geniposide has been researched along with Colitis, Ulcerative in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 2 (50.00) | 2.80 |
Authors | Studies |
---|---|
Bian, Y; Dou, DH; Gu, Y; Shi, JX; Shi, L; Yu, B; Yu, Y; Yuan, DP | 1 |
Ji, T; Jin, X; Li, R; Quan, Z; Tong, H; Xu, F; Xue, L; Yu, W; Zhuge, X | 1 |
Cao, Y; Fu, Y; Li, S; Li, Y; Liu, B; Liu, J; Lu, X; Shen, P; Zhang, N; Zhang, Z | 1 |
Li, CX; Li, YL; Lian, MQ; Lin, Y; Tang, ZY; Xu, B; Xu, M; Yu, CC | 1 |
4 other study(ies) available for geniposide and Colitis, Ulcerative
Article | Year |
---|---|
Geniposide promotes splenic Treg differentiation to alleviate colonic inflammation and intestinal barrier injury in ulcerative colitis mice.
Topics: Animals; Caco-2 Cells; Cell Differentiation; Colitis, Ulcerative; Disease Models, Animal; Forkhead Transcription Factors; Humans; Inflammation; Lipopolysaccharides; Mice; Mice, Inbred C57BL; Spleen; T-Lymphocytes, Regulatory; Tumor Necrosis Factor-alpha | 2022 |
Geniposide ameliorates dextran sulfate sodium-induced ulcerative colitis via KEAP1-Nrf2 signaling pathway.
Topics: Animals; Anti-Inflammatory Agents; Antioxidants; Caco-2 Cells; Colitis; Colitis, Ulcerative; Colon; Dextran Sulfate; Humans; Inflammation; Kelch-Like ECH-Associated Protein 1; Lipopolysaccharides; Mice; Mice, Inbred C57BL; Molecular Docking Simulation; NF-E2-Related Factor 2; NF-KappaB Inhibitor alpha; Signal Transduction | 2023 |
Administration of geniposide ameliorates dextran sulfate sodium-induced colitis in mice via inhibition of inflammation and mucosal damage.
Topics: Animals; Anti-Inflammatory Agents; Caco-2 Cells; Colitis; Colitis, Ulcerative; Colon; Cytokines; Dextran Sulfate; Disease Models, Animal; Humans; Inflammation; Inflammation Mediators; Intestinal Mucosa; Iridoids; Male; Mice; Mice, Inbred C57BL; NF-kappa B; PPAR gamma; Signal Transduction | 2017 |
Geniposide ameliorates TNBS-induced experimental colitis in rats via reducing inflammatory cytokine release and restoring impaired intestinal barrier function.
Topics: Animals; Anti-Inflammatory Agents, Non-Steroidal; Body Weight; Caco-2 Cells; Colitis, Ulcerative; Cytokines; Down-Regulation; Humans; Intestinal Mucosa; Iridoids; Male; Neutrophil Infiltration; Permeability; Rats, Sprague-Dawley; Sulfasalazine; Trinitrobenzenesulfonic Acid; Up-Regulation | 2017 |