fluoxetine has been researched along with Glial Cell Tumors in 8 studies
Fluoxetine: The first highly specific serotonin uptake inhibitor. It is used as an antidepressant and often has a more acceptable side-effects profile than traditional antidepressants.
fluoxetine : A racemate comprising equimolar amounts of (R)- and (S)-fluoxetine. A selective serotonin reuptake inhibitor (SSRI), it is used (generally as the hydrochloride salt) for the treatment of depression (and the depressive phase of bipolar disorder), bullimia nervosa, and obsessive-compulsive disorder.
N-methyl-3-phenyl-3-[4-(trifluoromethyl)phenoxy]propan-1-amine : An aromatic ether consisting of 4-trifluoromethylphenol in which the hydrogen of the phenolic hydroxy group is replaced by a 3-(methylamino)-1-phenylpropyl group.
| Excerpt | Relevance | Reference |
|---|---|---|
| " The purpose of this study was to evaluate the synergistic killing effect of fluoxetine (FL) with IR on glioma cancer cell (U-87 MG), as well as radioprotective effect of FL against cellular toxicity induced by IR on non-malignant human fibroblast cell (HFFF2)." | 7.96 | Fluoxetine as an antidepressant medicine improves the effects of ionizing radiation for the treatment of glioma. ( Asgarian-Omran, H; Farzipour, S; Ghasemi, A; Hassanzadeh, S; Hosseinimehr, SJ; Najafi, SH; Shafiee, F, 2020) |
| "Although temozolomide (TMZ) is the most effective chemotherapy agent for glioma, chemotherapy resistance has limited its clinical use." | 7.83 | Fluoxetine synergizes with temozolomide to induce the CHOP-dependent endoplasmic reticulum stress-related apoptosis pathway in glioma cells. ( Chen, MH; Chen, W; Ma, J; Sun, LL; Wang, DC; Wang, FZ; Wang, H; Wang, XD; Yang, YR, 2016) |
| "In C6 glioma cells, we studied acute administration of SSRI antidepressants - fluoxetine, sertraline and citalopram." | 7.78 | Effect of fluoxetine and adenosine receptor NECA agonist on G alpha q/11 protein of C6 glioma cells. ( Kováru, F; Kovárů, H; Lisá, V, 2012) |
| "We studied the effect of acute administration of fluoxetine or NECA agonist of adenosine receptor (GPCR) on C6 glioma cells and natural killer (NK) cell line, innate immunity." | 7.77 | Effect of fluoxetine or adenosine receptor NECA agonist on G-proteins of C6 glioma cells or NK immunocytes. ( Fiserová, A; Kovaru, F; Kovárů, H; Lisá, V; Ondráčkova, P, 2011) |
| " The purpose of this study was to evaluate the synergistic killing effect of fluoxetine (FL) with IR on glioma cancer cell (U-87 MG), as well as radioprotective effect of FL against cellular toxicity induced by IR on non-malignant human fibroblast cell (HFFF2)." | 3.96 | Fluoxetine as an antidepressant medicine improves the effects of ionizing radiation for the treatment of glioma. ( Asgarian-Omran, H; Farzipour, S; Ghasemi, A; Hassanzadeh, S; Hosseinimehr, SJ; Najafi, SH; Shafiee, F, 2020) |
| "Although temozolomide (TMZ) is the most effective chemotherapy agent for glioma, chemotherapy resistance has limited its clinical use." | 3.83 | Fluoxetine synergizes with temozolomide to induce the CHOP-dependent endoplasmic reticulum stress-related apoptosis pathway in glioma cells. ( Chen, MH; Chen, W; Ma, J; Sun, LL; Wang, DC; Wang, FZ; Wang, H; Wang, XD; Yang, YR, 2016) |
| "In C6 glioma cells, we studied acute administration of SSRI antidepressants - fluoxetine, sertraline and citalopram." | 3.78 | Effect of fluoxetine and adenosine receptor NECA agonist on G alpha q/11 protein of C6 glioma cells. ( Kováru, F; Kovárů, H; Lisá, V, 2012) |
| "We studied the effect of acute administration of fluoxetine or NECA agonist of adenosine receptor (GPCR) on C6 glioma cells and natural killer (NK) cell line, innate immunity." | 3.77 | Effect of fluoxetine or adenosine receptor NECA agonist on G-proteins of C6 glioma cells or NK immunocytes. ( Fiserová, A; Kovaru, F; Kovárů, H; Lisá, V; Ondráčkova, P, 2011) |
