Preclinical; Glycyrrhizic Acid; Humans; Hyperplasia; Methionine; Mice, Inbred BALB C; Mitogen-Activated Protein Kinases; NF-kappa B; Reactive Oxygen Species; Skin; Skin Aging; Sulfoxides; Ultraviolet Rays

2016
Glycyrrhizin attenuates mucus production by inhibition of MUC5AC mRNA expression in vivo and in vitro.
    Journal of pharmacological sciences, 2010, Volume: 113, Issue:1

    Topics: Animals; Cell Line; Disease Models, Animal; Dose-Response Relationship, Drug; Gene Expression Regulation; Glycyrrhizic Acid; Goblet Cells; Hyperplasia; Interleukin-4; Lipopolysaccharides; Lung Diseases, Obstructive; Male; Mice; Mice, Inbred ICR; Mucin 5AC; Mucus; Promoter Regions, Genetic; Transforming Growth Factor alpha

2010
Inhibition of neointimal hyperplasia in the rat carotid artery injury model by a HMGB1 inhibitor.
    Atherosclerosis, 2012, Volume: 224, Issue:2

    Topics: Angiotensin II; Animals; Anti-Inflammatory Agents; Antioxidants; Cardiovascular Agents; Carotid Arteries; Carotid Artery Injuries; Cell Differentiation; Cell Movement; Cell Proliferation; Cells, Cultured; Disease Models, Animal; Dose-Response Relationship, Drug; Glycyrrhizic Acid; HMGB1 Protein; Hyperplasia; Inflammation Mediators; Male; Muscle, Smooth, Vascular; Myocytes, Smooth Muscle; Neointima; NF-kappa B; Oxidative Stress; p38 Mitogen-Activated Protein Kinases; Phenotype; Rats; Rats, Sprague-Dawley; Receptors, Notch; Signal Transduction; Time Factors

2012