glutathione disulfide has been researched along with Aging, Premature in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (66.67) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Haramizu, S; Hase, T; Murase, T; Ota, N | 1 |
| Acuña-Castroviejo, D; Escames, G; García, JA; López, A; López, LC; Ortiz, F; Rodriguez, MI | 1 |
| Bing, G; Chae, JS; Jeong, JH; Jung, BD; Kim, HC; Kim, HJ; Kim, WK; Lee, SY; Park, ES; Shin, EJ; Wie, MB; Yen, TP | 1 |
3 other study(ies) available for glutathione disulfide and Aging, Premature
| Article | Year |
|---|---|
Catechins attenuate eccentric exercise-induced inflammation and loss of force production in muscle in senescence-accelerated mice.
Topics: Aging, Premature; Alanine Transaminase; Animals; Aspartate Aminotransferases; Calcium-Transporting ATPases; Catechin; Chemokine CCL2; Disease Models, Animal; Glutathione; Glutathione Disulfide; Inflammation; Interleukin-6; Male; Malondialdehyde; Mice; Muscle Contraction; Muscle, Skeletal; Oxidative Stress; Physical Endurance; Running; Sodium-Potassium-Exchanging ATPase; Tumor Necrosis Factor-alpha | 2011 |
Melatonin administration prevents cardiac and diaphragmatic mitochondrial oxidative damage in senescence-accelerated mice.
Topics: Aging, Premature; Animals; Antioxidants; Diaphragm; Glutathione; Glutathione Disulfide; Glutathione Peroxidase; Glutathione Reductase; Lipid Peroxidation; Male; Melatonin; Mice; Mice, Mutant Strains; Mitochondria; Mitochondria, Heart; Mitochondria, Muscle; Oxidation-Reduction; Oxidative Stress | 2007 |
Kainate-induced mitochondrial oxidative stress contributes to hippocampal degeneration in senescence-accelerated mice.
Topics: Aging, Premature; Animals; Caspase 3; Cytochromes c; Disease Models, Animal; Enzyme Activation; Glutathione; Glutathione Disulfide; Hippocampus; Ion Channels; Kainic Acid; Lipid Peroxidation; Lipofuscin; Male; Membrane Potential, Mitochondrial; Mice; Mice, Inbred Strains; Mitochondria; Mitochondrial Proteins; Nerve Degeneration; Neurons; Oxidation-Reduction; Oxidative Stress; Proteins; Proto-Oncogene Proteins c-fos; Seizures; Superoxide Dismutase; Time Factors; Uncoupling Protein 2 | 2008 |