| "Previous studies have demonstrated that chronic treatment of C6 glioma cells with the antidepressants desipramine and fluoxetine increases the Triton X-100 solubility of the G protein Gsalpha (Toki et al." | 3.71 | Chronic treatment of C6 glioma cells with antidepressant drugs results in a redistribution of Gsalpha. ( Donati, RJ; Rasenick, MM; Thukral, C, 2001) |
| "Gel electrophoresis of DNA was used for estimation of DNA changes caused in C6 glioma cells by treatment with psychotropic drugs (imipramine, amitryptiline and fluoxetine)." | 3.69 | Estimation of apoptosis in C6 glioma cells treated with antidepressants. ( Kovárů, H; Lisá, V; Lukásová, E; Rittich, B; Spanová, A, 1997) |
| "Management and resolution of the hydrocephalus was temporally associated with an improvement in his OCD compulsion symptoms." | 1.43 | Pediatric Obsessive-Compulsive Disorder Exacerbation and Obstructive Hydrocephalus: A Case Report. ( Bleakley, C; Leung, A; Loh, A; Saran, K; Stewart, SE, 2016) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (12.50) | 18.2507 |
| 2000's | 1 (12.50) | 29.6817 |
| 2010's | 5 (62.50) | 24.3611 |
| 2020's | 1 (12.50) | 2.80 |
| Authors | Studies |
|---|---|
| Hosseinimehr, SJ | 1 |
| Najafi, SH | 1 |
| Shafiee, F | 1 |
| Hassanzadeh, S | 1 |
| Farzipour, S | 1 |
| Ghasemi, A | 1 |
| Asgarian-Omran, H | 1 |
| Ma, J | 1 |
| Yang, YR | 1 |
| Chen, W | 1 |
| Chen, MH | 1 |
| Wang, H | 1 |
| Wang, XD | 1 |
| Sun, LL | 1 |
| Wang, FZ | 1 |
| Wang, DC | 1 |
| Leung, A | 1 |
| Bleakley, C | 1 |
| Loh, A | 1 |
| Saran, K | 1 |
| Stewart, SE | 1 |
| Kovárů, H | 3 |
| Kovaru, F | 2 |
| Ondráčkova, P | 1 |
| Lisá, V | 3 |
| Fiserová, A | 1 |
| Malynn, S | 1 |
| Campos-Torres, A | 1 |
| Moynagh, P | 1 |
| Haase, J | 1 |
| Spanová, A | 1 |
| Lukásová, E | 1 |
| Rittich, B | 1 |
| Donati, RJ | 1 |
| Thukral, C | 1 |
| Rasenick, MM | 1 |
8 other studies available for fluoxetine and Glial Cell Tumors
| Article | Year |
|---|---|
Fluoxetine as an antidepressant medicine improves the effects of ionizing radiation for the treatment of glioma.
Topics: Antidepressive Agents, Second-Generation; Apoptosis; Fluoxetine; Glioma; Humans; Radiation, Ionizing | 2020 |
Fluoxetine synergizes with temozolomide to induce the CHOP-dependent endoplasmic reticulum stress-related apoptosis pathway in glioma cells.
Topics: Animals; Antineoplastic Agents; Apoptosis; Brain Neoplasms; Caspase 3; Cell Line, Tumor; Cell Prolif | 2016 |
Pediatric Obsessive-Compulsive Disorder Exacerbation and Obstructive Hydrocephalus: A Case Report.
Topics: Brain Neoplasms; Child; Fluoxetine; Glioma; Humans; Hydrocephalus; Male; Obsessive-Compulsive Disord | 2016 |
Effect of fluoxetine or adenosine receptor NECA agonist on G-proteins of C6 glioma cells or NK immunocytes.
Topics: Adenosine-5'-(N-ethylcarboxamide); Animals; Antidepressive Agents, Tricyclic; Brain Neoplasms; Cell | 2011 |
Effect of fluoxetine and adenosine receptor NECA agonist on G alpha q/11 protein of C6 glioma cells.
Topics: Adenosine-5'-(N-ethylcarboxamide); Animals; Brain Neoplasms; Cell Line, Tumor; Citalopram; Depressio | 2012 |
The pro-inflammatory cytokine TNF-α regulates the activity and expression of the serotonin transporter (SERT) in astrocytes.
Topics: Astrocytes; Cell Line, Tumor; Citalopram; Fluoxetine; Glioma; Humans; Imidazoles; p38 Mitogen-Activa | 2013 |
Estimation of apoptosis in C6 glioma cells treated with antidepressants.
Topics: Amitriptyline; Antidepressive Agents; Apoptosis; DNA Fragmentation; Fluoxetine; Glioma; Humans; Imip | 1997 |
Chronic treatment of C6 glioma cells with antidepressant drugs results in a redistribution of Gsalpha.
Topics: Animals; Antidepressive Agents; Chlorpromazine; Desipramine; Fluoxetine; Glioma; GTP-Binding Protein | 2001 |