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galactose and Electron Transport Chain Deficiencies, Mitochondrial

galactose has been researched along with Electron Transport Chain Deficiencies, Mitochondrial in 14 studies

Research

Studies (14)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (7.14)29.6817
2010's10 (71.43)24.3611
2020's3 (21.43)2.80

Authors

AuthorsStudies
Cunha-Oliveira, T; Deus, CM; Oliveira, PJ; Pereira, FB; Pereira, SP; Raimundo, N1
Cao, Y; Liu, H; Liu, X; Song, M; Xiao, H; Xiao, J; Zhang, X1
Chaturbhuj, SG; Das, M; Gourishetti, K; Kanojia, U; Kishore, A; Krishnadas, N; Nayak, PG; Sankhe, R; Surubhotla, R1
Ito, K; Liu, C; Sekine, S; Song, B1
Araki, T; Nakazono, O; Sanuki, Y; Tsurui, K1
Couch, L; Guo, L; Qing, T; Ren, Z; Shi, L; Tolleson, WH; Xuan, J1
Beyrath, J; Iannetti, EF; Koopman, WJH; Smeitink, JAM; Willems, PHGM1
Hanaoka, K; Kato, J; Kawamura, T; Miyazaki, Y; Morimoto, K; Nakashima, A; Sugai, K; Takahashi, D; Takahashi, H; Tanaka, M; Ueda, H; Yamaguchi, Y; Yokoo, T1
Bennekou, SH; Carta, G; Cediel, A; Delp, J; Forsby, A; Funke, M; Gardner, I; Jennings, P; Leist, M; Rudolf, F; Toma, C; van de Water, B; van der Stel, W1
Chang, L; Guo, Z; Li, H; Liu, J; Liu, X; Long, J; Xiao, K; Yang, Y; Zhang, C; Zhao-Wilson, X1
Bolszak, M; Hakkola, J; Hinttala, R; Koivunen, P; Komulainen, T; Lodge, T; Morten, KJ; Pietilä, M; Poulton, J; Uusimaa, J1
Dou, DQ; Gao, MB; Li, CB; Li, HC; Li, XT1
Bai, Y; Fan, W; Haas, R; Huang, T; Huang, VH; Sharma, LK; Simon, M; Wallace, DC; Zhang, C1
Alexeyev, MF; Bonilla, G; Pastukh, V; Shokolenko, I; Venediktova, N; Wilson, GL1

Other Studies

14 other study(ies) available for galactose and Electron Transport Chain Deficiencies, Mitochondrial

ArticleYear
Mitochondrial remodeling in human skin fibroblasts from sporadic male Parkinson's disease patients uncovers metabolic and mitochondrial bioenergetic defects.
    Biochimica et biophysica acta. Molecular basis of disease, 2020, 03-01, Volume: 1866, Issue:3

    Topics: Aged; Energy Metabolism; Fibroblasts; Galactose; Glucose; Humans; Male; Metabolic Diseases; Middle Aged; Mitochondria; Mitochondrial Diseases; Oxidative Phosphorylation; Oxygen Consumption; Parkinson Disease; Pyruvic Acid; Skin; Substantia Nigra

2020
Astaxanthin attenuates d-galactose-induced brain aging in rats by ameliorating oxidative stress, mitochondrial dysfunction, and regulating metabolic markers.
    Food & function, 2020, May-01, Volume: 11, Issue:5

    Topics: Aging; Animals; Behavior, Animal; Biomarkers; Brain; Chromatography, Liquid; Galactose; Membrane Potential, Mitochondrial; Mitochondrial Diseases; Oxidative Stress; Rats; Rats, Sprague-Dawley; Tandem Mass Spectrometry; Xanthophylls

2020
Beta-Caryophyllene, a CB2R Selective Agonist, Protects Against Cognitive Impairment Caused by Neuro-inflammation and Not in Dementia Due to Ageing Induced by Mitochondrial Dysfunction.
    CNS & neurological disorders drug targets, 2021, Volume: 20, Issue:10

    Topics: Aging; Aluminum Chloride; Animals; Cognitive Dysfunction; Dementia; Disease Models, Animal; Galactose; Hippocampus; Lipid Peroxidation; Male; Maze Learning; Mitochondrial Diseases; Neuroinflammatory Diseases; Neuroprotective Agents; Oxidative Stress; Polycyclic Sesquiterpenes; Rats; Rats, Sprague-Dawley

2021
Use of Primary Rat Hepatocytes for Prediction of Drug-Induced Mitochondrial Dysfunction.
    Current protocols in toxicology, 2017, May-02, Volume: 72

    Topics: Animals; Cell Respiration; Chemical and Drug Induced Liver Injury; Culture Media; Energy Metabolism; Forecasting; Galactose; Hepatocytes; Hyperoxia; L-Lactate Dehydrogenase; Mitochondrial Diseases; Oxygen Consumption; Primary Cell Culture; Rats; Rats, Sprague-Dawley; Toxicity Tests

2017
A rapid mitochondrial toxicity assay utilizing rapidly changing cell energy metabolism.
    The Journal of toxicological sciences, 2017, Volume: 42, Issue:3

    Topics: Adenosine Triphosphate; Chemical and Drug Induced Liver Injury; Drug Discovery; Energy Metabolism; Galactose; Glycolysis; Hep G2 Cells; High-Throughput Screening Assays; Humans; Mitochondria, Liver; Mitochondrial Diseases; Oxidation-Reduction; Phosphorylation

