glycolaldehyde has been researched along with Inflammation in 6 studies
Inflammation: A pathological process characterized by injury or destruction of tissues caused by a variety of cytologic and chemical reactions. It is usually manifested by typical signs of pain, heat, redness, swelling, and loss of function.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Glycolaldehyde (GA) has been shown to impair cellular function in various disorders, including diabetes, and renal diseases." | 5.72 | Glycolaldehyde induces synergistic effects on vascular inflammation in TNF-α-stimulated vascular smooth muscle cells. ( Choi, IW; Gu, MJ; Ha, SK; Kim, Y; Lee, HW; Lee, SH; Yoo, G, 2022) |
| "Glycolaldehyde was enzymically formed from glyceraldehyde, the fructokinase/aldolase B product of fructose." | 5.35 | Hepatocyte inflammation model for cytotoxicity research: fructose or glycolaldehyde as a source of endogenous toxins. ( Bruce, J; Bruce, WR; Dong, Q; Feng, CY; Mehta, R; O'Brien, PJ; Wong, S, 2009) |
| " We have recently proposed another mechanism for aldehyde generation at sites of inflammation that involves myeloperoxidase, a heme enzyme secreted by activated phagocytes." | 3.69 | Human neutrophils employ the myeloperoxidase-hydrogen peroxide-chloride system to convert hydroxy-amino acids into glycolaldehyde, 2-hydroxypropanal, and acrolein. A mechanism for the generation of highly reactive alpha-hydroxy and alpha,beta-unsaturated ( Anderson, MM; Hazen, SL; Heinecke, JW; Hsu, FF, 1997) |
| "Glycolaldehyde (GA) has been shown to impair cellular function in various disorders, including diabetes, and renal diseases." | 1.72 | Glycolaldehyde induces synergistic effects on vascular inflammation in TNF-α-stimulated vascular smooth muscle cells. ( Choi, IW; Gu, MJ; Ha, SK; Kim, Y; Lee, HW; Lee, SH; Yoo, G, 2022) |
| "Glycolaldehyde was enzymically formed from glyceraldehyde, the fructokinase/aldolase B product of fructose." | 1.35 | Hepatocyte inflammation model for cytotoxicity research: fructose or glycolaldehyde as a source of endogenous toxins. ( Bruce, J; Bruce, WR; Dong, Q; Feng, CY; Mehta, R; O'Brien, PJ; Wong, S, 2009) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (16.67) | 18.2507 |
| 2000's | 2 (33.33) | 29.6817 |
| 2010's | 1 (16.67) | 24.3611 |
| 2020's | 2 (33.33) | 2.80 |
| Authors | Studies |
|---|---|
| Lee, HW | 1 |
| Gu, MJ | 1 |
| Yoo, G | 1 |
| Choi, IW | 1 |
| Lee, SH | 1 |
| Kim, Y | 1 |
| Ha, SK | 1 |
| Gomes Kjerulf, D | 1 |
| Wang, S | 1 |
| Omer, M | 1 |
| Pathak, A | 1 |
| Subramanian, S | 1 |
| Han, CY | 1 |
| Tang, C | 1 |
| den Hartigh, LJ | 1 |
| Shao, B | 1 |
| Chait, A | 1 |
| Feng, CY | 1 |
| Wong, S | 1 |
| Dong, Q | 1 |
| Bruce, J | 1 |
| Mehta, R | 1 |
| Bruce, WR | 1 |
| O'Brien, PJ | 1 |
| Buetler, TM | 1 |
| Latado, H | 1 |
| Leclerc, E | 1 |
| Weigle, B | 1 |
| Baumeyer, A | 1 |
| Heizmann, CW | 1 |
| Scholz, G | 1 |
| Anderson, MM | 2 |
| Heinecke, JW | 2 |
| Hazen, SL | 1 |
| Hsu, FF | 1 |
6 other studies available for glycolaldehyde and Inflammation
| Article | Year |
|---|---|
Glycolaldehyde induces synergistic effects on vascular inflammation in TNF-α-stimulated vascular smooth muscle cells.
Topics: Acetaldehyde; Animals; Cell Adhesion Molecules; Inflammation; Intercellular Adhesion Molecule-1; Mic | 2022 |
Glycation of HDL blunts its anti-inflammatory and cholesterol efflux capacities in vitro, but has no effect in poorly controlled type 1 diabetes subjects.
Topics: Acetaldehyde; Apolipoprotein A-I; Apolipoprotein A-II; Cholesterol; Diabetes Mellitus, Type 1; Gluco | 2020 |
Hepatocyte inflammation model for cytotoxicity research: fructose or glycolaldehyde as a source of endogenous toxins.
Topics: Acetaldehyde; Animals; Antioxidants; Dose-Response Relationship, Drug; Fructose; Glyoxal; Hepatocyte | 2009 |
Glycolaldehyde-modified β-lactoglobulin AGEs are unable to stimulate inflammatory signaling pathways in RAGE-expressing human cell lines.
Topics: Acetaldehyde; Cell Line; Cytokines; Detergents; Endotoxins; Glycation End Products, Advanced; Humans | 2011 |
Production of N(epsilon)-(carboxymethyl)lysine is impaired in mice deficient in NADPH oxidase: a role for phagocyte-derived oxidants in the formation of advanced glycation end products during inflammation.
Topics: Acetaldehyde; Animals; Glycation End Products, Advanced; Hydrogen Peroxide; Inflammation; Lysine; Me | 2003 |
Human neutrophils employ the myeloperoxidase-hydrogen peroxide-chloride system to convert hydroxy-amino acids into glycolaldehyde, 2-hydroxypropanal, and acrolein. A mechanism for the generation of highly reactive alpha-hydroxy and alpha,beta-unsaturated
Topics: Acetaldehyde; Acrolein; Aldehydes; Amino Acids; Catalase; Chlorides; Chromatography, High Pressure L | 1997 |