glucaric acid has been researched along with Alloxan Diabetes in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bhattacharya, S; Gachhui, R; Manna, P; Sil, PC | 2 |
| Bhattacharya, S; Rashid, K; Sil, PC | 1 |
3 other study(ies) available for glucaric acid and Alloxan Diabetes
| Article | Year |
|---|---|
D-saccharic acid-1,4-lactone ameliorates alloxan-induced diabetes mellitus and oxidative stress in rats through inhibiting pancreatic β-cells from apoptosis via mitochondrial dependent pathway.
Topics: Animals; Apoptosis; Diabetes Mellitus, Experimental; Dose-Response Relationship, Drug; Glucaric Acid; Insulin-Secreting Cells; Male; Membrane Potential, Mitochondrial; Mitochondria; Oxidative Stress; Random Allocation; Rats; Signal Transduction | 2011 |
Protective role of D-saccharic acid-1,4-lactone in alloxan induced oxidative stress in the spleen tissue of diabetic rats is mediated by suppressing mitochondria dependent apoptotic pathway.
Topics: Alloxan; Animals; Antioxidants; Apoptosis; Blood Glucose; Body Weight; Diabetes Mellitus, Experimental; Drug Evaluation, Preclinical; Glucaric Acid; Glycated Hemoglobin; Hypoglycemic Agents; Insulin; Lactones; Lipid Peroxidation; Male; Membrane Potential, Mitochondrial; Mitochondria; Oxidative Stress; Protein Carbonylation; Rats; Reactive Oxygen Species; Spleen; Tumor Necrosis Factor-alpha | 2012 |
D-saccharic acid 1,4-lactone protects diabetic rat kidney by ameliorating hyperglycemia-mediated oxidative stress and renal inflammatory cytokines via NF-κB and PKC signaling.
Topics: Animals; Cytokines; Diabetes Mellitus, Experimental; Glucaric Acid; Hyperglycemia; Inflammation Mediators; Kidney; Lactones; Male; NF-kappa B; Oxidative Stress; Protective Agents; Protein Kinase C; Random Allocation; Rats; Signal Transduction | 2013 |