farnesol has been researched along with Prostatic Neoplasms in 7 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 2 (28.57) | 18.2507 |
2000's | 2 (28.57) | 29.6817 |
2010's | 3 (42.86) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Lang, L; Shay, C; Teng, Y; Zhao, X | 1 |
Fernandes, NV; Jones, S; Katuru, R; Mo, H; Qu, H; Yeganehjoo, H; Yu, Z | 1 |
Engelmann, U; Epplen, R; Heidenreich, A; Ohlmann, CH; Stöckle, M | 1 |
Conaway, MC; Gregory, CW; McPherson, RA; Santen, RJ; Yue, W | 1 |
Blum, R; Erlich, S; Karunagaran, D; Kloog, Y; Liebling, R; Pinkas-Kramarski, R; Tal-Or, P | 1 |
Foster, BA; Ghosh, PM; Ghosh-Choudhury, N; Greenberg, NM; Kreisberg, JI; Mott, GE; Moyer, ML; Thomas, CA | 1 |
Fawcett, TW; Gorospe, M; Holbrook, NJ; Hudgins, WR; Shack, S | 1 |
7 other study(ies) available for farnesol and Prostatic Neoplasms
Article | Year |
---|---|
Combined targeting of Arf1 and Ras potentiates anticancer activity for prostate cancer therapeutics.
Topics: ADP-Ribosylation Factor 1; Animals; Antineoplastic Agents; Apoptosis; Benzaldehydes; Cell Line, Tumor; Cell Movement; Cell Proliferation; Drug Synergism; Farnesol; Humans; Male; Mice; Prostatic Neoplasms; Pyrimidines; ras Proteins; Salicylates; Signal Transduction; Xenograft Model Antitumor Assays | 2017 |
β-ionone induces cell cycle arrest and apoptosis in human prostate tumor cells.
Topics: Adenocarcinoma; Antineoplastic Agents; Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Carcinoma; Cell Cycle Checkpoints; Cell Line, Tumor; Dose-Response Relationship, Drug; Farnesol; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Inhibitory Concentration 50; Male; Norisoprenoids; Prostatic Neoplasms | 2013 |
Differential effects of ibandronate, docetaxel and farnesol treatment alone and in combination on the growth of prostate cancer cell lines.
Topics: Antineoplastic Agents; Antineoplastic Combined Chemotherapy Protocols; Cell Line, Tumor; Cell Survival; Diphosphonates; Docetaxel; Drug Synergism; Farnesol; Humans; Ibandronic Acid; Male; Mevalonic Acid; Prenylation; Prostatic Neoplasms; Signal Transduction; Taxoids | 2011 |
The novel Ras antagonist, farnesylthiosalicylate, suppresses growth of prostate cancer in vitro.
Topics: Antineoplastic Agents; Cell Division; Cell Line, Tumor; Culture Media; Dihydrotestosterone; Farnesol; Humans; Male; Mitogen-Activated Protein Kinases; Phosphatidylinositol 3-Kinases; Prostatic Neoplasms; Salicylates | 2004 |
Ras inhibition results in growth arrest and death of androgen-dependent and androgen-independent prostate cancer cells.
Topics: Amino Acid Chloromethyl Ketones; Androgens; Antineoplastic Agents; Apoptosis; Cell Cycle; Cell Cycle Proteins; Cell Line, Tumor; Cell Proliferation; Cell Survival; Cyclin D1; Dose-Response Relationship, Drug; Down-Regulation; E2F1 Transcription Factor; Farnesol; Humans; Male; Myotonin-Protein Kinase; Prostatic Neoplasms; Protein-Tyrosine Kinases; Proto-Oncogene Proteins c-akt; ras Proteins; Salicylates; Serine Proteinase Inhibitors; Signal Transduction | 2006 |
Role of RhoA activation in the growth and morphology of a murine prostate tumor cell line.
Topics: Actin Cytoskeleton; Adenocarcinoma; Alkyl and Aryl Transferases; Animals; Antineoplastic Agents; Apoptosis; Cell Adhesion; Cell Division; Cell Size; Diterpenes; Drug Interactions; Enzyme Activation; Farnesol; G1 Phase; Genes, ras; GTP-Binding Proteins; Guanosine Triphosphate; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Lovastatin; Male; Mevalonic Acid; Mice; Mice, Transgenic; Polyisoprenyl Phosphates; Prostatic Neoplasms; Protein Prenylation; Protein Processing, Post-Translational; Proto-Oncogene Proteins p21(ras); rac GTP-Binding Proteins; rhoA GTP-Binding Protein; Sesquiterpenes; Tumor Cells, Cultured | 1999 |
Activation of the cholesterol pathway and Ras maturation in response to stress.
Topics: Adenocarcinoma; Calcium-Calmodulin-Dependent Protein Kinases; Cholesterol; Diterpenes; Farnesol; Genes, ras; Heat-Shock Response; Humans; Hydroxymethylglutaryl CoA Reductases; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Hydroxymethylglutaryl-CoA-Reductases, NADP-dependent; Lovastatin; Male; Mevalonic Acid; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinases; Prostatic Neoplasms; Protein Prenylation; ras Proteins; Sterols; Stress, Physiological | 1999 |