furosemide has been researched along with Hypertrophy in 12 studies
Furosemide: A benzoic-sulfonamide-furan. It is a diuretic with fast onset and short duration that is used for EDEMA and chronic RENAL INSUFFICIENCY.
furosemide : A chlorobenzoic acid that is 4-chlorobenzoic acid substituted by a (furan-2-ylmethyl)amino and a sulfamoyl group at position 2 and 5 respectively. It is a diuretic used in the treatment of congestive heart failure.
Hypertrophy: General increase in bulk of a part or organ due to CELL ENLARGEMENT and accumulation of FLUIDS AND SECRETIONS, not due to tumor formation, nor to an increase in the number of cells (HYPERPLASIA).
| Excerpt | Relevance | Reference |
|---|---|---|
| "Furosemide treatment produces glomerular hypertrophy and augments glomerular capillary hydraulic pressure in the normal rat." | 7.70 | Long-term furosemide treatment in the normal rat: dissociation of glomerular hypertrophy and glomerulosclerosis. ( Lane, PH, 1999) |
| "We have examined the expression of insulin-like growth factor 1 (IGF-1), IGF binding protein-1 (IGFBP-1), and IGF binding protein-3 (IGFBP-3) in the rat distal nephron during increased cell work load and hypertrophy, induced by the diuretic, furosemide." | 7.69 | Tubular hypertrophy due to work load induced by furosemide is associated with increases of IGF-1 and IGFBP-1. ( Clemmons, DR; Kobayashi, S; Nogami, H; Roy, AK; Venkatachalam, MA, 1995) |
| "Furosemide treatment produces glomerular hypertrophy and augments glomerular capillary hydraulic pressure in the normal rat." | 3.70 | Long-term furosemide treatment in the normal rat: dissociation of glomerular hypertrophy and glomerulosclerosis. ( Lane, PH, 1999) |
| "We have examined the expression of insulin-like growth factor 1 (IGF-1), IGF binding protein-1 (IGFBP-1), and IGF binding protein-3 (IGFBP-3) in the rat distal nephron during increased cell work load and hypertrophy, induced by the diuretic, furosemide." | 3.69 | Tubular hypertrophy due to work load induced by furosemide is associated with increases of IGF-1 and IGFBP-1. ( Clemmons, DR; Kobayashi, S; Nogami, H; Roy, AK; Venkatachalam, MA, 1995) |
| "Congenital hepatic arteriovenous malformations are rarely seen in association with persistent neonatal pulmonary hypertension." | 1.42 | Regression of a large congenital hepatic arteriovenous malformation. ( Agha, HM; Hamza, H; Mostafa, FA; Zakaria, R, 2015) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 3 (25.00) | 18.7374 |
| 1990's | 5 (41.67) | 18.2507 |
| 2000's | 2 (16.67) | 29.6817 |
| 2010's | 2 (16.67) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Agha, HM | 1 |
| Zakaria, R | 1 |
| Mostafa, FA | 1 |
| Hamza, H | 1 |
| Arias-Santiago, S | 1 |
| Aneiros-Fernandez, J | 1 |
| Aceituno-Madera, P | 1 |
| Burkhardt-Perez, P | 1 |
| Naranjo Sintes, R | 1 |
| Jonassen, TE | 1 |
| Christensen, S | 1 |
| Marcussen, N | 1 |
| Petersen, JS | 1 |
| Madala Halagappa, VK | 1 |
| Tiwari, S | 1 |
| Riazi, S | 1 |
| Hu, X | 1 |
| Ecelbarger, CM | 1 |
| Hootman, SR | 1 |
| Ernst, SA | 1 |
| Kobayashi, S | 1 |
| Clemmons, DR | 1 |
| Nogami, H | 1 |
| Roy, AK | 1 |
| Venkatachalam, MA | 1 |
| Elzinga, LW | 1 |
| Rosen, S | 1 |
| Bennett, WM | 1 |
| Peiró, C | 1 |
| Angulo, J | 1 |
| Llergo, JL | 1 |
| Rodríguez-Mañas, L | 1 |
| Marín, J | 1 |
| Sánchez-Ferrer, CF | 1 |
| Lane, PH | 1 |
| Martines, F | 1 |
| Demetrio, G | 1 |
| Di Pentima, G | 1 |
| Batolo, D | 1 |
| Ishikawa, T | 1 |
| Kanno, T | 1 |
| Ben-Ishay, D | 1 |
| Levy, M | 1 |
| Birnbaum, D | 1 |
12 other studies available for furosemide and Hypertrophy
| Article | Year |
|---|---|
Regression of a large congenital hepatic arteriovenous malformation.
