ferrous chloride has been researched along with Parkinson Disease in 5 studies
*Parkinson Disease: A progressive, degenerative neurologic disease characterized by a TREMOR that is maximal at rest, retropulsion (i.e. a tendency to fall backwards), rigidity, stooped posture, slowness of voluntary movements, and a masklike facial expression. Pathologic features include loss of melanin containing neurons in the substantia nigra and other pigmented nuclei of the brainstem. LEWY BODIES are present in the substantia nigra and locus coeruleus but may also be found in a related condition (LEWY BODY DISEASE, DIFFUSE) characterized by dementia in combination with varying degrees of parkinsonism. (Adams et al., Principles of Neurology, 6th ed, p1059, pp1067-75) [MeSH]
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (20.00) | 18.2507 |
| 2000's | 3 (60.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 1 (20.00) | 2.80 |
| Authors | Studies |
|---|---|
| Casalis, L; De Cecco, E; Legname, G; Parisse, P; Perissinotto, F; Posocco, P; Rondelli, V; Scaini, D; Stani, C; Vaccari, L | 1 |
| Brundin, P; Dringen, R; Falsig, J; Leist, M; Lotharius, J; Payne, S; van Beek, J | 1 |
| Lev, N; Melamed, E; Offen, D | 1 |
| Krieglstein, K; Lingor, P; Unsicker, K | 1 |
| Choi, P; Chong, M; Cochran, E; Golts, N; Hardy, J; Lee, JM; Petrucelli, L; Snyder, H; Sparkman, D; Wolozin, B | 1 |
5 other study(ies) available for ferrous chloride and Parkinson Disease
| Article | Year |
|---|---|
Iron-mediated interaction of alpha synuclein with lipid raft model membranes.
Topics: alpha-Synuclein; Ferrous Compounds; Humans; Iron; Lipid Bilayers; Membrane Microdomains; Microscopy, Atomic Force; Mutagenesis, Site-Directed; Parkinson Disease; Protein Aggregates; Protein Binding; Spectroscopy, Fourier Transform Infrared | 2020 |
Progressive degeneration of human mesencephalic neuron-derived cells triggered by dopamine-dependent oxidative stress is dependent on the mixed-lineage kinase pathway.
Topics: Animals; Apoptosis; Carbazoles; Cell Line; Cells, Cultured; Cystine; Cytosol; Dopamine; Enzyme Activation; Ferrous Compounds; Glutathione; Humans; Hydrogen Peroxide; Indoles; JNK Mitogen-Activated Protein Kinases; Lipid Peroxidation; MAP Kinase Kinase Kinases; MAP Kinase Signaling System; Mesencephalon; Methamphetamine; Nerve Degeneration; Neurites; Neurons; Oxidation-Reduction; Oxidative Stress; Parkinson Disease; Protein Kinase Inhibitors; Proto-Oncogene Proteins c-akt; Rats; Superoxides | 2005 |
Proteasomal inhibition hypersensitizes differentiated neuroblastoma cells to oxidative damage.
Topics: alpha-Synuclein; Benzothiazoles; Cell Differentiation; Cell Line, Tumor; Cytoplasm; Ferrous Compounds; Humans; Immunohistochemistry; Molsidomine; Mutation; Neuroblastoma; Oxidative Stress; Parkinson Disease; Proteasome Inhibitors; Reactive Oxygen Species; Rotenone; Thiazoles; Ubiquitin | 2006 |
Midbrain dopaminergic neurons are protected from radical induced damage by GDF-5 application. Short communication.
Topics: Animals; Apoptosis; Bone Morphogenetic Proteins; Cell Survival; Cells, Cultured; Dopamine; Dose-Response Relationship, Drug; Ferrous Compounds; Free Radicals; Growth Differentiation Factor 5; Growth Substances; Lethal Dose 50; Mesencephalon; Neurons; Nitric Oxide Donors; Nitroprusside; Parkinson Disease; Rats; Rats, Wistar; Tyrosine 3-Monooxygenase | 1999 |
Co-association of parkin and alpha-synuclein.
Topics: Aged; alpha-Synuclein; Antibodies; Axons; Binding Sites; Brain; Dopamine; Ferrous Compounds; Humans; Immunohistochemistry; Lewy Bodies; Ligases; Nerve Tissue Proteins; Oxidative Stress; Parkinson Disease; Precipitin Tests; Synucleins; Tumor Cells, Cultured; Ubiquitin-Protein Ligases | 2001 |