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fomesafen and Neurodegenerative Diseases

fomesafen has been researched along with Neurodegenerative Diseases in 7 studies

Research

Studies (7)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's3 (42.86)18.2507
2000's3 (42.86)29.6817
2010's0 (0.00)24.3611
2020's1 (14.29)2.80

Authors

AuthorsStudies
Coppola, A; Cuccurullo, C; Striano, P1
Botto, M; Caspersen, C; Connolly, ES; Mazer, SP; Pinsky, DJ; Sosunov, SA; Stark, RI; Sughrue, ME; Ten, VS1
Aida, T; Banerjee, P; Danysz, W; Filipkowski, RK; Kaczmarek, L; Kiryk, A; Knapska, E; Meyza, K; Tanaka, K; Wilczynski, GM1
Stein, RB; Zehr, EP1
Motyl, R; Orłowiejska-Gillert, M; Pajak, A; Szczudlik, A; Słowik, A; Topór-Madry, R; Turaj, W1
Baba, M; Matsunaga, M; Okushima, T; Ozaki, I; Tanosaki, M1
Caviness, JN; Wszolek, ZK1

Reviews

2 review(s) available for fomesafen and Neurodegenerative Diseases

ArticleYear
Familial Adult Myoclonus Epilepsy: A Non-Coding Repeat Expansion Disorder of Cerebellar-Thalamic-Cortical Loop.
    Cells, 2023, 06-13, Volume: 12, Issue:12

    Topics: Adult; Epilepsies, Myoclonic; Humans; Myoclonus; Neurodegenerative Diseases; Reflex; Tremor

2023
What functions do reflexes serve during human locomotion?
    Progress in neurobiology, 1999, Volume: 58, Issue:2

    Topics: Animals; Humans; Locomotion; Muscle, Skeletal; Neurodegenerative Diseases; Posture; Reflex; Reflex, Stretch; Walking

1999

Other Studies

5 other study(ies) available for fomesafen and Neurodegenerative Diseases

ArticleYear
C1q-deficiency is neuroprotective against hypoxic-ischemic brain injury in neonatal mice.
    Stroke, 2005, Volume: 36, Issue:10

    Topics: Animals; Animals, Newborn; Brain; Brain Injuries; Cerebrovascular Circulation; Complement Activation; Complement C1q; Disease Models, Animal; Flow Cytometry; Hypoxia-Ischemia, Brain; Immunohistochemistry; Maze Learning; Mice; Mice, Inbred C57BL; Mice, Knockout; Mice, Transgenic; Neurodegenerative Diseases; Neuroprotective Agents; Neutrophils; Reflex; Regional Blood Flow; RNA, Messenger; Time Factors; Treatment Outcome; Up-Regulation

2005
Behavioral characterization of GLT1 (+/-) mice as a model of mild glutamatergic hyperfunction.
    Neurotoxicity research, 2008, Volume: 13, Issue:1

    Topics: Animals; Behavior, Animal; Body Size; Conditioning, Psychological; Disease Models, Animal; Excitatory Amino Acid Transporter 2; Exploratory Behavior; Female; Glutamic Acid; Immunohistochemistry; Male; Maze Learning; Mice; Mice, Mutant Strains; Neurodegenerative Diseases; Neurotoxins; Reflex

2008
[Cognitive impairment and cardiovascular disease risk factors. Project CASCADE Kraków. V. Disorders of higher cerebral functions in elderly people (65-78 years old)].
    Przeglad lekarski, 1998, Volume: 55, Issue:12

    Topics: Aged; Aphasia; Cardiovascular Diseases; Cerebrovascular Disorders; Cognition Disorders; Communication Disorders; Comorbidity; Female; Humans; Male; Mental Status Schedule; Neurodegenerative Diseases; Poland; Reflex; Risk Assessment

1998
[A case of clinically diagnosed corticobasal degeneration with unilateral cortical reflex myoclonus showing so-called giant SEP].
    Rinsho shinkeigaku = Clinical neurology, 1999, Volume: 39, Issue:7

    Topics: Aged; Basal Ganglia Diseases; Cerebral Cortex; Electric Stimulation; Evoked Potentials, Somatosensory; Female; Humans; Median Nerve; Myoclonus; Neurodegenerative Diseases; Reaction Time; Reflex

1999
Myoclonus in pallido-ponto-nigral degeneration.
    Advances in neurology, 2002, Volume: 89

    Topics: Electroencephalography; Electromyography; Evoked Potentials, Somatosensory; Globus Pallidus; Humans; Myoclonus; Neurodegenerative Diseases; Pons; Reaction Time; Reflex; Substantia Nigra

2002