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gamma-aminobutyric acid and Cranial Nerve II Injuries

gamma-aminobutyric acid has been researched along with Cranial Nerve II Injuries in 6 studies

gamma-Aminobutyric Acid: The most common inhibitory neurotransmitter in the central nervous system.
gamma-aminobutyric acid : A gamma-amino acid that is butanoic acid with the amino substituent located at C-4.

Research Excerpts

ExcerptRelevanceReference
"Amacrine cells identified by GABA and glycine labeling were not significantly affected by experimental glaucoma, with a mean decrease of 15% compared with bilaterally untreated control cells (557 +/- 186 neurons/mm [glaucoma] versus 653."3.73The effect of experimental glaucoma and optic nerve transection on amacrine cells in the rat retina. ( Kielczewski, JL; Pease, ME; Quigley, HA, 2005)
"Retinal metabolic changes in optic nerve transection (ONT) rat model were analyzed by (1)H magnetic resonance spectroscopy ((1)H-MRS)."1.37Retinal metabolic changes in an experimental model of optic nerve transection by ex vivo 1H magnetic resonance spectroscopy. ( Chen, F; Huang, M; Jiang, F; Lei, H; Li, S; Wang, X, 2011)
"Complete optic nerve transection was performed unilaterally in nine rats with survival for 1 (n = 4) and 3 (n = 5) months."1.33The effect of experimental glaucoma and optic nerve transection on amacrine cells in the rat retina. ( Kielczewski, JL; Pease, ME; Quigley, HA, 2005)

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19901 (16.67)18.7374
1990's0 (0.00)18.2507
2000's3 (50.00)29.6817
2010's1 (16.67)24.3611
2020's1 (16.67)2.80

Authors

AuthorsStudies
Zhu, Y1
Zhang, Y1
Qi, X1
Lian, Y1
Che, H1
Jia, J1
Yang, C1
Xu, Y1
Chi, X1
Jiang, W1
Li, Y1
Mi, J1
Yang, Y1
Li, X1
Tian, G1
Constantinou, S1
Fern, R1
Li, S1
Huang, M1
Wang, X2
Chen, F1
Lei, H1
Jiang, F1
Bui, BV1
Fortune, B1
Kielczewski, JL1
Pease, ME1
Quigley, HA1
Somogyi, J1
Eysel, U1
Hamori, J1

Other Studies

6 other studies available for gamma-aminobutyric acid and Cranial Nerve II Injuries

ArticleYear
GAD1 alleviates injury-induced optic neurodegeneration by inhibiting retinal ganglion cell apoptosis.
    Experimental eye research, 2022, Volume: 223

    Topics: Animals; Apoptosis; Disease Models, Animal; gamma-Aminobutyric Acid; Glutamate Decarboxylase; Glutam

2022
Conduction block and glial injury induced in developing central white matter by glycine, GABA, noradrenalin, or nicotine, studied in isolated neonatal rat optic nerve.
    Glia, 2009, Aug-15, Volume: 57, Issue:11

    Topics: Action Potentials; Animals; Animals, Newborn; Astrocytes; Cell Survival; gamma-Aminobutyric Acid; Gl

2009
Retinal metabolic changes in an experimental model of optic nerve transection by ex vivo 1H magnetic resonance spectroscopy.
    Neurochemical research, 2011, Volume: 36, Issue:12

    Topics: Animals; Aspartic Acid; Axotomy; Creatine; Female; gamma-Aminobutyric Acid; Glial Fibrillary Acidic

2011
Ganglion cell contributions to the rat full-field electroretinogram.
    The Journal of physiology, 2004, Feb-15, Volume: 555, Issue:Pt 1

    Topics: Animals; Electroretinography; gamma-Aminobutyric Acid; Optic Nerve Injuries; Photic Stimulation; Rat

2004
The effect of experimental glaucoma and optic nerve transection on amacrine cells in the rat retina.
    Investigative ophthalmology & visual science, 2005, Volume: 46, Issue:9

    Topics: Amacrine Cells; Animals; Axons; Cell Count; Disease Models, Animal; gamma-Aminobutyric Acid; Glaucom

2005
A quantitative study of morphological reorganization following chronic optic deafferentation in the adult cat dorsal lateral geniculate nucleus.
    The Journal of comparative neurology, 1987, Jan-15, Volume: 255, Issue:3

    Topics: Animals; Cats; gamma-Aminobutyric Acid; Geniculate Bodies; Microscopy, Electron; Neuronal Plasticity

1987