Page last updated: 2024-09-02

geniposide and Cognitive Decline

geniposide has been researched along with Cognitive Decline in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (33.33)24.3611
2020's2 (66.67)2.80

Authors

AuthorsStudies
Chen, T; He, L; Li, Y; Liu, S; Zheng, M1
Gao, W; Li, L; Liu, Y; Wang, X; Zhang, D; Zhang, Z1
Li, L; Liu, Y; Wang, X; Zhang, D; Zhang, Z1

Other Studies

3 other study(ies) available for geniposide and Cognitive Decline

ArticleYear
The protective effect of Geniposide on diabetic cognitive impairment through BTK/TLR4/NF-κB pathway.
    Psychopharmacology, 2020, Volume: 237, Issue:2

    Topics: Agammaglobulinaemia Tyrosine Kinase; Animals; Blood Glucose; Cell Line, Tumor; Cognitive Dysfunction; Diabetes Mellitus; Dose-Response Relationship, Drug; Humans; Iridoids; Male; Mice; Mice, Inbred ICR; NF-kappa B; Random Allocation; Signal Transduction; Streptozocin; Toll-Like Receptor 4; Treatment Outcome

2020
Geniposide effectively reverses cognitive impairment and inhibits pathological cerebral damage by regulating the mTOR Signal pathway in APP∕PS1 mice.
    Neuroscience letters, 2020, 02-16, Volume: 720

    Topics: Alzheimer Disease; Amyloid beta-Peptides; Animals; Cerebral Cortex; Cognitive Dysfunction; Disease Models, Animal; Hippocampus; Iridoids; Male; Mice, Inbred C57BL; Mice, Transgenic; Neuroprotective Agents; Peptide Fragments; Plaque, Amyloid; Signal Transduction; TOR Serine-Threonine Kinases

2020
Geniposide-mediated protection against amyloid deposition and behavioral impairment correlates with downregulation of mTOR signaling and enhanced autophagy in a mouse model of Alzheimer's disease.
    Aging, 2019, 01-26, Volume: 11, Issue:2

    Topics: Alzheimer Disease; Amyloid; Amyloid beta-Protein Precursor; Animals; Autophagy; Behavior, Animal; Cognitive Dysfunction; Down-Regulation; Gene Expression Regulation; Humans; Iridoids; Mice; Mice, Transgenic; Peptide Fragments; Plaque, Amyloid; Random Allocation; TOR Serine-Threonine Kinases

2019