Page last updated: 2024-09-04

glucagon and Ataxias, Hereditary

glucagon has been researched along with Ataxias, Hereditary in 1 studies

Research

Studies (1)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	1 (100.00)	29.6817
2010's	0 (0.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Alb, JG; Albin, RL; Bankaitis, VA; Cortese, JD; Hamilton, BA; Nagy, TR; Phillips, SE	1

Other Studies

1 other study(ies) available for glucagon and Ataxias, Hereditary

Article	Year
Mice lacking phosphatidylinositol transfer protein-alpha exhibit spinocerebellar degeneration, intestinal and hepatic steatosis, and hypoglycemia. The Journal of biological chemistry, 2003, Aug-29, Volume: 278, Issue:35 Topics: Adenosine Triphosphate; Animals; Brain; Carrier Proteins; Cerebellum; Dose-Response Relationship, Drug; Endoplasmic Reticulum; Fatty Acids; Genetic Vectors; Genotype; Glucagon; Glycogen; Hypoglycemia; In Situ Nick-End Labeling; Intestinal Diseases; Lipid Metabolism; Liver; Liver Diseases; Membrane Proteins; Mice; Mice, Inbred C57BL; Mice, Transgenic; Microscopy, Electron; Models, Genetic; Phenotype; Phospholipid Transfer Proteins; Proglucagon; Protein Precursors; Saccharomyces cerevisiae Proteins; Spinocerebellar Degenerations; Time Factors	2003

Article

Year

Mice lacking phosphatidylinositol transfer protein-alpha exhibit spinocerebellar degeneration, intestinal and hepatic steatosis, and hypoglycemia.

The Journal of biological chemistry, 2003, Aug-29, Volume: 278, Issue:35

Topics: Adenosine Triphosphate; Animals; Brain; Carrier Proteins; Cerebellum; Dose-Response Relationship, Drug; Endoplasmic Reticulum; Fatty Acids; Genetic Vectors; Genotype; Glucagon; Glycogen; Hypoglycemia; In Situ Nick-End Labeling; Intestinal Diseases; Lipid Metabolism; Liver; Liver Diseases; Membrane Proteins; Mice; Mice, Inbred C57BL; Mice, Transgenic; Microscopy, Electron; Models, Genetic; Phenotype; Phospholipid Transfer Proteins; Proglucagon; Protein Precursors; Saccharomyces cerevisiae Proteins; Spinocerebellar Degenerations; Time Factors

2003