fk 633 has been researched along with Deficiency of GP 2b 3a Complex in 1 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (100.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Arai, M; Honda, S; Kashiwagi, H; Kato, H; Kiyoi, T; Kosugi, S; Kurata, Y; Matsuzawa, Y; Tadokoro, S; Tomiyama, Y | 1 |
1 other study(ies) available for fk 633 and Deficiency of GP 2b 3a Complex
| Article | Year |
|---|---|
A naturally occurring Tyr143His alpha IIb mutation abolishes alpha IIb beta 3 function for soluble ligands but retains its ability for mediating cell adhesion and clot retraction: comparison with other mutations causing ligand-binding defects.
Topics: Adult; Allosteric Regulation; Amino Acid Substitution; Blood Platelets; Clot Retraction; Codon; Dipeptides; Female; Fibrinogen; Heterozygote; Humans; Ligands; Mutation, Missense; Platelet Adhesiveness; Platelet Glycoprotein GPIIb-IIIa Complex; Platelet Membrane Glycoprotein IIb; Point Mutation; Polymorphism, Restriction Fragment Length; Protein Binding; Protein Structure, Tertiary; Recombinant Fusion Proteins; RNA, Messenger; Solubility; Structure-Activity Relationship; Thrombasthenia; Transfection | 2003 |