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flumazenil and Epidermolysis Bullosa, Junctional

flumazenil has been researched along with Epidermolysis Bullosa, Junctional in 1 studies

Flumazenil: A potent benzodiazepine receptor antagonist. Since it reverses the sedative and other actions of benzodiazepines, it has been suggested as an antidote to benzodiazepine overdoses.
flumazenil : An organic heterotricyclic compound that is 5,6-dihydro-4H-imidazo[1,5-a][1,4]benzodiazepine which is substituted at positions 3, 5, 6, and 8 by ethoxycarbonyl, methyl, oxo, and fluoro groups, respectively. It is used as an antidote to benzodiazepine overdose.

Epidermolysis Bullosa, Junctional: Form of epidermolysis bullosa having onset at birth or during the neonatal period and transmitted through autosomal recessive inheritance. It is characterized by generalized blister formation, extensive denudation, and separation and cleavage of the basal cell plasma membranes from the basement membrane.

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Mayer, B1
Silló, P1
Mazán, M1
Pintér, D1
Medvecz, M1
Has, C1
Castiglia, D1
Petit, F1
Charlesworth, A1
Hatvani, Z1
Pamjav, H1
Kárpáti, S1

Other Studies

1 other study available for flumazenil and Epidermolysis Bullosa, Junctional

ArticleYear
A unique LAMB3 splice-site mutation with founder effect from the Balkans causes lethal epidermolysis bullosa in several European countries.
    The British journal of dermatology, 2016, Volume: 175, Issue:4

    Topics: Amino Acid Substitution; Cell Adhesion Molecules; DNA, Complementary; Emigration and Immigration; Ep

2016