galactosamine has been researched along with Systemic Inflammatory Response Syndrome in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (66.67) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Abshagen, K; Kuhla, A; Menger, MD; Norden, J; Vollmar, B | 1 |
| Algül, H; Geisler, F; Riemann, M; Schmid, RM | 1 |
| Brouckaert, P; Hochepied, T; Libert, C; Van Molle, W | 1 |
3 other study(ies) available for galactosamine and Systemic Inflammatory Response Syndrome
| Article | Year |
|---|---|
RAGE blockade and hepatic microcirculation in experimental endotoxaemic liver failure.
Topics: Animals; Antibodies; Endotoxemia; Fluorescent Antibody Technique; Galactosamine; Immunity, Innate; Lipopolysaccharides; Liver; Liver Circulation; Liver Failure; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Microcirculation; Receptor for Advanced Glycation End Products; Receptors, Immunologic; Systemic Inflammatory Response Syndrome; Up-Regulation | 2013 |
Questioning current concepts in acute pancreatitis: endotoxin contamination of porcine pancreatic elastase is responsible for experimental pancreatitis-associated distant organ failure.
Topics: Acute Disease; Adaptor Proteins, Signal Transducing; Animals; Antigens, Differentiation; Bacterial Toxins; Cell Line; Cells, Cultured; Drug Contamination; Endotoxins; Galactosamine; Hot Temperature; Hydrolysis; Macrophages; Mice; Mice, Inbred C57BL; Mice, Knockout; Myeloid Differentiation Factor 88; NF-kappa B; Pancreatic Elastase; Pancreatitis; Polymyxin B; Receptors, Immunologic; Swine; Systemic Inflammatory Response Syndrome; Tumor Necrosis Factor-alpha | 2005 |
The major acute-phase protein, serum amyloid P component, in mice is not involved in endogenous resistance against tumor necrosis factor alpha-induced lethal hepatitis, shock, and skin necrosis.
Topics: Animals; Galactosamine; Hepatitis, Animal; Interleukin-1; Mice; Mice, Inbred C57BL; Necrosis; Serum Amyloid P-Component; Skin; Systemic Inflammatory Response Syndrome; Tumor Necrosis Factor-alpha | 2000 |