Page last updated: 2024-10-27

glimepiride and Reperfusion Injury

glimepiride has been researched along with Reperfusion Injury in 2 studies

glimepiride: structure given in first source

Reperfusion Injury: Adverse functional, metabolic, or structural changes in tissues that result from the restoration of blood flow to the tissue (REPERFUSION) following ISCHEMIA.

Research Excerpts

ExcerptRelevanceReference
" In resistance vessels, venous occlusion plethysmography was used to measure the dilator response to acetylcholine (ACh) [area under ACh dose-response curve (ACh AUC)]."1.37Postconditioning protects against human endothelial ischaemia-reperfusion injury via subtype-specific KATP channel activation and is mimicked by inhibition of the mitochondrial permeability transition pore. ( Bhavsar, DD; Charakida, M; Deanfield, JE; Loukogeorgakis, SP; MacAllister, RJ; Okorie, MI; Ridout, D, 2011)
"Glimepiride (10 microM) treatment for the first 10 min of reperfusion significantly reduced infarct size from 67."1.35Glimepiride treatment upon reperfusion limits infarct size via the phosphatidylinositol 3-kinase/Akt pathway in rabbit hearts. ( Miyazaki, M; Nakaya, H; Nishida, H; Nomura, M; Sato, T, 2009)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (50.00)29.6817
2010's1 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Nishida, H1
Sato, T1
Nomura, M1
Miyazaki, M1
Nakaya, H1
Okorie, MI1
Bhavsar, DD1
Ridout, D1
Charakida, M1
Deanfield, JE1
Loukogeorgakis, SP1
MacAllister, RJ1

Other Studies

2 other studies available for glimepiride and Reperfusion Injury

ArticleYear
Glimepiride treatment upon reperfusion limits infarct size via the phosphatidylinositol 3-kinase/Akt pathway in rabbit hearts.
    Journal of pharmacological sciences, 2009, Volume: 109, Issue:2

    Topics: Animals; In Vitro Techniques; Myocardial Infarction; Phosphatidylinositol 3-Kinases; Phosphorylation

2009
Postconditioning protects against human endothelial ischaemia-reperfusion injury via subtype-specific KATP channel activation and is mimicked by inhibition of the mitochondrial permeability transition pore.
    European heart journal, 2011, Volume: 32, Issue:10

    Topics: Acetylcholine; Adult; Analysis of Variance; Brachial Artery; Cyclosporine; Endothelium, Vascular; Fe

2011