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gamma-aminobutyric acid and Arterial Occlusive Diseases

gamma-aminobutyric acid has been researched along with Arterial Occlusive Diseases in 3 studies

gamma-Aminobutyric Acid: The most common inhibitory neurotransmitter in the central nervous system.
gamma-aminobutyric acid : A gamma-amino acid that is butanoic acid with the amino substituent located at C-4.

Arterial Occlusive Diseases: Pathological processes which result in the partial or complete obstruction of ARTERIES. They are characterized by greatly reduced or absence of blood flow through these vessels. They are also known as arterial insufficiency.

Research Excerpts

ExcerptRelevanceReference
"Glycine was about 100 times more potent than GABA and effective in a concentration of 0."1.26Inhibition of vasopressin release to carotid occlusion by gamma-aminobutyric acid and glycine. ( Feldberg, W; Rocha e Silva, M, 1981)

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19902 (66.67)18.7374
1990's1 (33.33)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Feldberg, W1
Rocha e Silva, M1
Melani, A1
Pantoni, L1
Corsi, C1
Bianchi, L1
Monopoli, A1
Bertorelli, R1
Pepeu, G1
Pedata, F1
Klausmair, A1
Philippu, A1

Other Studies

3 other studies available for gamma-aminobutyric acid and Arterial Occlusive Diseases

ArticleYear
Inhibition of vasopressin release to carotid occlusion by gamma-aminobutyric acid and glycine.
    British journal of pharmacology, 1981, Volume: 72, Issue:1

    Topics: Animals; Arterial Occlusive Diseases; Blood Pressure; Brain Stem; Carotid Artery Diseases; Cats; Dos

1981
Striatal outflow of adenosine, excitatory amino acids, gamma-aminobutyric acid, and taurine in awake freely moving rats after middle cerebral artery occlusion: correlations with neurological deficit and histopathological damage.
    Stroke, 1999, Volume: 30, Issue:11

    Topics: Adenosine; Animals; Arterial Occlusive Diseases; Brain Ischemia; Cerebral Cortex; Chromatography, Hi

1999
Carotid occlusion increases the release of endogenous GABA in the nucleus of the solitary tract.
    Naunyn-Schmiedeberg's archives of pharmacology, 1989, Volume: 340, Issue:6 Pt 2

    Topics: Animals; Arterial Occlusive Diseases; Blood Pressure; Carotid Artery Diseases; Cats; Female; gamma-A

1989