glycerophosphoinositol 4,5-bisphosphate has been researched along with Arrhythmia in 5 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (20.00) | 29.6817 |
2010's | 3 (60.00) | 24.3611 |
2020's | 1 (20.00) | 2.80 |
Authors | Studies |
---|---|
Fujita, T; Hiraishi, K; Hu, Y; Inoue, R; Kurahara, LH; Li, Q; Shioi, N; Zhu, X | 1 |
An, H; Huang, Y; Pang, C; Ren, S; Xing, C; Zhan, Y | 1 |
Abderemane-Ali, F; Amarouch, MY; Baró, I; Brasseur, R; Coyan, FC; Loussouarn, G; Marionneau, C; Mérot, J; Mordel, J; Nicolas, CS; Piron, J; Steenman, M; Thomas, A | 1 |
Baró, I; Loussouarn, G | 1 |
Behr, E; Bendahhou, S; Cobo, AM; Donaldson, MR; Escolar, DM; Fu, YH; Jensen, JL; Mozaffar, T; Pereira, S; Poza, JJ; Ptácek, LJ; Suarez, WA; Szepetowski, P; Tawil, R; Tristani-Firouzi, M; Wagstaff, J | 1 |
1 review(s) available for glycerophosphoinositol 4,5-bisphosphate and Arrhythmia
Article | Year |
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Neural modulation of ion channels in cardiac arrhythmias: clinical implications and future investigations.
Topics: Arrhythmias, Cardiac; Caveolin 3; Computer Simulation; Cyclic AMP; Death, Sudden, Cardiac; Humans; Induced Pluripotent Stem Cells; Ion Channels; Long QT Syndrome; Neurotransmitter Agents; Phosphatidylinositol 4,5-Diphosphate; Signal Transduction | 2010 |
4 other study(ies) available for glycerophosphoinositol 4,5-bisphosphate and Arrhythmia
Article | Year |
---|---|
An Arrhythmic Mutation E7K Facilitates TRPM4 Channel Activation via Enhanced PIP
Topics: Action Potentials; Arrhythmias, Cardiac; Gain of Function Mutation; HEK293 Cells; Humans; Phosphatidylinositol 4,5-Diphosphate; TRPM Cation Channels | 2021 |
Hydrocinnamic Acid Inhibits the Currents of WT and SQT3 Syndrome-Related Mutants of Kir2.1 Channel.
Topics: Arrhythmias, Cardiac; Heart Conduction System; Heart Defects, Congenital; HEK293 Cells; Humans; Membrane Potentials; Molecular Docking Simulation; Mutation; Phenylpropionates; Phosphatidylinositol 4,5-Diphosphate; Potassium Channels, Inwardly Rectifying | 2017 |
A long QT mutation substitutes cholesterol for phosphatidylinositol-4,5-bisphosphate in KCNQ1 channel regulation.
Topics: Animals; Arrhythmias, Cardiac; Brugada Syndrome; Cardiac Conduction System Disease; Cell Line; Chlorocebus aethiops; Cholesterol; COS Cells; Heart Conduction System; KCNQ1 Potassium Channel; Long QT Syndrome; Magnesium; Mutation; Phosphatidylinositol 4,5-Diphosphate | 2014 |
PIP2 binding residues of Kir2.1 are common targets of mutations causing Andersen syndrome.
Topics: Abnormalities, Multiple; Arrhythmias, Cardiac; Binding Sites; Female; Genetic Predisposition to Disease; Humans; Male; Muscle Weakness; Mutation; Paralysis; Pedigree; Phenotype; Phosphatidylinositol 4,5-Diphosphate; Potassium Channels, Inwardly Rectifying; Syndrome | 2003 |