Compounds > glutamic acid

glutamic acid and Developmental Psychomotor Disorders

glutamic acid has been researched along with Developmental Psychomotor Disorders in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (50.00)29.6817
2010's2 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Agostinho, P; Augusto, E; Boison, D; Chen, JF; Cunha, RA; Matos, M; Shen, HY; Wang, Y; Wang, YT; Wei, CJ1
Ao, Y; Cantle, JP; Lin, Q; Lu, XH; Mody, I; Olsen, RW; Sofroniew, MV; Sun, YE; Tao, J; Wei, W; Wu, H; Yang, XW1
Brinco, F; Vasques, V; Viegas, CM; Wajner, M1
Cauli, O; Erceg, S; Felipo, V; Llansola, M1

Other Studies

4 other study(ies) available for glutamic acid and Developmental Psychomotor Disorders

ArticleYear
Deletion of adenosine A2A receptors from astrocytes disrupts glutamate homeostasis leading to psychomotor and cognitive impairment: relevance to schizophrenia.
    Biological psychiatry, 2015, Dec-01, Volume: 78, Issue:11

    Topics: Animals; Astrocytes; Cognition Disorders; Disease Models, Animal; Dizocilpine Maleate; Excitatory Amino Acid Antagonists; Excitatory Amino Acid Transporter 2; Gene Expression Regulation; Glial Fibrillary Acidic Protein; Glutamic Acid; Homeostasis; Kainic Acid; Locomotion; Mice; Mice, Inbred C57BL; Mice, Transgenic; Psychomotor Disorders; Pyrimidines; Receptor, Adenosine A2A; Receptors, N-Methyl-D-Aspartate; Synaptosomes; Time Factors; Triazoles

2015
Deletion of astroglial Dicer causes non-cell-autonomous neuronal dysfunction and degeneration.
    The Journal of neuroscience : the official journal of the Society for Neuroscience, 2011, Jun-01, Volume: 31, Issue:22

    Topics: Animals; Astrocytes; Cerebellum; Gene Expression Profiling; Gene Expression Regulation, Developmental; Glial Fibrillary Acidic Protein; Glutamic Acid; In Vitro Techniques; Integrases; Male; Mice; Mice, Neurologic Mutants; Mice, Transgenic; Nerve Degeneration; Nerve Tissue Proteins; Patch-Clamp Techniques; Psychomotor Disorders; Purkinje Cells; Ribonuclease III; Signal Transduction

2011
Creatine prevents behavioral alterations caused by methylmalonic acid administration into the hippocampus of rats in the open field task.
    Journal of the neurological sciences, 2006, May-15, Volume: 244, Issue:1-2

    Topics: Animals; Avoidance Learning; Brain Diseases, Metabolic, Inborn; Creatine; Disease Models, Animal; Energy Metabolism; Excitatory Amino Acid Antagonists; Exploratory Behavior; Glutamic Acid; Habituation, Psychophysiologic; Hippocampus; Male; Memory; Memory Disorders; Methylmalonic Acid; Neuropsychological Tests; Psychomotor Disorders; Rats; Rats, Wistar; Receptors, N-Methyl-D-Aspartate; Succinic Acid; Synaptic Transmission

2006
Hypolocomotion in rats with chronic liver failure is due to increased glutamate and activation of metabotropic glutamate receptors in substantia nigra.
    Journal of hepatology, 2006, Volume: 45, Issue:5

    Topics: Animals; Basal Ganglia; Chromones; Chronic Disease; GABA Antagonists; gamma-Aminobutyric Acid; Glutamic Acid; Hepatic Encephalopathy; Liver Failure; Male; Models, Animal; Motor Activity; Motor Cortex; Portacaval Shunt, Surgical; Psychomotor Disorders; Rats; Rats, Wistar; Receptors, Metabotropic Glutamate; Substantia Nigra; Subthalamic Nucleus

2006