glucosamine has been researched along with Carotid Arteriopathies, Traumatic in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (50.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chen, M; Chen, YF; Gong, K; Li, R; Oparil, S; Shen, H; Xu, L; Xu, O; Yao, D; Zhang, F; Zhang, Z; Zhu, H | 1 |
| Chatham, JC; Chen, YF; Feng, W; Majid-Hassan, E; Miller, AP; Nöt, LG; Oparil, S; Xing, D; Zhang, Y | 1 |
2 other study(ies) available for glucosamine and Carotid Arteriopathies, Traumatic
| Article | Year |
|---|---|
O-Linked β-N-Acetylglucosamine Modification of A20 Enhances the Inhibition of NF-κB (Nuclear Factor-κB) Activation and Elicits Vascular Protection After Acute Endoluminal Arterial Injury.
Topics: Acetylglucosamine; Animals; Anti-Inflammatory Agents; Carotid Artery Injuries; Cell Proliferation; Cells, Cultured; Cytokines; Disease Models, Animal; DNA-Binding Proteins; Glucosamine; Glycosylation; Male; Muscle, Smooth, Vascular; Myocytes, Smooth Muscle; Neointima; NF-kappa B; Rats, Sprague-Dawley; Signal Transduction; Tumor Necrosis Factor alpha-Induced Protein 3 | 2018 |
Increased protein O-GlcNAc modification inhibits inflammatory and neointimal responses to acute endoluminal arterial injury.
Topics: Acetylglucosamine; Animals; Anti-Inflammatory Agents; Carotid Arteries; Carotid Artery Injuries; Disease Models, Animal; Female; Glucosamine; Glycosylation; Inflammation Mediators; Monocytes; Neutrophil Infiltration; Ovariectomy; Oximes; Phenylcarbamates; Protein Processing, Post-Translational; Rats; Rats, Sprague-Dawley; Time Factors | 2008 |