Topics: Arteriovenous Malformations; Captopril; Diuretics; Female; Furosemide; Heart Failure; Hepatic Veins; | 2015 |
Hypertrophic lichenoid eruption due to furosemide.
Topics: Administration, Topical; Adrenal Cortex Hormones; Aged; Dermatologic Agents; Diuretics; Drug Eruptio | 2010 |
Intrarenal octreotide treatment prevents sodium retention in liver cirrhotic rats: evidence for direct effects within the thick ascending limb of Henle's loop.
Topics: Animals; Female; Furosemide; Hemodynamics; Hypertrophy; Kidney; Liver Cirrhosis, Biliary; Liver Cirr | 2006 |
Chronic candesartan alters expression and activity of NKCC2, NCC, and ENaC in the obese Zucker rat.
Topics: Amiloride; Angiotensin II; Angiotensin II Type 1 Receptor Blockers; Animals; Benzimidazoles; Bipheny | 2008 |
Characterization of muscarinic acetylcholine receptors in the avian salt gland.
Topics: Adaptation, Physiological; Animals; Binding, Competitive; Furosemide; Hypertrophy; Microscopy, Elect | 1981 |
Tubular hypertrophy due to work load induced by furosemide is associated with increases of IGF-1 and IGFBP-1.
Topics: Animals; Carrier Proteins; Disease Models, Animal; Furosemide; Hypertrophy; Immunohistochemistry; In | 1995 |
Dissociation of glomerular filtration rate from tubulointerstitial fibrosis in experimental chronic cyclosporine nephropathy: role of sodium intake.
Topics: Animals; Atrophy; Cyclosporine; Furosemide; Glomerular Filtration Rate; Hypertrophy; Loop of Henle; | 1993 |
Angiotensin II mediates cell hypertrophy in vascular smooth muscle cultures from hypertensive Ren-2 transgenic rats by an amiloride- and furosemide-sensitive mechanism.
Topics: Angiotensin II; Animals; Animals, Genetically Modified; Biological Transport; Cell Size; Cells, Cult | 1997 |
Long-term furosemide treatment in the normal rat: dissociation of glomerular hypertrophy and glomerulosclerosis.
Topics: Animals; Furosemide; Glomerulosclerosis, Focal Segmental; Hypertrophy; Kidney Cortex; Kidney Glomeru | 1999 |
[Morphologic studies of the juxtaglomerular apparatus of the rat treated with furosemide].
Topics: Angiotensin II; Animals; Furosemide; Hyperplasia; Hypertrophy; Juxtaglomerular Apparatus; Kidney; Ra | 1977 |
Dissociation of CCK-8-induced fluid secretion from protein secretion by ion-transport blockers in rat pancreas.
Topics: 4-Acetamido-4'-isothiocyanatostilbene-2,2'-disulfonic Acid; Amiloride; Animals; Esters; Furosemide; | 1992 |
Self-induced secondary hyperaldosteronism simulating Bartter's syndrome.
Topics: Adult; Alkalosis; Diagnosis, Differential; Female; Furosemide; Humans; Hyperaldosteronism; Hypertrop | 1972 |