gingerol and Myocarditis

gingerol has been researched along with Myocarditis* in 1 studies

Other Studies

1 other study(ies) available for gingerol and Myocarditis

Article	Year
[6]-Gingerol Ameliorates ISO-Induced Myocardial Fibrosis by Reducing Oxidative Stress, Inflammation, and Apoptosis through Inhibition of TLR4/MAPKs/NF-κB Pathway. Molecular nutrition & food research, 2020, Volume: 64, Issue:13 [6]-Gingerol is one of the primary pungent constituents of ginger. While [6]-gingerol has many pharmacological effects, its benefits for myocardial fibrosis, including its exact role and underlying mechanisms, remain largely unexplored. The present study is designed to characterize the cardio-protective effects of [6]-gingerol in myocardial fibrosis mice and possible underlying mechanisms.. The protective effect of [6]-gingerol in mice with ISO-induced myocardial fibrosis may be related to the inhibition of oxidative stress, inflammation, and apoptosis, potentially through the TLR4/MAPKs/NF-κB signaling pathway. Topics: Adrenergic beta-Agonists; Animals; Apoptosis; Calcium; Cardiotonic Agents; Catechols; Electrocardiography; Fatty Alcohols; Heart; Isoproterenol; Male; Mice; Mitogen-Activated Protein Kinases; Myocarditis; Myocardium; NF-kappa B; Organ Size; Oxidative Stress; Toll-Like Receptor 4	2020

Article

Year

[6]-Gingerol Ameliorates ISO-Induced Myocardial Fibrosis by Reducing Oxidative Stress, Inflammation, and Apoptosis through Inhibition of TLR4/MAPKs/NF-κB Pathway.

Molecular nutrition & food research, 2020, Volume: 64, Issue:13

[6]-Gingerol is one of the primary pungent constituents of ginger. While [6]-gingerol has many pharmacological effects, its benefits for myocardial fibrosis, including its exact role and underlying mechanisms, remain largely unexplored. The present study is designed to characterize the cardio-protective effects of [6]-gingerol in myocardial fibrosis mice and possible underlying mechanisms.. The protective effect of [6]-gingerol in mice with ISO-induced myocardial fibrosis may be related to the inhibition of oxidative stress, inflammation, and apoptosis, potentially through the TLR4/MAPKs/NF-κB signaling pathway.

Topics: Adrenergic beta-Agonists; Animals; Apoptosis; Calcium; Cardiotonic Agents; Catechols; Electrocardiography; Fatty Alcohols; Heart; Isoproterenol; Male; Mice; Mitogen-Activated Protein Kinases; Myocarditis; Myocardium; NF-kappa B; Organ Size; Oxidative Stress; Toll-Like Receptor 4

2020