glutamine has been researched along with Long QT Syndrome in 5 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 2 (40.00) | 18.2507 |
2000's | 2 (40.00) | 29.6817 |
2010's | 1 (20.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Aydin, M; Bilik, MZ; Ekinci, A; Ekinci, C; Ibiloglu, I; Ozaydogdu, N; Ulger, BV; Yazgan, UC; Yildiz, A; Yuksel, M | 1 |
Carmeliet, P; Fredj, S; Kass, RS; Lindegger, N; Sampson, KJ | 1 |
Amin, AS; Bhuiyan, ZA; Carvalho, JS; Ghamdi, SA; Gong, Q; Homfray, T; Mannens, MM; Momenah, TS; Wilde, AA; Zhou, Z | 1 |
Kitabatake, A; Makita, N; Matsuoka, R; Nagashima, M; Shirai, N; Tohse, N; Yamada, Y | 1 |
Furutani, M; Furutani, Y; Hino, Y; Imamura, S; Kamisago, M; Kimura, M; Kojima, Y; Matsuoka, R; Momma, K; Morikawa, Y; Takao, A; Yamagishi, H | 1 |
5 other study(ies) available for glutamine and Long QT Syndrome
Article | Year |
---|---|
The protective role of glutamine against acute induced toxicity in rats.
Topics: Animals; Antioxidants; Cardiotonic Agents; Edema; Electrocardiography; Glutamine; Heart Diseases; Insecticides; Long QT Syndrome; Male; Myocardium; Nitriles; Oxidative Stress; Pyrethrins; Rats; Rats, Wistar | 2015 |
Altered Na+ channels promote pause-induced spontaneous diastolic activity in long QT syndrome type 3 myocytes.
Topics: Animals; Anti-Arrhythmia Agents; Diastole; Electrophysiology; Flecainide; Gene Deletion; Genetic Variation; Glutamine; Heart; Long QT Syndrome; Lysine; Mice; Mice, Transgenic; Myocytes, Cardiac; Patch-Clamp Techniques; Proline; Sodium Channels | 2006 |
Recurrent intrauterine fetal loss due to near absence of HERG: clinical and functional characterization of a homozygous nonsense HERG Q1070X mutation.
Topics: Abortion, Spontaneous; Adult; Codon, Nonsense; Consanguinity; Death, Sudden, Cardiac; Ether-A-Go-Go Potassium Channels; Female; Fetal Death; Glutamine; Homozygote; Humans; Infant, Newborn; Long QT Syndrome; Male; Pedigree; Pilot Projects; Polymorphism, Genetic; Pregnancy; Pregnancy Trimester, Third; Recurrence; Risk Factors | 2008 |
A de novo missense mutation of human cardiac Na+ channel exhibiting novel molecular mechanisms of long QT syndrome.
Topics: Animals; Arginine; Electric Conductivity; Female; Glutamine; Humans; Infant; Long QT Syndrome; Mutagenesis, Site-Directed; Myocardium; NAV1.5 Voltage-Gated Sodium Channel; Patch-Clamp Techniques; Sodium; Sodium Channels; Xenopus | 1998 |
A de novo missense mutation (R1623Q) of the SCN5A gene in a Japanese girl with sporadic long QT sydrome. Mutations in brief no. 140. Online.
Topics: Amino Acid Substitution; Arginine; Female; Glutamine; Humans; Long QT Syndrome; Mutation, Missense; NAV1.5 Voltage-Gated Sodium Channel; Polymorphism, Single-Stranded Conformational; Sequence Deletion; Sodium Channels | 1998 |