Page last updated: 2024-09-05

f 1394 and Disease Models, Animal

f 1394 has been researched along with Disease Models, Animal in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	1 (50.00)	29.6817
2010's	1 (50.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Bander, I; Blachford, C; Davis, M; Dehn, S; Feig, JE; Fisher, EA; Kusunoki, J; Mayne, J; Miller, C; Rong, JX; Rudel, LL; Tabas, I; Taubman, MB; Thorp, E; Wilson, M	1
Aragane, K; Chiwata, T; Fujinami, K; Kano, K; Kitamine, T; Kojima, K; Kozono, H; Kusunoki, J; Sekine, Y	1

Other Studies

2 other study(ies) available for f 1394 and Disease Models, Animal

Article	Year
ACAT inhibition reduces the progression of preexisting, advanced atherosclerotic mouse lesions without plaque or systemic toxicity. Arteriosclerosis, thrombosis, and vascular biology, 2013, Volume: 33, Issue:1 Topics: Acetyl-CoA C-Acyltransferase; Animals; Aorta; Aortic Diseases; Apolipoproteins E; Apoptosis; Atherosclerosis; Cholesterol; Cyclohexanes; Diet, Atherogenic; Dioxanes; Disease Models, Animal; Disease Progression; Enzyme Inhibitors; Foam Cells; Male; Mice; Mice, Knockout; Necrosis; Plaque, Atherosclerotic; Thromboplastin	2013
Postprandial hyperlipidemia in streptozotocin-induced diabetic rats is due to abnormal increase in intestinal acyl coenzyme A:cholesterol acyltransferase activity. Arteriosclerosis, thrombosis, and vascular biology, 2000, Volume: 20, Issue:1 Topics: Animals; Cholesterol; Cyclohexanes; Diabetes Mellitus, Experimental; Dietary Fats; Dioxanes; Disease Models, Animal; Eating; Enzyme Inhibitors; Hyperlipidemias; Intestine, Small; Lymphatic System; Male; Rats; Rats, Sprague-Dawley; Risk Factors; Sterol O-Acyltransferase; Triglycerides	2000

Article

Year

ACAT inhibition reduces the progression of preexisting, advanced atherosclerotic mouse lesions without plaque or systemic toxicity.

Arteriosclerosis, thrombosis, and vascular biology, 2013, Volume: 33, Issue:1

Topics: Acetyl-CoA C-Acyltransferase; Animals; Aorta; Aortic Diseases; Apolipoproteins E; Apoptosis; Atherosclerosis; Cholesterol; Cyclohexanes; Diet, Atherogenic; Dioxanes; Disease Models, Animal; Disease Progression; Enzyme Inhibitors; Foam Cells; Male; Mice; Mice, Knockout; Necrosis; Plaque, Atherosclerotic; Thromboplastin

2013

Postprandial hyperlipidemia in streptozotocin-induced diabetic rats is due to abnormal increase in intestinal acyl coenzyme A:cholesterol acyltransferase activity.

Arteriosclerosis, thrombosis, and vascular biology, 2000, Volume: 20, Issue:1

Topics: Animals; Cholesterol; Cyclohexanes; Diabetes Mellitus, Experimental; Dietary Fats; Dioxanes; Disease Models, Animal; Eating; Enzyme Inhibitors; Hyperlipidemias; Intestine, Small; Lymphatic System; Male; Rats; Rats, Sprague-Dawley; Risk Factors; Sterol O-Acyltransferase; Triglycerides

2000