glycyrrhizic acid has been researched along with Anoxemia in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 2 (50.00) | 2.80 |
Authors | Studies |
---|---|
Bian, H; Xia, M; Xu, Y; Yang, X | 1 |
Ali, AI; Chibaatar, E; Guo, Y; Le, K; Wu, S | 1 |
Cai, J; Chen, AF; Chen, D; Huang, Y; Jiang, R; Tao, M; Teng, Y; Wang, J; Wang, Q; Xia, A; Xue, M; Zhou, S; Zhu, Z | 1 |
Bhagat, S; Biswas, I; Khan, GA; Singh, B; Surya Kumari, S | 1 |
1 review(s) available for glycyrrhizic acid and Anoxemia
Article | Year |
---|---|
Metabolic dysfunction associated fatty liver disease and coronavirus disease 2019: clinical relationship and current management.
Topics: Age Factors; Angiotensin-Converting Enzyme 2; Anti-Inflammatory Agents; Chemical and Drug Induced Liver Injury; COVID-19; COVID-19 Drug Treatment; Cytokines; Dipeptides; Gene Expression Regulation; Glucose; Glycyrrhizic Acid; Humans; Hypoxia; Liver; Lung; Non-alcoholic Fatty Liver Disease; Receptors, Virus; SARS-CoV-2; Severity of Illness Index | 2021 |
3 other study(ies) available for glycyrrhizic acid and Anoxemia
Article | Year |
---|---|
Alarmin HMGB1 Plays a Detrimental Role in Hippocampal Dysfunction Caused by Hypoxia-Ischemia Insult in Neonatal Mice: Evidence from the Application of the HMGB1 Inhibitor Glycyrrhizin.
Topics: Alarmins; Animals; Animals, Newborn; Glycyrrhizic Acid; Hippocampus; HMGB1 Protein; Hypoxia; Ischemia; Mice | 2020 |
Hypoxic trophoblast HMGB1 induces endothelial cell hyperpermeability via the TRL-4/caveolin-1 pathway.
Topics: Animals; Caveolin 1; Cell Line; Cell Membrane Permeability; Culture Media, Conditioned; Female; Gene Expression Regulation; Glycyrrhizic Acid; HMGB1 Protein; Human Umbilical Vein Endothelial Cells; Humans; Hypoxia; Mice; Mice, Inbred C57BL; Mice, Transgenic; Pregnancy; RNA, Small Interfering; Signal Transduction; Toll-Like Receptor 4; Trophoblasts | 2014 |
HMGB1 facilitates hypoxia-induced vWF upregulation through TLR2-MYD88-SP1 pathway.
Topics: Animals; Antibodies, Blocking; Cells, Cultured; Diabetes Mellitus, Type 2; Glycyrrhizic Acid; HMGB1 Protein; Humans; Hypoxia; Immunoglobulins; Insulin Resistance; Mice; Molecular Targeted Therapy; Myeloid Differentiation Factor 88; RNA, Small Interfering; Signal Transduction; Toll-Like Receptor 2; Up-Regulation; von Willebrand Factor | 2016 |