glycine has been researched along with Left Ventricular Hypertrophy in 6 studies
| Excerpt | Relevance | Reference |
|---|---|---|
| "Association of polymorphic markers G7831A of ACE gene, Lys198Asn of endothelin-1 (EDN1) gene, and 4a/4b of NOS3 gene with characteristics of structure and function of the left ventricle was studied in 70 (31 men and 39 women) natives of Yakutia with hypertension." | 7.73 | [Angiotensin converting enzyme, NO-synthase, and endothelin-1 genes and left ventricular hypertrophy in natives of Yakutia with hypertensive disease]. ( Antipina, VV; Makarova, ER; Minushkina, LO; Nosikov, VV; Petrova, IR; Romanova, TA; Sidorenko, BA; Zateĭshikov, DA, 2005) |
| " These results suggest that sepiapterin prevents concentric LVH and dilatory remodeling after TAC primarily by increasing the bioavailability of NO." | 5.42 | Sepiapterin prevents left ventricular hypertrophy and dilatory remodeling induced by pressure overload in rats. ( Fujita, M; Iwasaka, T; Otani, H; Shimazu, T; Shiojima, I; Yoshioka, K, 2015) |
| "Association of polymorphic markers G7831A of ACE gene, Lys198Asn of endothelin-1 (EDN1) gene, and 4a/4b of NOS3 gene with characteristics of structure and function of the left ventricle was studied in 70 (31 men and 39 women) natives of Yakutia with hypertension." | 3.73 | [Angiotensin converting enzyme, NO-synthase, and endothelin-1 genes and left ventricular hypertrophy in natives of Yakutia with hypertensive disease]. ( Antipina, VV; Makarova, ER; Minushkina, LO; Nosikov, VV; Petrova, IR; Romanova, TA; Sidorenko, BA; Zateĭshikov, DA, 2005) |
| " These results suggest that sepiapterin prevents concentric LVH and dilatory remodeling after TAC primarily by increasing the bioavailability of NO." | 1.42 | Sepiapterin prevents left ventricular hypertrophy and dilatory remodeling induced by pressure overload in rats. ( Fujita, M; Iwasaka, T; Otani, H; Shimazu, T; Shiojima, I; Yoshioka, K, 2015) |
| "Fabry disease is an X-linked lysosomal storage disorder caused by mutations of the α-galactosidase A gene (GLA), and the disease is a relatively prevalent cause of left ventricular hypertrophy mimicking idiopathic hypertrophic cardiomyopathy." | 1.37 | Clinical and genetic investigation of a Japanese family with cardiac fabry disease. Identification of a novel α-galactosidase A missense mutation (G195V). ( Hasebe, N; Ishihara, T; Kawabe, J; Kikuchi, K; Kobayashi, M; Maruyama, H; Nakagawa, N; Ota, H; Sakamoto, N; Sasaki, Y; Seino, U; Takahashi, F; Takenaka, T; Takeuchi, T; Tanabe, Y; Yamauchi, A, 2011) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (33.33) | 29.6817 |
| 2010's | 4 (66.67) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Cauley, E | 1 |
| Wang, X | 1 |
| Dyavanapalli, J | 1 |
| Sun, K | 2 |
| Garrott, K | 1 |
| Kuzmiak-Glancy, S | 1 |
| Kay, MW | 1 |
| Mendelowitz, D | 1 |
| Yoshioka, K | 1 |
| Otani, H | 1 |
| Shimazu, T | 1 |
| Fujita, M | 1 |
| Iwasaka, T | 1 |
| Shiojima, I | 1 |
| Nakagawa, N | 1 |
| Maruyama, H | 1 |
| Ishihara, T | 1 |
| Seino, U | 1 |
| Kawabe, J | 1 |
| Takahashi, F | 1 |
| Kobayashi, M | 1 |
| Yamauchi, A | 1 |
| Sasaki, Y | 1 |
| Sakamoto, N | 1 |
| Ota, H | 1 |
| Tanabe, Y | 1 |
| Takeuchi, T | 1 |
| Takenaka, T | 1 |
| Kikuchi, K | 1 |
| Hasebe, N | 1 |
| Fantinelli, JC | 1 |
| González Arbeláez, LF | 1 |
| Pérez Núñez, IA | 1 |
| Mosca, SM | 1 |
| Minushkina, LO | 1 |
| Petrova, IR | 1 |
| Romanova, TA | 1 |
| Antipina, VV | 1 |
| Makarova, ER | 1 |
| Zateĭshikov, DA | 1 |
| Nosikov, VV | 1 |
| Sidorenko, BA | 1 |
| Fu, C | 1 |
| Wang, H | 1 |
| Wang, S | 1 |
| Shi, Y | 1 |
| Zhou, X | 1 |
| Chen, J | 1 |
| Song, X | 1 |
| Xue, H | 1 |
| Hui, R | 1 |
6 other studies available for glycine and Left Ventricular Hypertrophy
| Article | Year |
|---|---|
Neurotransmission to parasympathetic cardiac vagal neurons in the brain stem is altered with left ventricular hypertrophy-induced heart failure.
Topics: Animals; Brain Stem; Disease Models, Animal; Excitatory Postsynaptic Potentials; GABAergic Neurons; | 2015 |
Sepiapterin prevents left ventricular hypertrophy and dilatory remodeling induced by pressure overload in rats.
Topics: Animals; Aorta; Biopterins; Capillaries; Cell Size; Constriction; Dilatation, Pathologic; Enzyme Inh | 2015 |
Clinical and genetic investigation of a Japanese family with cardiac fabry disease. Identification of a novel α-galactosidase A missense mutation (G195V).
Topics: Adult; alpha-Galactosidase; Amino Acid Substitution; Bundle-Branch Block; Coronary Angiography; Coro | 2011 |
Protective effects of N-(2-mercaptopropionyl)-glycine against ischemia-reperfusion injury in hypertrophied hearts.
Topics: Animals; Blood Pressure; Cardiotonic Agents; Glutathione; Glycine; Hypertension; Hypertrophy, Left V | 2013 |
[Angiotensin converting enzyme, NO-synthase, and endothelin-1 genes and left ventricular hypertrophy in natives of Yakutia with hypertensive disease].
Topics: Adult; Alanine; Asparagine; Blood Flow Velocity; Endothelin-1; Female; Gene Frequency; Genotype; Gly | 2005 |
Association of beta 1-adrenergic receptor gene polymorphisms with left ventricular hypertrophy in human essential hypertension.
Topics: Aged; Amino Acid Substitution; Arginine; China; Cross-Sectional Studies; Female; Genotype; Glycine; | 2008 |