fluorexon has been researched along with Disease Exacerbation in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 2 (50.00) | 18.2507 |
2000's | 1 (25.00) | 29.6817 |
2010's | 1 (25.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Dyment, NA; Li, Q; Rowe, DW; Siu, SY; Sundberg, JP; Uitto, J | 1 |
Fan, YB; Li, HX; Liu, YZ; Wang, Z; Yi, ZC; Yin, Y; Zhuang, FY | 1 |
Autio, J; Hietala, EL; Larmas, M | 1 |
Goto, S; Kobayashi, Y; Moriya, H; Tanno, T; Yamazaki, M | 1 |
4 other study(ies) available for fluorexon and Disease Exacerbation
Article | Year |
---|---|
Variable patterns of ectopic mineralization in Enpp1asj-2J mice, a model for generalized arterial calcification of infancy.
Topics: Animals; Anthraquinones; Connective Tissue; Demeclocycline; Disease Progression; Fluoresceins; Fluorescent Dyes; Genetic Predisposition to Disease; Heterozygote; Homozygote; Mice, Inbred BALB C; Mice, Mutant Strains; Microscopy, Fluorescence; Mutation; Phenotype; Phosphoric Diester Hydrolases; Pyrophosphatases; Time Factors; Vascular Calcification | 2016 |
Tellimagrandin I enhances gap junctional communication and attenuates the tumor phenotype of human cervical carcinoma HeLa cells in vitro.
Topics: Antineoplastic Agents; Connexin 43; Disease Progression; Dose-Response Relationship, Drug; Drugs, Chinese Herbal; Female; Fluoresceins; Gallic Acid; Gap Junctions; Gene Expression Regulation, Neoplastic; Glucosides; HeLa Cells; Humans; Hydrolyzable Tannins; In Vitro Techniques; Isoquinolines; Phenotype; Uterine Cervical Neoplasms | 2006 |
The effect of early weaning on dentin formation and dentinal caries in rats.
Topics: Analysis of Variance; Animals; Anti-Bacterial Agents; Coloring Agents; Dental Caries; Dental Enamel; Dentin; Dentinogenesis; Diet; Diet, Cariogenic; Dietary Sucrose; Disease Progression; Female; Fluoresceins; Fluorescent Dyes; Male; Molar; Oxytetracycline; Rats; Rats, Sprague-Dawley; Tooth Calcification; Weaning | 1997 |
Regional variations in the progression of bone loss in two different mouse osteopenia models.
Topics: Aging; Animals; Body Weight; Bone Development; Bone Diseases, Metabolic; Disease Progression; Female; Fluoresceins; Humerus; Mice; Mice, Inbred ICR; Mice, Mutant Strains; Osteoporosis; Parietal Bone; Spine; Tetracycline; Tibia | 1998 |