glycidyl nitrate has been researched along with Genetic Predisposition in 16 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (6.25) | 18.2507 |
| 2000's | 12 (75.00) | 29.6817 |
| 2010's | 2 (12.50) | 24.3611 |
| 2020's | 1 (6.25) | 2.80 |
| Authors | Studies |
|---|---|
| Chassaing, B | 1 |
| Bonnefond, A; Dhennin, V; Frade-Proud'Hon-Clerc, S; Frenois, F; Froguel, P; Fumery, M; Gower-Rousseau, C; Guillon-Dellac, N; Sand, O; Smol, T; Vaillant, E; Vasseur, F | 1 |
| Crielaard, W; Deng, DM; Rao, VA; ten Cate, JM; Urch, JE; van Aalten, DM | 1 |
| Balfour Sartor, R; Bleich, A; Hedrich, HJ; Hopf, S; Li, F; Mähler, M; van Lith, HA | 1 |
| Bleich, A; Buer, J; Bumann, D; de Buhr, MF; Goyert, SM; Hedrich, HJ; Hofmann, C; Obermeier, F; Westendorf, AM; Zschemisch, NH | 1 |
| Guo, YS; Shi, GY; Xiong, Y; Xu, WG; Xu, YP; Yang, L | 1 |
| Bonen, DK; Brant, SR; Chen, FF; Cho, JH; Duerr, RH; Foster, SJ; Inohara, N; Nicolae, DL; Nuñez, G; Ogura, Y; Saab, L; Tanabe, T | 1 |
| Crespo, J; Fernandez-Luna, JL; Fontalba, A; Foster, SJ; Fukase, K; Gutierrez, O; Hashimoto, M; Inamura, S; Inohara, N; Kusumoto, S; Moran, AP; Nuñez, G; Ogura, Y; Pons, F | 1 |
| Girardin, SE; Hugot, JP; Sansonetti, PJ | 1 |
| de Jong, DJ; Ferwerda, G; Girardin, SE; Kullberg, BJ; Netea, MG; van der Meer, JW | 1 |
| Arimura, A; Hikita, I; Hirasawa, T; Horikawa, T; Imura, K; Matsutani, T; Sakata, T; Yoshioka, T | 1 |
| Angus, DC; Ding, J; Ferrell, RE; Garcia, M; Harris, TB; Kellum, JA; Kritchevsky, SB; Li, R; Newman, AB; Wunderink, RG; Yaffe, K; Yende, S; Zmuda, J | 1 |
| de Jong, DJ; Drenth, JP; Ferwerda, G; Joosten, LA; Kullberg, BJ; Netea, MG; Van der Meer, JW | 1 |
| Benko, S; Girardin, SE; Le Bourhis, L | 1 |
| Chrousos, GP; Crofford, L; Karalis, K; Wilder, RL | 1 |
| Adam, A; Colman, RW; Isordia-Salas, I; Li, F; Pixley, RA; Sainz, I; Sartor, RB | 1 |
2 review(s) available for glycidyl nitrate and Genetic Predisposition
| Article | Year |
|---|---|
Lessons from Nod2 studies: towards a link between Crohn's disease and bacterial sensing.
Topics: Animals; Bacterial Infections; Carrier Proteins; Crohn Disease; Genetic Predisposition to Disease; Humans; Intracellular Signaling Peptides and Proteins; Nod2 Signaling Adaptor Protein; Peptidoglycan | 2003 |
Nod1 and Nod2 in innate immunity and human inflammatory disorders.
Topics: Bacteria; Crohn Disease; Genetic Predisposition to Disease; Humans; Immunity, Innate; Mutation; Nod1 Signaling Adaptor Protein; Nod2 Signaling Adaptor Protein; Peptidoglycan; Sarcoidosis; Signal Transduction; Syndrome | 2007 |
14 other study(ies) available for glycidyl nitrate and Genetic Predisposition
| Article | Year |
|---|---|
When NOD ligands become antidotes.
Topics: Antidotes; Crohn Disease; Genetic Predisposition to Disease; Humans; Inflammation; Ligands; Mutation; Nod2 Signaling Adaptor Protein; Peptidoglycan | 2022 |
A Novel Rare Missense Variation of the
Topics: Adolescent; Adult; Alleles; Child; Crohn Disease; Exome Sequencing; Female; Genetic Association Studies; Genetic Predisposition to Disease; Genotype; Heterozygote; Humans; Immunity, Innate; Male; Mutation; Mutation, Missense; Nod2 Signaling Adaptor Protein; Peptidoglycan; Polymorphism, Single Nucleotide; Protein Conformation | 2019 |
Streptococcus mutans SMU.623c codes for a functional, metal-dependent polysaccharide deacetylase that modulates interactions with salivary agglutinin.
Topics: Agglutinins; Amidohydrolases; Bacterial Proteins; Carbohydrate Sequence; Cell Adhesion; Chitin; Chromosomes, Bacterial; DNA, Bacterial; Gene Knockout Techniques; Genetic Predisposition to Disease; Metals; Muramidase; Oligosaccharides; Peptidoglycan; Polymerase Chain Reaction; Saliva; Streptococcus mutans | 2009 |
Genetic dissection of granulomatous enterocolitis and arthritis in the intramural peptidoglycan-polysaccharide-treated rat model of IBD.
Topics: Animals; Arthritis; Disease Models, Animal; Enterocolitis; Female; Genetic Linkage; Genetic Predisposition to Disease; Granulomatous Disease, Chronic; Inflammatory Bowel Diseases; Peptidoglycan; Quantitative Trait Loci; Rats; Rats, Inbred F344; Rats, Inbred Lew | 2009 |
Analysis of Cd14 as a genetic modifier of experimental inflammatory bowel disease (IBD) in mice.
