gliotoxin has been researched along with B-Cell Chronic Lymphocytic Leukemia in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 1 (50.00) | 24.3611 |
2020's | 1 (50.00) | 2.80 |
Authors | Studies |
---|---|
Ali, F; Araghi, M; Demirtas, D; Hilgarth, M; Hubmann, R; Jäger, U; Rendeiro, AF; Schmidl, C; Schnabl, S; Shehata, M; Staber, PB; Valent, P; Zielinski, C | 1 |
Demirtas, D; Hilgarth, M; Hubmann, R; Jäger, U; Ponath, E; Reiter, M; Schnabl, S; Shehata, M; Sieghart, W; Valent, P; Zielinski, C | 1 |
2 other study(ies) available for gliotoxin and B-Cell Chronic Lymphocytic Leukemia
Article | Year |
---|---|
Targeting Nuclear NOTCH2 by Gliotoxin Recovers a Tumor-Suppressor NOTCH3 Activity in CLL.
Topics: Adult; Aged; Aged, 80 and over; Amyloid Precursor Protein Secretases; Antineoplastic Agents; Apoptosis; Benzazepines; Cell Survival; Dose-Response Relationship, Drug; Down-Regulation; Female; Gliotoxin; Humans; Lectins, C-Type; Leukemia, Lymphocytic, Chronic, B-Cell; Male; Middle Aged; Nuclear Receptor Subfamily 4, Group A, Member 1; Receptor, Notch2; Receptor, Notch3; Receptors, IgE; Regulatory Elements, Transcriptional; RNA, Messenger; Signal Transduction; Tumor Cells, Cultured | 2020 |
Gliotoxin is a potent NOTCH2 transactivation inhibitor and efficiently induces apoptosis in chronic lymphocytic leukaemia (CLL) cells.
Topics: Aged; Aged, 80 and over; Apoptosis; Cell Line, Tumor; Coculture Techniques; Depression, Chemical; DNA Mutational Analysis; Female; Gene Expression Regulation, Leukemic; Gliotoxin; Humans; Immunoglobulin Heavy Chains; Inhibitory Concentration 50; Leukemia, Lymphocytic, Chronic, B-Cell; Male; Middle Aged; Neoplasm Proteins; NF-kappa B; Nuclear Receptor Subfamily 4, Group A, Member 1; Proteasome Endopeptidase Complex; Receptor, Notch2; Receptor, Notch3; Receptors, Notch; Stromal Cells; Transcription Factors; Transcriptional Activation; Tumor Cells, Cultured | 2013 |