Compounds > glutamic acid

glutamic acid and Growth Disorders

glutamic acid has been researched along with Growth Disorders in 7 studies

Research

Studies (7)

TimeframeStudies, this research(%)All Research%
pre-19901 (14.29)18.7374
1990's1 (14.29)18.2507
2000's2 (28.57)29.6817
2010's3 (42.86)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Bhattarai, JP; Cho, DH; Han, SK; Lee, JC; Nguyen, HT; Park, SJ1
Collman, RG; Gill, AJ; Kolson, DL; Kovacsics, CE; Vance, PJ1
Kiess, W; Klammt, J; Kratzsch, J; Kruis, T; Müller, E; Pfäffle, R; Schlicke, M; Schmidt, G; van de Leur, HS; Wallborn, T; Wüller, S1
Chian, D; Hornig, M; Lipkin, WI1
Breuning, MH; Kant, SG; Karperien, M; Kruithof, MF; Pereira, AM; Romijn, JA; van der Kamp, HJ; van Duyvenvoorde, HA; Walenkamp, MJ; Wit, JM1
Jacobsen, DW; Miyatake, K; Nakano, Y; Pezacka, EH; Saido, H; Toyoshima, S; Watanabe, F1
Kiseleva, EV1

Other Studies

7 other study(ies) available for glutamic acid and Growth Disorders

ArticleYear
Enhanced GABA action on the substantia gelatinosa neurons of the medullary dorsal horn in the offspring of streptozotocin-injected mice.
    Journal of diabetes and its complications, 2015, Volume: 29, Issue:5

    Topics: Animals; Diabetes Mellitus, Experimental; Diabetes, Gestational; Diabetic Neuropathies; Facial Nerve Diseases; Facial Pain; Female; Fetal Development; gamma-Aminobutyric Acid; Glutamic Acid; Glycine; Growth Disorders; Male; Mice, Inbred ICR; Pregnancy; Pregnancy in Diabetics; Streptozocin; Substantia Gelatinosa; Synaptic Transmission; Trigeminal Caudal Nucleus; Up-Regulation

2015
Induction of Heme Oxygenase-1 Deficiency and Associated Glutamate-Mediated Neurotoxicity Is a Highly Conserved HIV Phenotype of Chronic Macrophage Infection That Is Resistant to Antiretroviral Therapy.
    Journal of virology, 2015, Volume: 89, Issue:20

    Topics: Anemia, Hemolytic; Animals; Anti-HIV Agents; Drug Resistance, Viral; Gene Expression; Glutamic Acid; Growth Disorders; Heme Oxygenase-1; HIV-1; HIV-2; Human Immunodeficiency Virus Proteins; Humans; Iron Metabolism Disorders; Macrophage Activation; Macrophages; nef Gene Products, Human Immunodeficiency Virus; Neuroglia; Phenotype; Primary Cell Culture; Rats; Rats, Sprague-Dawley; Viral Regulatory and Accessory Proteins; Virus Replication; vpr Gene Products, Human Immunodeficiency Virus

2015
A heterozygous mutation of the insulin-like growth factor-I receptor causes retention of the nascent protein in the endoplasmic reticulum and results in intrauterine and postnatal growth retardation.
    The Journal of clinical endocrinology and metabolism, 2010, Volume: 95, Issue:5

    Topics: Amino Acid Substitution; Birth Weight; Body Size; Cognition Disorders; Endoplasmic Reticulum; Exons; Female; Fetal Growth Retardation; Glutamic Acid; Growth Disorders; Heterozygote; Humans; Infant, Newborn; Insulin-Like Growth Factor Binding Protein 3; Insulin-Like Growth Factor I; Intellectual Disability; Microcephaly; Mutation, Missense; Polymerase Chain Reaction; Receptor, IGF Type 1; Valine

2010
Neurotoxic effects of postnatal thimerosal are mouse strain dependent.
    Molecular psychiatry, 2004, Volume: 9, Issue:9

    Topics: Animals; Autistic Disorder; Autoimmune Diseases; Behavior, Animal; Exploratory Behavior; Female; Genetic Predisposition to Disease; Glutamic Acid; Growth Disorders; Hippocampus; Male; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Mice, Inbred Strains; Neurons; Neurotoxins; Preservatives, Pharmaceutical; Sex Factors; Species Specificity; Statistics, Nonparametric; Thimerosal; Vaccines

2004
A variable degree of intrauterine and postnatal growth retardation in a family with a missense mutation in the insulin-like growth factor I receptor.
    The Journal of clinical endocrinology and metabolism, 2006, Volume: 91, Issue:8

    Topics: Adult; Base Sequence; Body Height; Bone Density; DNA Mutational Analysis; DNA, Complementary; Failure to Thrive; Female; Fetal Growth Retardation; Fibroblasts; Glutamic Acid; Growth Disorders; Heterozygote; Humans; Infant; Insulin-Like Growth Factor I; Lysine; Microcephaly; Mutation, Missense; Phosphorylation; Polymerase Chain Reaction; Receptor, IGF Type 1; Sequence Analysis, DNA; Signal Transduction

2006
Accumulation of methylmalonic acid caused by vitamin B12-deficiency disrupts normal cellular metabolism in rat liver.
    The British journal of nutrition, 1996, Volume: 75, Issue:6

    Topics: Animals; Carbon; Cells, Cultured; Glucose; Glutamic Acid; Growth Disorders; Liver; Male; Methylmalonic Acid; Mitochondria, Liver; Rats; Rats, Wistar; Vitamin B 12 Deficiency

1996
[Functional growth retardation and its correction].
    Pediatriia, 1989, Issue:11

    Topics: Adolescent; Anabolic Agents; Arginine; Aspartic Acid; Child; Clomiphene; Drug Therapy, Combination; Female; Glutamates; Glutamic Acid; Growth Disorders; Humans; Male; Nandrolone; Nandrolone Decanoate

1989