glycine has been researched along with Glucose Intolerance in 11 studies
Glucose Intolerance: A pathological state in which BLOOD GLUCOSE level is less than approximately 140 mg/100 ml of PLASMA at fasting, and above approximately 200 mg/100 ml plasma at 30-, 60-, or 90-minute during a GLUCOSE TOLERANCE TEST. This condition is seen frequently in DIABETES MELLITUS, but also occurs with other diseases and MALNUTRITION.
| Excerpt | Relevance | Reference |
|---|---|---|
| " In addition, the plasma level of glycine is decreased in obesity, and the decrease is strongly correlated with the severity of insulin resistance." | 8.12 | Glycine Supplementation in Obesity Worsens Glucose Intolerance through Enhanced Liver Gluconeogenesis. ( Alves, A; Bassot, A; Bloise, AMNLG; Chanon, S; Drevet Mulard, E; Godet, M; Lamarche, F; Lefebvre, R; Loizon, E; Morio, B; Panthu, B; Pinteur, C; Rautureau, GJP, 2022) |
| " In addition, the plasma level of glycine is decreased in obesity, and the decrease is strongly correlated with the severity of insulin resistance." | 4.12 | Glycine Supplementation in Obesity Worsens Glucose Intolerance through Enhanced Liver Gluconeogenesis. ( Alves, A; Bassot, A; Bloise, AMNLG; Chanon, S; Drevet Mulard, E; Godet, M; Lamarche, F; Lefebvre, R; Loizon, E; Morio, B; Panthu, B; Pinteur, C; Rautureau, GJP, 2022) |
| " Since we demonstrated that glycine supplementation can reduce inflammation and muscle wasting, we hypothesized that glycine supplementation during CR would preserve muscle mass in mice." | 3.83 | Glycine supplementation during calorie restriction accelerates fat loss and protects against further muscle loss in obese mice. ( Caldow, MK; Chee, A; Godeassi, DP; Ham, DJ; Koopman, R; Lynch, GS, 2016) |
| "Insulin resistance was evaluated with fasting insulin, fasting glucose/insulin ratio, and homeostasis model assessment index for insulin resistance (HOMAIR)." | 1.33 | Abnormal glucose tolerance and insulin resistance in polycystic ovary syndrome amongst the Taiwanese population- not correlated with insulin receptor substrate-1 Gly972Arg/Ala513Pro polymorphism. ( Gong, KB; Ku, DC; Kuo, TC; Liang, SF; Lin, TC; Wu, MJ; Yen, JM, 2006) |
| " We conclude that SNPs in PPARD modify the conversion from IGT to type 2 diabetes, particularly in combination with the SNPs of PGC-1A and PPARG2." | 1.33 | Single nucleotide polymorphisms of PPARD in combination with the Gly482Ser substitution of PGC-1A and the Pro12Ala substitution of PPARG2 predict the conversion from impaired glucose tolerance to type 2 diabetes: the STOP-NIDDM trial. ( Andrulionyte, L; Chiasson, JL; Laakso, M; Peltola, P, 2006) |
| "None of the NIDDM or impaired glucose tolerant patients had this polymorphism." | 1.29 | Lack of association between the Gly40Ser polymorphism in the glucagon receptor gene and NIDDM in Finland. ( Groop, L; Huang, X; Lehto, M; Orho, M, 1995) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 3 (27.27) | 18.2507 |
| 2000's | 5 (45.45) | 29.6817 |
| 2010's | 2 (18.18) | 24.3611 |
| 2020's | 1 (9.09) | 2.80 |
| Authors | Studies |
|---|---|
| Alves, A | 1 |
| Lamarche, F | 1 |
| Lefebvre, R | 1 |
| Drevet Mulard, E | 1 |
| Bassot, A | 1 |
| Chanon, S | 1 |
| Loizon, E | 1 |
| Pinteur, C | 1 |
| Bloise, AMNLG | 1 |
| Godet, M | 1 |
| Rautureau, GJP | 1 |
| Panthu, B | 1 |
| Morio, B | 1 |
| Caldow, MK | 1 |
| Ham, DJ | 1 |
| Godeassi, DP | 1 |
| Chee, A | 1 |
| Lynch, GS | 1 |
| Koopman, R | 1 |
| Ogawa, W | 1 |
| Iwamoto, K | 1 |
| Mori, H | 1 |
| Hashiramoto, M | 1 |
| Miyake, K | 1 |
| Sakaguchi, K | 1 |
| Kasuga, M | 1 |
| Keung, W | 1 |
| Ussher, JR | 1 |
| Jaswal, JS | 1 |
| Raubenheimer, M | 1 |
| Lam, VH | 1 |
| Wagg, CS | 1 |
| Lopaschuk, GD | 1 |
