glycogen and Muscular-Dystrophy--Duchenne

glycogen has been researched along with Muscular-Dystrophy--Duchenne* in 2 studies

Other Studies

2 other study(ies) available for glycogen and Muscular-Dystrophy--Duchenne

Article	Year
Elevated GLUT4 and glycogenin protein abundance correspond to increased glycogen content in the soleus muscle of mdx mice with no benefit associated with taurine supplementation. Physiological reports, 2018, Volume: 6, Issue:5 Topics: Animals; Glucose Transporter Type 4; Glucosyltransferases; Glycogen; Glycoproteins; Male; Mice; Mice, Inbred C57BL; Mice, Inbred mdx; Muscle Fibers, Skeletal; Muscular Dystrophy, Duchenne; Taurine	2018
A pilot study of muscle plasma protein changes after exercise. Muscle & nerve, 2014, Volume: 49, Issue:2 Creatine kinase (CK) and myoglobin (Mb) do not possess all good qualities as biomarkers of skeletal muscle damage. We investigated the utility of troponin I (TnI) and telethonin (Tcap) as markers and examined their temporal profiles after skeletal muscle damage.. Plasma profiles were measured before and after exercise in 3 groups: subjects affected by either Becker muscular dystrophy or McArdle disease, and healthy subjects.. Mb and TnI appeared early in the blood, and the increase of TnI was only observed in patients with muscle disease. The CK increase was more delayed in plasma. Tcap was not detectable at any time.. Our results suggest that TnI is a marker of more severe damage signifying sarcomeric damage, and it could therefore be an important supplement to CK and Mb in clinical practice. Tcap is not useful as a marker for skeletal muscle damage. Topics: Adolescent; Adult; Biomarkers; Case-Control Studies; Connectin; Creatine Kinase; Energy Metabolism; Exercise; Female; Glycogen; Glycogen Storage Disease Type V; Humans; Male; Middle Aged; Muscle Fibers, Skeletal; Muscle, Skeletal; Muscular Dystrophy, Duchenne; Pilot Projects; Sarcomeres; Troponin I; Young Adult	2014

Article

Year

Elevated GLUT4 and glycogenin protein abundance correspond to increased glycogen content in the soleus muscle of mdx mice with no benefit associated with taurine supplementation.

Physiological reports, 2018, Volume: 6, Issue:5

Topics: Animals; Glucose Transporter Type 4; Glucosyltransferases; Glycogen; Glycoproteins; Male; Mice; Mice, Inbred C57BL; Mice, Inbred mdx; Muscle Fibers, Skeletal; Muscular Dystrophy, Duchenne; Taurine

2018

A pilot study of muscle plasma protein changes after exercise.

Muscle & nerve, 2014, Volume: 49, Issue:2

Creatine kinase (CK) and myoglobin (Mb) do not possess all good qualities as biomarkers of skeletal muscle damage. We investigated the utility of troponin I (TnI) and telethonin (Tcap) as markers and examined their temporal profiles after skeletal muscle damage.. Plasma profiles were measured before and after exercise in 3 groups: subjects affected by either Becker muscular dystrophy or McArdle disease, and healthy subjects.. Mb and TnI appeared early in the blood, and the increase of TnI was only observed in patients with muscle disease. The CK increase was more delayed in plasma. Tcap was not detectable at any time.. Our results suggest that TnI is a marker of more severe damage signifying sarcomeric damage, and it could therefore be an important supplement to CK and Mb in clinical practice. Tcap is not useful as a marker for skeletal muscle damage.

Topics: Adolescent; Adult; Biomarkers; Case-Control Studies; Connectin; Creatine Kinase; Energy Metabolism; Exercise; Female; Glycogen; Glycogen Storage Disease Type V; Humans; Male; Middle Aged; Muscle Fibers, Skeletal; Muscle, Skeletal; Muscular Dystrophy, Duchenne; Pilot Projects; Sarcomeres; Troponin I; Young Adult

2014