flufenamic acid has been researched along with Hepatoblastoma in 2 studies
Flufenamic Acid: An anthranilic acid derivative with analgesic, anti-inflammatory, and antipyretic properties. It is used in musculoskeletal and joint disorders and administered by mouth and topically. (From Martindale, The Extra Pharmacopoeia, 30th ed, p16)
flufenamic acid : An aromatic amino acid consisting of anthranilic acid carrying an N-(trifluoromethyl)phenyl substituent. An analgesic and anti-inflammatory, it is used in rheumatic disorders.
Hepatoblastoma: A malignant neoplasm occurring in young children, primarily in the liver, composed of tissue resembling embryonal or fetal hepatic epithelium, or mixed epithelial and mesenchymal tissues. (Stedman, 25th ed)
| Excerpt | Relevance | Reference |
|---|---|---|
| " In this study we investigated the induction of apoptosis by 4-aminopyridine (4-AP), a K(+) channel blocker, and its mechanism in HepG2 human hepatoblastoma cells." | 7.70 | Ca2+ influx mediates apoptosis induced by 4-aminopyridine, a K+ channel blocker, in HepG2 human hepatoblastoma cells. ( Jung, MW; Kang, GH; Kang, YS; Kim, JA; Lee, SH; Lee, YS, 2000) |
| "The signaling mechanism of tamoxifen (TAM)-induced apoptosis was investigated in HepG2 human hepatoblastoma cells which do not express the estrogen receptor (ER)." | 7.70 | Involvement of Ca2+ influx in the mechanism of tamoxifen-induced apoptosis in HepG2 human hepatoblastoma cells. ( Jung, MW; Kang, YS; Kim, JA; Lee, SH; Lee, YS, 1999) |
| " In this study we investigated the induction of apoptosis by 4-aminopyridine (4-AP), a K(+) channel blocker, and its mechanism in HepG2 human hepatoblastoma cells." | 3.70 | Ca2+ influx mediates apoptosis induced by 4-aminopyridine, a K+ channel blocker, in HepG2 human hepatoblastoma cells. ( Jung, MW; Kang, GH; Kang, YS; Kim, JA; Lee, SH; Lee, YS, 2000) |
| "The signaling mechanism of tamoxifen (TAM)-induced apoptosis was investigated in HepG2 human hepatoblastoma cells which do not express the estrogen receptor (ER)." | 3.70 | Involvement of Ca2+ influx in the mechanism of tamoxifen-induced apoptosis in HepG2 human hepatoblastoma cells. ( Jung, MW; Kang, YS; Kim, JA; Lee, SH; Lee, YS, 1999) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (50.00) | 18.2507 |
| 2000's | 1 (50.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Kim, JA | 2 |
| Kang, YS | 2 |
| Jung, MW | 2 |
| Kang, GH | 1 |
| Lee, SH | 2 |
| Lee, YS | 2 |
2 other studies available for flufenamic acid and Hepatoblastoma
| Article | Year |
|---|---|
Ca2+ influx mediates apoptosis induced by 4-aminopyridine, a K+ channel blocker, in HepG2 human hepatoblastoma cells.
Topics: 4-Aminopyridine; Apoptosis; Calcium; Calcium Channel Blockers; Cell Survival; Chelating Agents; DNA | 2000 |
Involvement of Ca2+ influx in the mechanism of tamoxifen-induced apoptosis in HepG2 human hepatoblastoma cells.
Topics: Apoptosis; Calcium; Calcium Channel Blockers; Cell Survival; Chelating Agents; DNA Fragmentation; Do | 1999 |