Page last updated: 2024-08-22

galactosamine and Infections, Staphylococcal

galactosamine has been researched along with Infections, Staphylococcal in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	1 (50.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	1 (50.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Cisney, ED; Fernandez, S; Kissner, TL; Ruthel, G; Saikh, KU; Ulrich, RG	1
Chow, AW; De Boer, ML; Kum, WW; Pang, LT	1

Other Studies

2 other study(ies) available for galactosamine and Infections, Staphylococcal

Article	Year
MyD88-dependent pro-inflammatory cytokine response contributes to lethal toxicity of staphylococcal enterotoxin B in mice. Innate immunity, 2011, Volume: 17, Issue:5 Topics: Animals; Cells, Cultured; Cytokines; Disease Models, Animal; Enterotoxins; Female; Galactosamine; Gene Expression Regulation; Humans; Inflammation Mediators; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Mice, Knockout; Myeloid Differentiation Factor 88; Shock, Septic; Spleen; Staphylococcal Infections	2011
Co-production of staphylococcal enterotoxin A with toxic shock syndrome toxin-1 (TSST-1) enhances TSST-1 mediated mortality in a D-galactosamine sensitized mouse model of lethal shock. Microbial pathogenesis, 1999, Volume: 27, Issue:2 Topics: Animals; Bacterial Toxins; Blotting, Southern; Disease Models, Animal; Enterotoxins; Female; Galactosamine; Humans; Mice; Mice, Inbred BALB C; Plasmids; Recombination, Genetic; Shock, Septic; Staphylococcal Infections; Staphylococcus aureus; Superantigens; Virulence	1999

Article

Year

MyD88-dependent pro-inflammatory cytokine response contributes to lethal toxicity of staphylococcal enterotoxin B in mice.

Innate immunity, 2011, Volume: 17, Issue:5

Topics: Animals; Cells, Cultured; Cytokines; Disease Models, Animal; Enterotoxins; Female; Galactosamine; Gene Expression Regulation; Humans; Inflammation Mediators; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Mice, Knockout; Myeloid Differentiation Factor 88; Shock, Septic; Spleen; Staphylococcal Infections

2011

Co-production of staphylococcal enterotoxin A with toxic shock syndrome toxin-1 (TSST-1) enhances TSST-1 mediated mortality in a D-galactosamine sensitized mouse model of lethal shock.

Microbial pathogenesis, 1999, Volume: 27, Issue:2

Topics: Animals; Bacterial Toxins; Blotting, Southern; Disease Models, Animal; Enterotoxins; Female; Galactosamine; Humans; Mice; Mice, Inbred BALB C; Plasmids; Recombination, Genetic; Shock, Septic; Staphylococcal Infections; Staphylococcus aureus; Superantigens; Virulence

1999