ezogabine and Benign Familial Infantile Convulsions
ezogabine has been researched along with Benign Familial Infantile Convulsions in 6 studies
Research
Studies (6)
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 4 (66.67) | 29.6817 |
| 2010's | 2 (33.33) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
Authors
| Authors | Studies |
|---|---|
| Ambrosino, P; Barrese, V; Cilio, MR; Miceli, F; Migliore, M; Soldovieri, MV; Taglialatela, M | 1 |
| Bock, M; Danker, T; De Jonghe, P; Guenther, E; Ilina, EI; Jezutkovic, N; Lerche, H; Löffler, H; Maljevic, S; Mandelstam, S; Orhan, G; Reichel, SN; Scheffer, IE; Schepers, D; Suls, A; Weckhuysen, S | 1 |
| Johnson, BN; Qiu, C; Tallent, MK | 1 |
| Martire, M; Miceli, F; Soldovieri, MV; Taglialatela, M | 1 |
| Lerche, H; Maljevic, S; Wuttke, TV | 1 |
| Cooper, EC | 1 |
Reviews
3 review(s) available for ezogabine and Benign Familial Infantile Convulsions
| Article | Year |
|---|---|
Molecular pharmacology and therapeutic potential of neuronal Kv7-modulating drugs.
Topics: Aminopyridines; Animals; Binding Sites; Carbamates; Epilepsy, Benign Neonatal; Hearing Loss; Humans; KCNQ Potassium Channels; KCNQ1 Potassium Channel; KCNQ2 Potassium Channel; KCNQ3 Potassium Channel; Phenylenediamines | 2008 |
Nervous system KV7 disorders: breakdown of a subthreshold brake.
Topics: Anticonvulsants; Carbamates; Epilepsy, Benign Neonatal; Humans; KCNQ Potassium Channels; Mutation; Nervous System Diseases; Peripheral Nervous System Diseases; Phenylenediamines | 2008 |
Potassium channels: how genetic studies of epileptic syndromes open paths to new therapeutic targets and drugs.
Topics: Animals; Anticonvulsants; Carbamates; Clinical Trials as Topic; Drug Design; Epilepsy; Epilepsy, Benign Neonatal; Humans; Mutation; Neurons; Phenylenediamines; Potassium Channels; Potassium Channels, Voltage-Gated; Receptors, Muscarinic; Synaptic Transmission | 2001 |
Other Studies
3 other study(ies) available for ezogabine and Benign Familial Infantile Convulsions
| Article | Year |
|---|---|
Genotype-phenotype correlations in neonatal epilepsies caused by mutations in the voltage sensor of K(v)7.2 potassium channel subunits.
Topics: Amino Acid Substitution; Animals; Anticonvulsants; Carbamates; CHO Cells; Cricetinae; Cricetulus; Epilepsy, Benign Neonatal; Genotype; Humans; KCNQ2 Potassium Channel; KCNQ3 Potassium Channel; Models, Molecular; Mutation, Missense; Phenotype; Phenylenediamines; Pyramidal Cells; Structural Homology, Protein | 2013 |
Dominant-negative effects of KCNQ2 mutations are associated with epileptic encephalopathy.
Topics: Animals; Carbamates; Epilepsy, Benign Neonatal; Genetic Predisposition to Disease; Humans; KCNQ2 Potassium Channel; Membrane Potentials; Mutation, Missense; Oocytes; Phenylenediamines; Potassium Channels, Voltage-Gated; Xenopus | 2014 |
K+ M-current regulates the transition to seizures in immature and adult hippocampus.
Topics: Action Potentials; Animals; Anticonvulsants; Carbamates; Disease Models, Animal; Epilepsy, Benign Neonatal; Hippocampus; Humans; Indoles; Male; Mutation; Phenylenediamines; Potassium Channel Blockers; Potassium Channels, Voltage-Gated; Pyridines; Rats; Rats, Sprague-Dawley; Receptors, N-Methyl-D-Aspartate; Seizures | 2007 |