Topics: Animals; Base Sequence; Cell Line; Female; Genetic Predisposition to Disease; Inflammatory Bowel Diseases; Interleukin-10; Lipopolysaccharide Receptors; Lipopolysaccharides; Male; Mice; Mice, Inbred C3H; Mice, Inbred C57BL; Molecular Sequence Data; Peptidoglycan; Polymorphism, Genetic; Quantitative Trait Loci | 2009 |
The effect of peptidoglycan stimulation on basophil-mediated atopic responses during pregnancy and in newborns.
Topics: Adult; Asthma; Basophil Degranulation Test; Basophils; Cells, Cultured; Cytokines; Dose-Response Relationship, Drug; Female; Fetal Blood; Genetic Predisposition to Disease; Histamine Release; Humans; Hypersensitivity; Immunity, Innate; Infant; Infant, Newborn; Interleukin-4; Ligands; Peptidoglycan; Postpartum Period; Pregnancy; Pregnancy Complications; RNA, Messenger; Toll-Like Receptor 2 | 2011 |
Crohn's disease-associated NOD2 variants share a signaling defect in response to lipopolysaccharide and peptidoglycan.
Topics: Alleles; Carrier Proteins; Case-Control Studies; Cell Line; Crohn Disease; Frameshift Mutation; Genetic Predisposition to Disease; Genetic Variation; Genotype; Heterozygote; Humans; Intracellular Signaling Peptides and Proteins; Jews; Lipopolysaccharides; Nod2 Signaling Adaptor Protein; Peptidoglycan; Signal Transduction | 2003 |
Host recognition of bacterial muramyl dipeptide mediated through NOD2. Implications for Crohn's disease.
Topics: Acetylmuramyl-Alanyl-Isoglutamine; Bacillus subtilis; Carrier Proteins; Cell Line; Crohn Disease; Escherichia coli; Genetic Predisposition to Disease; Humans; Intracellular Signaling Peptides and Proteins; Lipopolysaccharides; Mutagenesis, Site-Directed; NF-kappa B; Nod2 Signaling Adaptor Protein; Peptidoglycan; Recombinant Proteins; Substrate Specificity; Transfection | 2003 |
NOD2 3020insC mutation and the pathogenesis of Crohn's disease: impaired IL-1beta production points to a loss-of-function phenotype.
Topics: Acetylmuramyl-Alanyl-Isoglutamine; Alleles; Case-Control Studies; Crohn Disease; Gene Expression Regulation; Genetic Predisposition to Disease; Homozygote; Humans; Interleukin-1; Intracellular Signaling Peptides and Proteins; Leukocytes, Mononuclear; Lipopeptides; Mutation; Nod2 Signaling Adaptor Protein; Oligopeptides; Peptidoglycan; Polymerase Chain Reaction; Signal Transduction; Toll-Like Receptor 2 | 2005 |
Impact of T-cell receptor Vbeta haplotypes on the development of dermatitis in DS-Nh mice: synergistic production of interleukin-13 caused by staphylococcal enterotoxin C and peptide glycans from Staphylococcus aureus.
Topics: Animals; Cell Proliferation; Cells, Cultured; Collagen; Dermatitis, Atopic; Enterotoxins; Genetic Predisposition to Disease; Haplotypes; Interleukin-13; Killer Cells, Natural; Ligands; Lymphocyte Activation; Mice; Mice, Inbred Strains; Peptidoglycan; Polymerase Chain Reaction; Receptors, Antigen, T-Cell, alpha-beta; Spleen; Staphylococcus aureus; Superantigens; Toll-Like Receptor 2 | 2007 |
4G/5G plasminogen activator inhibitor-1 polymorphisms and haplotypes are associated with pneumonia.
Topics: Aged; Blood; Cohort Studies; Community-Acquired Infections; Cytokines; Female; Genetic Predisposition to Disease; Genotype; Haplotypes; Humans; Lipopolysaccharides; Longitudinal Studies; Male; Osmolar Concentration; Peptidoglycan; Phenotype; Plasminogen Activator Inhibitor 1; Pneumonia; Polymorphism, Genetic; Polymorphism, Single Nucleotide; White People | 2007 |
Crohn's disease patients homozygous for the 3020insC NOD2 mutation have a defective NOD2/TLR4 cross-tolerance to intestinal stimuli.
Topics: Bacteria; Cells, Cultured; Colon; Crohn Disease; Enterocytes; Genetic Predisposition to Disease; Humans; Immune Tolerance; Intestinal Mucosa; Mutation; Nod2 Signaling Adaptor Protein; Peptidoglycan; Toll-Like Receptor 4; Tumor Necrosis Factor-alpha | 2008 |
Glucocorticoid and/or glucocorticoid antagonist effects in inflammatory disease-susceptible Lewis rats and inflammatory disease-resistant Fischer rats.
Topics: Adrenal Glands; Animals; Arthritis; Carrageenan; Dexamethasone; Disease Susceptibility; Genetic Predisposition to Disease; Inflammation; Male; Mifepristone; Organ Size; Peptidoglycan; Polysaccharides, Bacterial; Rats; Rats, Inbred F344; Rats, Inbred Lew; Spleen; Streptococcus; Thymus Gland | 1995 |
Kininogen deficiency modulates chronic intestinal inflammation in genetically susceptible rats.
Topics: Animals; Blood Proteins; Chronic Disease; Enterocolitis; Genetic Predisposition to Disease; Kallikrein-Kinin System; Kininogen, High-Molecular-Weight; Metabolism, Inborn Errors; Peptidoglycan; Polymers; Polysaccharides; Rats; Rats, Inbred BN; Rats, Inbred Lew | 2002 |