2017
Mitochondrial dysfunction induced by leflunomide and its active metabolite.
    Toxicology, 2018, 03-01, Volume: 396-397

    Topics: Adenosine Triphosphate; Anti-Inflammatory Agents, Non-Steroidal; Bongkrekic Acid; Chemical and Drug Induced Liver Injury; Endoplasmic Reticulum Stress; Galactose; Glucose; Hep G2 Cells; Humans; Isoxazoles; L-Lactate Dehydrogenase; Leflunomide; Membrane Potential, Mitochondrial; Mitochondria, Liver; Mitochondrial Diseases; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Oxidative Phosphorylation

2018
Rescue from galactose-induced death of Leigh Syndrome patient cells by pyruvate and NAD
    Cell death & disease, 2018, 11-14, Volume: 9, Issue:11

    Topics: Adenosine Triphosphate; Aspartic Acid; Cell Death; Culture Media; Electron Transport Complex I; Fibroblasts; Galactose; Gene Expression; Glycolysis; Humans; Ketoglutaric Acids; Leigh Disease; Malates; Mitochondria; Mitochondrial Diseases; Mutation; NAD; NADH Dehydrogenase; Oxaloacetic Acid; Primary Cell Culture; Pyruvic Acid; Skin

2018
Maternally inherited diabetes and deafness complicated by mesangial galactose-deficient IgA1 deposits: a case report.
    BMC nephrology, 2018, 12-10, Volume: 19, Issue:1

    Topics: Adult; Deafness; Diabetes Mellitus, Type 2; Female; Galactose; Humans; Immunoglobulin A; Mesangial Cells; Mitochondrial Diseases; Pedigree

2018
Development of a neurotoxicity assay that is tuned to detect mitochondrial toxicants.
    Archives of toxicology, 2019, Volume: 93, Issue:6

    Topics: Carbohydrate Metabolism; Culture Media; Electron Transport; Electron Transport Complex I; Galactose; Glucose; Humans; Mitochondria; Mitochondrial Diseases; Neural Stem Cells; Neurites; Neurotoxicity Syndromes; Toxicity Tests; Uncoupling Agents

2019
D-galactose induces a mitochondrial complex I deficiency in mouse skeletal muscle: potential benefits of nutrient combination in ameliorating muscle impairment.
    Journal of medicinal food, 2014, Volume: 17, Issue:3

    Topics: Animals; Disease Models, Animal; Electron Transport Complex I; Female; Galactose; Humans; Male; Mice; Mice, Inbred C57BL; Mitochondria; Mitochondrial Diseases; Muscle, Skeletal

2014
Sodium valproate induces mitochondrial respiration dysfunction in HepG2 in vitro cell model.
    Toxicology, 2015, May-04, Volume: 331

    Topics: Adenosine Triphosphate; Anticonvulsants; Cell Death; Cell Proliferation; Cell Respiration; Chemical and Drug Induced Liver Injury; Dose-Response Relationship, Drug; Electron Transport Chain Complex Proteins; Galactose; Glucose; Hep G2 Cells; Hepatocytes; Humans; Membrane Potential, Mitochondrial; Mitochondria, Liver; Mitochondrial Diseases; Oxidative Phosphorylation; Oxidative Stress; Pyruvic Acid; Reactive Oxygen Species; Superoxide Dismutase; Time Factors; Valproic Acid

2015
Protective effects on mitochondria and anti-aging activity of polysaccharides from cultivated fruiting bodies of Cordyceps militaris.
    The American journal of Chinese medicine, 2010, Volume: 38, Issue:6

    Topics: Aging; Animals; Antioxidants; Cordyceps; Cysteine; Disease Models, Animal; Dose-Response Relationship, Drug; Fruiting Bodies, Fungal; Galactose; Hydroxyl Radical; Injections, Subcutaneous; Liver; Male; Mice; Mice, Inbred Strains; Mitochondria; Mitochondrial Diseases; Phytotherapy; Polysaccharides; Reactive Oxygen Species; Superoxides; Thiobarbituric Acid Reactive Substances

2010
Heteroplasmic mutations of the mitochondrial genome cause paradoxical effects on mitochondrial functions.
    FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 2012, Volume: 26, Issue:12

    Topics: Adenosine Triphosphatases; Adult; Apoptosis; Carrier Proteins; Cell Proliferation; Cells, Cultured; Electron Transport Complex I; Galactose; Genome, Mitochondrial; Humans; Hybrid Cells; Male; Membrane Proteins; Mitochondria; Mitochondrial Diseases; Mitochondrial Proton-Translocating ATPases; Oxidative Phosphorylation; Oxygen Consumption; Point Mutation; Reactive Oxygen Species

2012
Selective elimination of mutant mitochondrial genomes as therapeutic strategy for the treatment of NARP and MILS syndromes.
    Gene therapy, 2008, Volume: 15, Issue:7

    Topics: Adenosine Triphosphate; Adenoviridae; Blood Platelets; Cell Line, Tumor; Cell Proliferation; Cell Respiration; Coculture Techniques; Deoxyribonucleases, Type II Site-Specific; DNA, Mitochondrial; Galactose; Gene Deletion; Genetic Engineering; Genetic Markers; Genetic Therapy; Genetic Vectors; Genome, Mitochondrial; Humans; Lactic Acid; Mitochondrial Diseases; Mutation; Oxidative Phosphorylation; Transduction, Genetic

2008