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flutamide and Hepatitis

flutamide has been researched along with Hepatitis in 1 studies

Flutamide: An antiandrogen with about the same potency as cyproterone in rodent and canine species.

Hepatitis: INFLAMMATION of the LIVER.

Research Excerpts

ExcerptRelevanceReference
"Although studies have shown that administration of testosterone receptor antagonist, flutamide, following trauma-hemorrhage, improves hepatic, cardiovascular, and immune functions, the precise cellular/molecular mechanisms responsible for producing these salutary effects remain largely unknown."7.74Flutamide protects against trauma-hemorrhage-induced liver injury via attenuation of the inflammatory response, oxidative stress, and apopotosis. ( Bland, KI; Chaudry, IH; Choudhry, MA; Hsieh, CH; Kan, WH; Raju, R; Schwacha, MG, 2008)
"Although studies have shown that administration of testosterone receptor antagonist, flutamide, following trauma-hemorrhage, improves hepatic, cardiovascular, and immune functions, the precise cellular/molecular mechanisms responsible for producing these salutary effects remain largely unknown."3.74Flutamide protects against trauma-hemorrhage-induced liver injury via attenuation of the inflammatory response, oxidative stress, and apopotosis. ( Bland, KI; Chaudry, IH; Choudhry, MA; Hsieh, CH; Kan, WH; Raju, R; Schwacha, MG, 2008)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Kan, WH1
Hsieh, CH1
Schwacha, MG1
Choudhry, MA1
Raju, R1
Bland, KI1
Chaudry, IH1

Other Studies

1 other study available for flutamide and Hepatitis

ArticleYear
Flutamide protects against trauma-hemorrhage-induced liver injury via attenuation of the inflammatory response, oxidative stress, and apopotosis.
    Journal of applied physiology (Bethesda, Md. : 1985), 2008, Volume: 105, Issue:2

    Topics: Androgen Antagonists; Animals; Apoptosis; Blotting, Western; Cell Separation; Chemokines; Cytokines;

2008