glutamic acid has been researched along with Cardiomyopathy, Hypertrophic Obstructive in 11 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 3 (27.27) | 18.2507 |
| 2000's | 1 (9.09) | 29.6817 |
| 2010's | 7 (63.64) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Aguiar-Pulido, V; Huang, W; Kazmierczak, K; Narasimhan, G; Szczesna-Cordary, D; Zhou, Z | 1 |
| Armel, TZ; Leinwand, LA | 1 |
| Swindle, N; Tikunova, SB | 1 |
| Borovikov, YS; Karpicheva, OE; Redwood, CS; Rysev, NA | 2 |
| Kim, DS; Kim, HS; Kim, SY; Lee, CH; Park, YE; Yang, DH | 1 |
| Fischer, S; Geeves, MA; Janco, M; Lehman, W; Li, XE; Marston, SB; Suphamungmee, W | 1 |
| Kogaki, S; Kurotobi, S; Nasuno, S; Ohta, M; Okabe, H; Ozono, K; Sakai, N; Takahashi, K; Taniike, M; Wada, K | 1 |
| Adachi, K; Harada, H; Kimura, A; Koga, Y; Nishi, H; Sasazuki, T; Toshima, H | 1 |
| Bing, W; Esposito, G; Marston, SB; Purcell, IF; Redwood, CS; Watkins, H | 1 |
| Morimoto, S; Nakaura, H; Ohtsuki, I; Yanaga, F | 1 |
11 other study(ies) available for glutamic acid and Cardiomyopathy, Hypertrophic Obstructive
| Article | Year |
|---|---|
Gene expression patterns in transgenic mouse models of hypertrophic cardiomyopathy caused by mutations in myosin regulatory light chain.
Topics: Algorithms; Animals; Arginine; Cardiomyopathy, Hypertrophic; Computational Biology; Gene Expression Profiling; Gene Expression Regulation; Glutamic Acid; Glutamine; Lysine; Mice; Mice, Transgenic; Multigene Family; Mutation; Myocardium; Myosin Light Chains; Oligonucleotide Array Sequence Analysis; Phenotype; Principal Component Analysis; Valine | 2016 |
A mutation in the beta-myosin rod associated with hypertrophic cardiomyopathy has an unexpected molecular phenotype.
Topics: Amino Acid Substitution; Cardiomyopathy, Hypertrophic; Glutamic Acid; Humans; Lysine; Mutation; Phenotype; Protein Stability; Protein Structure, Secondary; Thermodynamics; Ventricular Myosins | 2010 |
Hypertrophic cardiomyopathy-linked mutation D145E drastically alters calcium binding by the C-domain of cardiac troponin C.
Topics: Amino Acid Substitution; Aspartic Acid; Calcium; Calcium Signaling; Cardiomyopathy, Hypertrophic; Glutamic Acid; Humans; Magnesium; Muscle Contraction; Mutation; Protein Binding; Protein Structure, Tertiary; Troponin C | 2010 |
Hypertrophic cardiomyopathy-causing Asp175asn and Glu180gly Tpm1 mutations shift tropomyosin strands further towards the open position during the ATPase cycle.
Topics: Actins; Adenosine Triphosphatases; Amino Acid Substitution; Aspartic Acid; Cardiomyopathy, Hypertrophic; Glutamic Acid; Humans; Mutation; Protein Conformation; Tropomyosin | 2011 |
Nemaline myopathy and non-fatal hypertrophic cardiomyopathy caused by a novel ACTA1 E239K mutation.
Topics: Actins; Amino Acid Sequence; Cardiomyopathy, Hypertrophic; Glutamic Acid; Humans; Lysine; Male; Molecular Sequence Data; Muscle, Skeletal; Myopathies, Nemaline; Point Mutation; Young Adult | 2011 |
The effect of the Asp175Asn and Glu180Gly TPM1 mutations on actin-myosin interaction during the ATPase cycle.
Topics: Actins; Actomyosin; Adenosine Triphosphatases; Amino Acid Substitution; Animals; Asparagine; Aspartic Acid; Cardiomyopathy, Hypertrophic; Fluorescent Dyes; Glutamic Acid; Glycine; Humans; Muscle Contraction; Mutation; Myosin Subfragments; Naphthalenesulfonates; Peptide Fragments; Protein Structure, Secondary; Rabbits; Spectrometry, Fluorescence; Tropomyosin | 2012 |
The flexibility of two tropomyosin mutants, D175N and E180G, that cause hypertrophic cardiomyopathy.
Topics: Actins; Amino Acid Substitution; Asparagine; Aspartic Acid; Calcium; Cardiomyopathy, Hypertrophic; Glutamic Acid; Glycine; Humans; Microscopy, Electron; Models, Chemical; Molecular Dynamics Simulation; Mutation; Myosins; Protein Structure, Secondary; Tropomyosin | 2012 |
A novel mutation in the PTPN11 gene in a patient with Noonan syndrome and rapidly progressive hypertrophic cardiomyopathy.
Topics: Cardiomyopathy, Hypertrophic; Cardiovascular Agents; Chromosomes, Human, Pair 12; Drug Therapy, Combination; Glutamic Acid; Glutamine; Humans; Imidazoles; Infant; Intracellular Signaling Peptides and Proteins; Male; Mutation, Missense; Noonan Syndrome; Propranolol; Protein Tyrosine Phosphatase, Non-Receptor Type 11; Protein Tyrosine Phosphatases; Treatment Outcome | 2005 |
Possible gene dose effect of a mutant cardiac beta-myosin heavy chain gene on the clinical expression of familial hypertrophic cardiomyopathy.
Topics: Adenine; Adult; Amino Acid Sequence; Animals; Base Sequence; Cardiomyopathy, Hypertrophic; Chickens; Codon; DNA Primers; Echocardiography; Female; Glutamates; Glutamic Acid; Guanine; Humans; Lysine; Male; Molecular Sequence Data; Myocardium; Myosins; Pedigree; Point Mutation; Polymerase Chain Reaction; Rats; Sequence Homology, Amino Acid | 1994 |
Effects of two hypertrophic cardiomyopathy mutations in alpha-tropomyosin, Asp175Asn and Glu180Gly, on Ca2+ regulation of thin filament motility.
Topics: Actins; Animals; Asparagine; Aspartic Acid; Cardiomyopathy, Hypertrophic; Glutamic Acid; Glycine; Muscle, Skeletal; Mutation; Myosin Subfragments; Phalloidine; Rabbits; Rhodamines; Tropomyosin; Troponin | 1997 |
Effects of missense mutations Phe110Ile and Glu244Asp in human cardiac troponin T on force generation in skinned cardiac muscle fibers.
Topics: Animals; Aspartic Acid; Base Sequence; Calcium; Cardiomyopathy, Hypertrophic; DNA Primers; Glutamic Acid; Humans; Isoleucine; Male; Muscle Fibers, Skeletal; Mutation, Missense; Myocardium; Phenylalanine; Rabbits; Troponin T | 1999 |