fluoxetine has been researched along with Brain Edema in 3 studies
Fluoxetine: The first highly specific serotonin uptake inhibitor. It is used as an antidepressant and often has a more acceptable side-effects profile than traditional antidepressants.
fluoxetine : A racemate comprising equimolar amounts of (R)- and (S)-fluoxetine. A selective serotonin reuptake inhibitor (SSRI), it is used (generally as the hydrochloride salt) for the treatment of depression (and the depressive phase of bipolar disorder), bullimia nervosa, and obsessive-compulsive disorder.
N-methyl-3-phenyl-3-[4-(trifluoromethyl)phenoxy]propan-1-amine : An aromatic ether consisting of 4-trifluoromethylphenol in which the hydrogen of the phenolic hydroxy group is replaced by a 3-(methylamino)-1-phenylpropyl group.
Brain Edema: Increased intracellular or extracellular fluid in brain tissue. Cytotoxic brain edema (swelling due to increased intracellular fluid) is indicative of a disturbance in cell metabolism, and is commonly associated with hypoxic or ischemic injuries (see HYPOXIA, BRAIN). An increase in extracellular fluid may be caused by increased brain capillary permeability (vasogenic edema), an osmotic gradient, local blockages in interstitial fluid pathways, or by obstruction of CSF flow (e.g., obstructive HYDROCEPHALUS). (From Childs Nerv Syst 1992 Sep; 8(6):301-6)
Excerpt | Relevance | Reference |
---|---|---|
"Fluoxetine, an anti-depressant drug, has recently been shown to provide neuroprotection in central nervous system injury, but its roles in subarachnoid hemorrhage (SAH) remain unclear." | 7.88 | Fluoxetine is Neuroprotective in Early Brain Injury via its Anti-inflammatory and Anti-apoptotic Effects in a Rat Experimental Subarachnoid Hemorrhage Model. ( Guo, YS; Hao, DJ; Hu, HM; Huang, DG; Hui, H; Li, B; Wang, B; Wang, XD; Zhang, HP, 2018) |
"Fluoxetine administration attenuated BBB disruption, brain edema, and improved neurological function after SAH." | 7.88 | Fluoxetine attenuates neuroinflammation in early brain injury after subarachnoid hemorrhage: a possible role for the regulation of TLR4/MyD88/NF-κB signaling pathway. ( Cai, J; Chen, G; Chen, JS; Guan, GP; Li, JR; Liu, FY; Pan, HZ; Qian, C; Ruan, W; Wang, C; Wang, L, 2018) |
"Fluoxetine, an anti-depressant drug, has recently been shown to provide neuroprotection in central nervous system injury, but its roles in subarachnoid hemorrhage (SAH) remain unclear." | 3.88 | Fluoxetine is Neuroprotective in Early Brain Injury via its Anti-inflammatory and Anti-apoptotic Effects in a Rat Experimental Subarachnoid Hemorrhage Model. ( Guo, YS; Hao, DJ; Hu, HM; Huang, DG; Hui, H; Li, B; Wang, B; Wang, XD; Zhang, HP, 2018) |
"Fluoxetine administration attenuated BBB disruption, brain edema, and improved neurological function after SAH." | 3.88 | Fluoxetine attenuates neuroinflammation in early brain injury after subarachnoid hemorrhage: a possible role for the regulation of TLR4/MyD88/NF-κB signaling pathway. ( Cai, J; Chen, G; Chen, JS; Guan, GP; Li, JR; Liu, FY; Pan, HZ; Qian, C; Ruan, W; Wang, C; Wang, L, 2018) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (33.33) | 29.6817 |
2010's | 2 (66.67) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Hu, HM | 1 |
Li, B | 1 |
Wang, XD | 1 |
Guo, YS | 1 |
Hui, H | 1 |
Zhang, HP | 1 |
Wang, B | 1 |
Huang, DG | 1 |
Hao, DJ | 1 |
Liu, FY | 1 |
Cai, J | 1 |
Wang, C | 1 |
Ruan, W | 1 |
Guan, GP | 1 |
Pan, HZ | 1 |
Li, JR | 1 |
Qian, C | 1 |
Chen, JS | 1 |
Wang, L | 1 |
Chen, G | 1 |
Michalak, A | 1 |
Rose, C | 1 |
Butterworth, RF | 1 |
3 other studies available for fluoxetine and Brain Edema
Article | Year |
---|---|
Fluoxetine is Neuroprotective in Early Brain Injury via its Anti-inflammatory and Anti-apoptotic Effects in a Rat Experimental Subarachnoid Hemorrhage Model.
Topics: Animals; Apoptosis; Blood-Brain Barrier; Brain Edema; Cytokines; Disease Models, Animal; Fluoxetine; | 2018 |
Fluoxetine attenuates neuroinflammation in early brain injury after subarachnoid hemorrhage: a possible role for the regulation of TLR4/MyD88/NF-κB signaling pathway.
Topics: Animals; Blood-Brain Barrier; Brain Edema; Brain Injuries; Calcium-Binding Proteins; Cytokines; Dise | 2018 |
Loss of noradrenaline transporter sites in frontal cortex of rats with acute (ischemic) liver failure.
Topics: Acute Disease; Ammonia; Animals; Brain Edema; Carrier Proteins; Fluoxetine; Frontal Lobe; Hepatic En | 2001 |