Page last updated: 2024-08-23

fomesafen and Injury, Myocardial Reperfusion

fomesafen has been researched along with Injury, Myocardial Reperfusion in 3 studies

Research

Studies (3)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	2 (66.67)	29.6817
2010's	1 (33.33)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Buchholz, B; Del Mauro, JS; Deutsch, ACR; Donato, M; Gelpi, RJ; Höcht, C; Perez, V; Rodríguez, M	1
Chang, KJ; Gengo, PJ; Gross, GJ; Holt, JD; O'Neill, SJ; Pendergast, W; Watson, MJ; Wei, K	1
Fu, LW; Longhurst, JC; Tjen-A-Looi, SC	1

Reviews

1 review(s) available for fomesafen and Injury, Myocardial Reperfusion

Article	Year
Cardiac sympathetic afferent activation provoked by myocardial ischemia and reperfusion. Mechanisms and reflexes. Annals of the New York Academy of Sciences, 2001, Volume: 940 Topics: Animals; Heart; Myocardial Ischemia; Myocardial Reperfusion Injury; Neurons, Afferent; Reflex; Sympathetic Nervous System	2001

Other Studies

2 other study(ies) available for fomesafen and Injury, Myocardial Reperfusion

Article	Year
Changes in the loading conditions induced by vagal stimulation modify the myocardial infarct size through sympathetic-parasympathetic interactions. Pflugers Archiv : European journal of physiology, 2015, Volume: 467, Issue:7 Topics: Adrenergic beta-1 Receptor Antagonists; Animals; Anti-Arrhythmia Agents; Atropine; Catecholamines; Glycogen Synthase Kinase 3; Glycogen Synthase Kinase 3 beta; Hemodynamics; Male; Myocardial Infarction; Myocardial Reperfusion Injury; Propanolamines; Proto-Oncogene Proteins c-akt; Rabbits; Reflex; Signal Transduction; Sympathetic Nervous System; Vagus Nerve	2015
ARD-353 [4-((2R,5S)-4-(R)-(4-diethylcarbamoylphenyl)(3-hydroxyphenyl)methyl)-2,5-dimethylpiperazin-1-ylmethyl)benzoic acid], a novel nonpeptide delta receptor agonist, reduces myocardial infarct size without central effects. The Journal of pharmacology and experimental therapeutics, 2006, Volume: 316, Issue:1 Topics: Animals; Benzoates; Benzylidene Compounds; Binding, Competitive; Cardiotonic Agents; Catalepsy; Dose-Response Relationship, Drug; Hemodynamics; In Vitro Techniques; Male; Mice; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; Naltrexone; Piperazines; Radioligand Assay; Rats; Rats, Sprague-Dawley; Receptors, Opioid, delta; Receptors, Opioid, kappa; Receptors, Opioid, mu; Reflex; Seizures	2006

Article

Year

Changes in the loading conditions induced by vagal stimulation modify the myocardial infarct size through sympathetic-parasympathetic interactions.

Pflugers Archiv : European journal of physiology, 2015, Volume: 467, Issue:7

Topics: Adrenergic beta-1 Receptor Antagonists; Animals; Anti-Arrhythmia Agents; Atropine; Catecholamines; Glycogen Synthase Kinase 3; Glycogen Synthase Kinase 3 beta; Hemodynamics; Male; Myocardial Infarction; Myocardial Reperfusion Injury; Propanolamines; Proto-Oncogene Proteins c-akt; Rabbits; Reflex; Signal Transduction; Sympathetic Nervous System; Vagus Nerve

2015

ARD-353 [4-((2R,5S)-4-(R)-(4-diethylcarbamoylphenyl)(3-hydroxyphenyl)methyl)-2,5-dimethylpiperazin-1-ylmethyl)benzoic acid], a novel nonpeptide delta receptor agonist, reduces myocardial infarct size without central effects.

The Journal of pharmacology and experimental therapeutics, 2006, Volume: 316, Issue:1

Topics: Animals; Benzoates; Benzylidene Compounds; Binding, Competitive; Cardiotonic Agents; Catalepsy; Dose-Response Relationship, Drug; Hemodynamics; In Vitro Techniques; Male; Mice; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; Naltrexone; Piperazines; Radioligand Assay; Rats; Rats, Sprague-Dawley; Receptors, Opioid, delta; Receptors, Opioid, kappa; Receptors, Opioid, mu; Reflex; Seizures

2006