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fluoxetine and Electron Transport Chain Deficiencies, Mitochondrial

fluoxetine has been researched along with Electron Transport Chain Deficiencies, Mitochondrial in 1 studies

Fluoxetine: The first highly specific serotonin uptake inhibitor. It is used as an antidepressant and often has a more acceptable side-effects profile than traditional antidepressants.
fluoxetine : A racemate comprising equimolar amounts of (R)- and (S)-fluoxetine. A selective serotonin reuptake inhibitor (SSRI), it is used (generally as the hydrochloride salt) for the treatment of depression (and the depressive phase of bipolar disorder), bullimia nervosa, and obsessive-compulsive disorder.
N-methyl-3-phenyl-3-[4-(trifluoromethyl)phenoxy]propan-1-amine : An aromatic ether consisting of 4-trifluoromethylphenol in which the hydrogen of the phenolic hydroxy group is replaced by a 3-(methylamino)-1-phenylpropyl group.

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Wen, L1
Jin, Y1
Li, L1
Sun, S1
Cheng, S1
Zhang, S1
Zhang, Y1
Svenningsson, P1

Other Studies

1 other study available for fluoxetine and Electron Transport Chain Deficiencies, Mitochondrial

ArticleYear
Exercise prevents raphe nucleus mitochondrial overactivity in a rat depression model.
    Physiology & behavior, 2014, Jun-10, Volume: 132

    Topics: Adenosine Triphosphate; Analysis of Variance; Animals; Antidepressive Agents, Second-Generation; Bra

2014