floxacillin has been researched along with Genetic Predisposition in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (50.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Hripcsak, G; Overby, CL; Shen, Y | 1 |
Andrews, E; Daly, AK | 1 |
Daly, AK; Day, CP | 1 |
Goldstein, DB; Urban, TJ; Watkins, PB | 1 |
2 review(s) available for floxacillin and Genetic Predisposition
Article | Year |
---|---|
Flucloxacillin-induced liver injury.
Topics: Animals; Anti-Bacterial Agents; Bile Acids and Salts; Chemical and Drug Induced Liver Injury; Cholestasis; Europe; Floxacillin; Genetic Predisposition to Disease; Homeostasis; Humans; Liver Diseases | 2008 |
Genetic association studies in drug-induced liver injury.
Topics: Adverse Drug Reaction Reporting Systems; Amoxicillin-Potassium Clavulanate Combination; Anti-Bacterial Agents; Anti-Inflammatory Agents, Non-Steroidal; Antitubercular Agents; Arylamine N-Acetyltransferase; Chemical and Drug Induced Liver Injury; Diclofenac; Floxacillin; Genetic Association Studies; Genetic Predisposition to Disease; HLA-B Antigens; HLA-DR Antigens; HLA-DRB1 Chains; Humans; Phenotype; Polymorphism, Single Nucleotide; Risk Assessment; Risk Factors | 2009 |
2 other study(ies) available for floxacillin and Genetic Predisposition
Article | Year |
---|---|
Estimating heritability of drug-induced liver injury from common variants and implications for future study designs.
Topics: Amoxicillin-Potassium Clavulanate Combination; beta-Lactamase Inhibitors; Case-Control Studies; Chemical and Drug Induced Liver Injury; Chromosomes, Human, Pair 6; Floxacillin; Genetic Predisposition to Disease; Genome-Wide Association Study; Humans | 2014 |
Genetic basis of susceptibility to drug-induced liver injury: what have we learned and where do we go from here?
Topics: Arylamine N-Acetyltransferase; Chemical and Drug Induced Liver Injury; Floxacillin; Genetic Predisposition to Disease; Genome-Wide Association Study; Humans; Isoniazid; Polymorphism, Single Nucleotide; STAT4 Transcription Factor | 2012 |