flumazenil has been researched along with Epidermolysis Bullosa, Junctional in 1 studies
Flumazenil: A potent benzodiazepine receptor antagonist. Since it reverses the sedative and other actions of benzodiazepines, it has been suggested as an antidote to benzodiazepine overdoses.
flumazenil : An organic heterotricyclic compound that is 5,6-dihydro-4H-imidazo[1,5-a][1,4]benzodiazepine which is substituted at positions 3, 5, 6, and 8 by ethoxycarbonyl, methyl, oxo, and fluoro groups, respectively. It is used as an antidote to benzodiazepine overdose.
Epidermolysis Bullosa, Junctional: Form of epidermolysis bullosa having onset at birth or during the neonatal period and transmitted through autosomal recessive inheritance. It is characterized by generalized blister formation, extensive denudation, and separation and cleavage of the basal cell plasma membranes from the basement membrane.
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 1 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Mayer, B | 1 |
Silló, P | 1 |
Mazán, M | 1 |
Pintér, D | 1 |
Medvecz, M | 1 |
Has, C | 1 |
Castiglia, D | 1 |
Petit, F | 1 |
Charlesworth, A | 1 |
Hatvani, Z | 1 |
Pamjav, H | 1 |
Kárpáti, S | 1 |
1 other study available for flumazenil and Epidermolysis Bullosa, Junctional
Article | Year |
---|---|
A unique LAMB3 splice-site mutation with founder effect from the Balkans causes lethal epidermolysis bullosa in several European countries.
Topics: Amino Acid Substitution; Cell Adhesion Molecules; DNA, Complementary; Emigration and Immigration; Ep | 2016 |