gefitinib has been researched along with Cardiac Hypertrophy in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Carter, RL; Gold, JI; Grisanti, LA; Koch, WJ; Repas, AA; Talarico, JA; Tilley, DG | 1 |
Arimoto, T; Honda, Y; Kadomatsu, K; Kadowaki, S; Kinoshita, D; Kishida, S; Kubota, I; Miyamoto, T; Narumi, T; Netsu, S; Nishiyama, S; Shishido, T; Takahashi, H; Takahashi, T; Takeishi, Y; Watanabe, T | 1 |
Ahmad, SF; Al-Alallah, IA; Alhaider, AA; Anazi, FE; Ansari, MA; Assiri, MA; Attafi, IM; Belali, OM; Korashy, HM | 1 |
3 other study(ies) available for gefitinib and Cardiac Hypertrophy
Article | Year |
---|---|
Temporal and gefitinib-sensitive regulation of cardiac cytokine expression via chronic β-adrenergic receptor stimulation.
Topics: Adrenergic beta-Agonists; Animals; Apoptosis; Cardiomegaly; Cells, Cultured; Chemokine CCL2; ErbB Receptors; Fibrosis; Gefitinib; Heart Ventricles; Isoproterenol; Male; Mice; Mice, Inbred C57BL; Myocardial Contraction; Myocytes, Cardiac; Myofibroblasts; Quinazolines; RNA, Messenger; Tumor Necrosis Factor-alpha; Ventricular Remodeling | 2015 |
Midkine Deteriorates Cardiac Remodeling via Epidermal Growth Factor Receptor Signaling in Chronic Kidney Disease.
Topics: Animals; Animals, Newborn; Cardiomegaly; Cells, Cultured; Cytokines; ErbB Receptors; Gefitinib; Mice; Mice, Inbred C57BL; Mice, Knockout; Midkine; Myocytes, Cardiac; Nephrectomy; Phosphorylation; Quinazolines; Rats; Rats, Sprague-Dawley; Renal Insufficiency, Chronic; Sensitivity and Specificity; Signal Transduction; Ventricular Remodeling | 2016 |
Molecular mechanisms of cardiotoxicity of gefitinib in vivo and in vitro rat cardiomyocyte: Role of apoptosis and oxidative stress.
Topics: Animals; Antineoplastic Agents; Apoptosis; Cardiomegaly; Cardiotoxicity; Caspase 3; Cell Line; Dose-Response Relationship, Drug; Gefitinib; Gene Expression Regulation; Heme Oxygenase (Decyclizing); Male; Myocytes, Cardiac; Oxidative Stress; Protein Kinase Inhibitors; Quinazolines; Rats, Wistar; Reactive Oxygen Species; Signal Transduction; Tumor Suppressor Protein p53 | 2016 |