Page last updated: 2024-08-23

etoposide and Progeria

etoposide has been researched along with Progeria in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	2 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Arnold, R; Djabali, K; Henriques, G; Liu, C	1
Hutchison, CJ; Lattanzi, G; Muter, J; Richards, SA; Ritchie, P	1

Other Studies

2 other study(ies) available for etoposide and Progeria

Article	Year
Inhibition of JAK-STAT Signaling with Baricitinib Reduces Inflammation and Improves Cellular Homeostasis in Progeria Cells. Cells, 2019, 10-18, Volume: 8, Issue:10 Topics: Adenosine Triphosphate; Alopecia; Arthritis; Autophagy; Azetidines; Blotting, Western; Cell Cycle; Cells, Cultured; Cellular Senescence; Child; Child, Preschool; Data Mining; Etoposide; Female; Humans; Immunohistochemistry; Inflammation; Janus Kinase 1; Janus Kinase 2; Lamin Type A; Lipodystrophy; Male; Mutation; Progeria; Purines; Pyrazoles; Reactive Oxygen Species; STAT1 Transcription Factor; STAT3 Transcription Factor; Sulfonamides; Vascular Diseases	2019
The accumulation of un-repairable DNA damage in laminopathy progeria fibroblasts is caused by ROS generation and is prevented by treatment with N-acetyl cysteine. Human molecular genetics, 2011, Oct-15, Volume: 20, Issue:20 Topics: Acetylcysteine; Age Factors; Aged, 80 and over; Antineoplastic Agents; Child; Contracture; DNA Breaks, Double-Stranded; DNA Damage; DNA Repair; Etoposide; Fibroblasts; Humans; Hydrogen Peroxide; Male; Oxidants; Progeria; Reactive Oxygen Species; Skin Abnormalities	2011

Article

Year

Inhibition of JAK-STAT Signaling with Baricitinib Reduces Inflammation and Improves Cellular Homeostasis in Progeria Cells.

Cells, 2019, 10-18, Volume: 8, Issue:10

Topics: Adenosine Triphosphate; Alopecia; Arthritis; Autophagy; Azetidines; Blotting, Western; Cell Cycle; Cells, Cultured; Cellular Senescence; Child; Child, Preschool; Data Mining; Etoposide; Female; Humans; Immunohistochemistry; Inflammation; Janus Kinase 1; Janus Kinase 2; Lamin Type A; Lipodystrophy; Male; Mutation; Progeria; Purines; Pyrazoles; Reactive Oxygen Species; STAT1 Transcription Factor; STAT3 Transcription Factor; Sulfonamides; Vascular Diseases

2019

The accumulation of un-repairable DNA damage in laminopathy progeria fibroblasts is caused by ROS generation and is prevented by treatment with N-acetyl cysteine.

Human molecular genetics, 2011, Oct-15, Volume: 20, Issue:20

Topics: Acetylcysteine; Age Factors; Aged, 80 and over; Antineoplastic Agents; Child; Contracture; DNA Breaks, Double-Stranded; DNA Damage; DNA Repair; Etoposide; Fibroblasts; Humans; Hydrogen Peroxide; Male; Oxidants; Progeria; Reactive Oxygen Species; Skin Abnormalities

2011