| Barbato, A | 1 |
| Russo, P | 1 |
| Venezia, A | 1 |
| Strazzullo, V | 1 |
| Siani, A | 1 |
| Cappuccio, FP | 1 |
| Fanelli, M | 1 |
| Filippi, E | 1 |
| Sentinelli, F | 1 |
| Romeo, S | 1 |
| Fallarino, M | 1 |
| Buzzetti, R | 1 |
| Leonetti, F | 1 |
| Baroni, MG | 1 |
| Lin, TC | 1 |
| Yen, JM | 1 |
| Gong, KB | 1 |
| Kuo, TC | 1 |
| Ku, DC | 1 |
| Liang, SF | 1 |
| Wu, MJ | 1 |
| Andrulionyte, L | 1 |
| Peltola, P | 1 |
| Chiasson, JL | 1 |
| Laakso, M | 1 |
| Huang, X | 1 |
| Orho, M | 1 |
| Lehto, M | 1 |
| Groop, L | 1 |
| Chuang, LM | 1 |
| Lai, CS | 1 |
| Yeh, JI | 1 |
| Wu, HP | 1 |
| Tai, TY | 1 |
| Lin, BJ | 1 |
| Odawara, M | 1 |
| Tachi, Y | 1 |
| Yamashita, K | 1 |
11 other studies available for glycine and Glucose Intolerance
| Article | Year |
|---|---|
Glycine Supplementation in Obesity Worsens Glucose Intolerance through Enhanced Liver Gluconeogenesis.
Topics: Animals; Diet, High-Fat; Dietary Supplements; Gluconeogenesis; Glucose Intolerance; Glycine; Insulin | 2022 |
Glycine supplementation during calorie restriction accelerates fat loss and protects against further muscle loss in obese mice.
Topics: Animals; Body Composition; Body Mass Index; Caloric Restriction; Cytokines; Diet, High-Fat; Dietary | 2016 |
Two related cases of type A insulin resistance with compound heterozygous mutations of the insulin receptor gene.
Topics: Adolescent; Amino Acid Substitution; Aspartic Acid; Female; Genetic Carrier Screening; Genetic Varia | 2009 |
Inhibition of carnitine palmitoyltransferase-1 activity alleviates insulin resistance in diet-induced obese mice.
Topics: Animals; Anti-Obesity Agents; Carbohydrate Metabolism; Carnitine O-Palmitoyltransferase; Cell Membra | 2013 |
Analysis of Gly40Ser polymorphism of the glucagon receptor (GCGR) gene in different ethnic groups.
Topics: Adult; Asian People; Black People; Blood Glucose; Cardiovascular Diseases; Diabetes Mellitus; Female | 2003 |
The Gly482Ser missense mutation of the peroxisome proliferator-activated receptor gamma coactivator-1 alpha (PGC-1 alpha) gene associates with reduced insulin sensitivity in normal and glucose-intolerant obese subjects.
Topics: Adult; Amino Acid Substitution; Female; Genetic Predisposition to Disease; Glucose Intolerance; Glyc | 2005 |
Abnormal glucose tolerance and insulin resistance in polycystic ovary syndrome amongst the Taiwanese population- not correlated with insulin receptor substrate-1 Gly972Arg/Ala513Pro polymorphism.
Topics: Adolescent; Adult; Alanine; Amino Acid Substitution; Arginine; Diabetes Mellitus; Female; Glucose In | 2006 |
Single nucleotide polymorphisms of PPARD in combination with the Gly482Ser substitution of PGC-1A and the Pro12Ala substitution of PPARG2 predict the conversion from impaired glucose tolerance to type 2 diabetes: the STOP-NIDDM trial.
Topics: Amino Acid Substitution; Diabetes Mellitus, Type 2; Disease Progression; Female; Glucose Intolerance | 2006 |
Lack of association between the Gly40Ser polymorphism in the glucagon receptor gene and NIDDM in Finland.
Topics: Alleles; Amino Acid Sequence; Base Sequence; Diabetes Mellitus, Type 2; DNA Primers; Finland; France | 1995 |
No association between the Gly971Arg variant of the insulin receptor substrate 1 gene and NIDDM in the Taiwanese population.
Topics: Adult; Amino Acid Sequence; Arginine; Base Sequence; Child; Diabetes Mellitus, Type 2; DNA Primers; | 1996 |
Absence of association between the Gly40-->Ser mutation in the human glucagon receptor and Japanese patients with non-insulin-dependent diabetes mellitus or impaired glucose tolerance.
Topics: Adult; Aged; Asian People; Codon; Diabetes Mellitus, Type 2; Exons; Female; Glucose Intolerance; Gly | 1996 |