fluoxetine has been researched along with Subarachnoid Hemorrhage in 4 studies
Fluoxetine: The first highly specific serotonin uptake inhibitor. It is used as an antidepressant and often has a more acceptable side-effects profile than traditional antidepressants.
fluoxetine : A racemate comprising equimolar amounts of (R)- and (S)-fluoxetine. A selective serotonin reuptake inhibitor (SSRI), it is used (generally as the hydrochloride salt) for the treatment of depression (and the depressive phase of bipolar disorder), bullimia nervosa, and obsessive-compulsive disorder.
N-methyl-3-phenyl-3-[4-(trifluoromethyl)phenoxy]propan-1-amine : An aromatic ether consisting of 4-trifluoromethylphenol in which the hydrogen of the phenolic hydroxy group is replaced by a 3-(methylamino)-1-phenylpropyl group.
Subarachnoid Hemorrhage: Bleeding into the intracranial or spinal SUBARACHNOID SPACE, most resulting from INTRACRANIAL ANEURYSM rupture. It can occur after traumatic injuries (SUBARACHNOID HEMORRHAGE, TRAUMATIC). Clinical features include HEADACHE; NAUSEA; VOMITING, nuchal rigidity, variable neurological deficits and reduced mental status.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Fluoxetine, an anti-depressant drug, has recently been shown to provide neuroprotection in central nervous system injury, but its roles in subarachnoid hemorrhage (SAH) remain unclear." | 7.88 | Fluoxetine is Neuroprotective in Early Brain Injury via its Anti-inflammatory and Anti-apoptotic Effects in a Rat Experimental Subarachnoid Hemorrhage Model. ( Guo, YS; Hao, DJ; Hu, HM; Huang, DG; Hui, H; Li, B; Wang, B; Wang, XD; Zhang, HP, 2018) |
| "Fluoxetine administration attenuated BBB disruption, brain edema, and improved neurological function after SAH." | 7.88 | Fluoxetine attenuates neuroinflammation in early brain injury after subarachnoid hemorrhage: a possible role for the regulation of TLR4/MyD88/NF-κB signaling pathway. ( Cai, J; Chen, G; Chen, JS; Guan, GP; Li, JR; Liu, FY; Pan, HZ; Qian, C; Ruan, W; Wang, C; Wang, L, 2018) |
| "Fluoxetine is a well-studied serotonin selective reuptake inhibitor (SSRI)." | 5.72 | Fluoxetine attenuates apoptosis in early brain injury after subarachnoid hemorrhage through Notch1/ASK1/p38 MAPK signaling pathway. ( Li, C; Liu, M; Su, W; Zhong, W, 2022) |
| "Fluoxetine, an anti-depressant drug, has recently been shown to provide neuroprotection in central nervous system injury, but its roles in subarachnoid hemorrhage (SAH) remain unclear." | 3.88 | Fluoxetine is Neuroprotective in Early Brain Injury via its Anti-inflammatory and Anti-apoptotic Effects in a Rat Experimental Subarachnoid Hemorrhage Model. ( Guo, YS; Hao, DJ; Hu, HM; Huang, DG; Hui, H; Li, B; Wang, B; Wang, XD; Zhang, HP, 2018) |
| "Fluoxetine administration attenuated BBB disruption, brain edema, and improved neurological function after SAH." | 3.88 | Fluoxetine attenuates neuroinflammation in early brain injury after subarachnoid hemorrhage: a possible role for the regulation of TLR4/MyD88/NF-κB signaling pathway. ( Cai, J; Chen, G; Chen, JS; Guan, GP; Li, JR; Liu, FY; Pan, HZ; Qian, C; Ruan, W; Wang, C; Wang, L, 2018) |
| "Fluoxetine is a well-studied serotonin selective reuptake inhibitor (SSRI)." | 1.72 | Fluoxetine attenuates apoptosis in early brain injury after subarachnoid hemorrhage through Notch1/ASK1/p38 MAPK signaling pathway. ( Li, C; Liu, M; Su, W; Zhong, W, 2022) |
| "Fluoxetine was administered via intravenous route 6 h after SAH." | 1.46 | Fluoxetine-enhanced autophagy ameliorates early brain injury via inhibition of NLRP3 inflammasome activation following subrachnoid hemorrhage in rats. ( Chen, G; Chen, JS; Chen, JY; Fan, LF; Gu, C; Li, JR; Nie, S; Peng, YC; Wang, C; Wang, L; Wang, ZJ; Wu, C; Xu, HZ; Yan, F, 2017) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (75.00) | 24.3611 |
| 2020's | 1 (25.00) | 2.80 |
| Authors | Studies |
|---|---|
| Liu, M | 1 |
| Zhong, W | 1 |
| Li, C | 1 |
| Su, W | 1 |
| Li, JR | 2 |
| Xu, HZ | 1 |
| Nie, S | 1 |
| Peng, YC | 1 |
| Fan, LF | 1 |
| Wang, ZJ | 1 |
| Wu, C | 1 |
| Yan, F | 1 |
| Chen, JY | 1 |
| Gu, C | 1 |
| Wang, C | 2 |
| Chen, JS | 2 |
| Wang, L | 2 |
| Chen, G | 2 |
| Hu, HM | 1 |
| Li, B | 1 |
| Wang, XD | 1 |
| Guo, YS | 1 |
| Hui, H | 1 |
| Zhang, HP | 1 |
| Wang, B | 1 |
| Huang, DG | 1 |
| Hao, DJ | 1 |
| Liu, FY | 1 |
| Cai, J | 1 |
| Ruan, W | 1 |
| Guan, GP | 1 |
| Pan, HZ | 1 |
| Qian, C | 1 |
4 other studies available for fluoxetine and Subarachnoid Hemorrhage
| Article | Year |
|---|---|
Fluoxetine attenuates apoptosis in early brain injury after subarachnoid hemorrhage through Notch1/ASK1/p38 MAPK signaling pathway.
Topics: Animals; Apoptosis; Brain Injuries; Fluoxetine; Mice; Mice, Inbred C57BL; Neuroprotective Agents; p3 | 2022 |
Fluoxetine-enhanced autophagy ameliorates early brain injury via inhibition of NLRP3 inflammasome activation following subrachnoid hemorrhage in rats.
Topics: Animals; Anti-Inflammatory Agents; Autophagy; Brain Injuries; Fluoxetine; Inflammasomes; Male; NLR F | 2017 |
Fluoxetine is Neuroprotective in Early Brain Injury via its Anti-inflammatory and Anti-apoptotic Effects in a Rat Experimental Subarachnoid Hemorrhage Model.
Topics: Animals; Apoptosis; Blood-Brain Barrier; Brain Edema; Cytokines; Disease Models, Animal; Fluoxetine; | 2018 |
Fluoxetine attenuates neuroinflammation in early brain injury after subarachnoid hemorrhage: a possible role for the regulation of TLR4/MyD88/NF-κB signaling pathway.
Topics: Animals; Blood-Brain Barrier; Brain Edema; Brain Injuries; Calcium-Binding Proteins; Cytokines; Dise | 2018 |