Page last updated: 2024-09-03

glycerophosphoinositol 4,5-bisphosphate and Arrhythmia

glycerophosphoinositol 4,5-bisphosphate has been researched along with Arrhythmia in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (20.00)29.6817
2010's3 (60.00)24.3611
2020's1 (20.00)2.80

Authors

AuthorsStudies
Fujita, T; Hiraishi, K; Hu, Y; Inoue, R; Kurahara, LH; Li, Q; Shioi, N; Zhu, X1
An, H; Huang, Y; Pang, C; Ren, S; Xing, C; Zhan, Y1
Abderemane-Ali, F; Amarouch, MY; Baró, I; Brasseur, R; Coyan, FC; Loussouarn, G; Marionneau, C; Mérot, J; Mordel, J; Nicolas, CS; Piron, J; Steenman, M; Thomas, A1
Baró, I; Loussouarn, G1
Behr, E; Bendahhou, S; Cobo, AM; Donaldson, MR; Escolar, DM; Fu, YH; Jensen, JL; Mozaffar, T; Pereira, S; Poza, JJ; Ptácek, LJ; Suarez, WA; Szepetowski, P; Tawil, R; Tristani-Firouzi, M; Wagstaff, J1

Reviews

1 review(s) available for glycerophosphoinositol 4,5-bisphosphate and Arrhythmia

ArticleYear
Neural modulation of ion channels in cardiac arrhythmias: clinical implications and future investigations.
    Heart rhythm, 2010, Volume: 7, Issue:6

    Topics: Arrhythmias, Cardiac; Caveolin 3; Computer Simulation; Cyclic AMP; Death, Sudden, Cardiac; Humans; Induced Pluripotent Stem Cells; Ion Channels; Long QT Syndrome; Neurotransmitter Agents; Phosphatidylinositol 4,5-Diphosphate; Signal Transduction

2010

Other Studies

4 other study(ies) available for glycerophosphoinositol 4,5-bisphosphate and Arrhythmia

ArticleYear
An Arrhythmic Mutation E7K Facilitates TRPM4 Channel Activation via Enhanced PIP
    Cells, 2021, 04-22, Volume: 10, Issue:5

    Topics: Action Potentials; Arrhythmias, Cardiac; Gain of Function Mutation; HEK293 Cells; Humans; Phosphatidylinositol 4,5-Diphosphate; TRPM Cation Channels

2021
Hydrocinnamic Acid Inhibits the Currents of WT and SQT3 Syndrome-Related Mutants of Kir2.1 Channel.
    The Journal of membrane biology, 2017, Volume: 250, Issue:5

    Topics: Arrhythmias, Cardiac; Heart Conduction System; Heart Defects, Congenital; HEK293 Cells; Humans; Membrane Potentials; Molecular Docking Simulation; Mutation; Phenylpropionates; Phosphatidylinositol 4,5-Diphosphate; Potassium Channels, Inwardly Rectifying

2017
A long QT mutation substitutes cholesterol for phosphatidylinositol-4,5-bisphosphate in KCNQ1 channel regulation.
    PloS one, 2014, Volume: 9, Issue:3

    Topics: Animals; Arrhythmias, Cardiac; Brugada Syndrome; Cardiac Conduction System Disease; Cell Line; Chlorocebus aethiops; Cholesterol; COS Cells; Heart Conduction System; KCNQ1 Potassium Channel; Long QT Syndrome; Magnesium; Mutation; Phosphatidylinositol 4,5-Diphosphate

2014
PIP2 binding residues of Kir2.1 are common targets of mutations causing Andersen syndrome.
    Neurology, 2003, Jun-10, Volume: 60, Issue:11

    Topics: Abnormalities, Multiple; Arrhythmias, Cardiac; Binding Sites; Female; Genetic Predisposition to Disease; Humans; Male; Muscle Weakness; Mutation; Paralysis; Pedigree; Phenotype; Phosphatidylinositol 4,5-Diphosphate; Potassium Channels, Inwardly Rectifying; Syndrome

2003