Compounds > etomoxir
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etomoxir and Benign Neoplasms, Brain

etomoxir has been researched along with Benign Neoplasms, Brain in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (66.67)24.3611
2020's1 (33.33)2.80

Authors

AuthorsStudies
Bianchetti, E; Bruce, JN; Canoll, P; Humala, N; Karpel-Massler, G; Kleiner, G; Mahajan, A; Mela, A; Nguyen, TTT; Quinzii, CM; Sanchez-Quintero, MJ; Shang, E; Shu, C; Siegelin, MD; Torrini, C; Westhoff, MA; Zhang, Y; Zhao, J1
Affleck, VS; Joshi, AR; Lin, H; Maxwell, R; Patel, S; Stoll, EA; Turnbull, DM; Wilson, I1
Croteau, NJ; Ferrick, DA; Pike, LS; Smift, AL; Wu, M1

Other Studies

3 other study(ies) available for etomoxir and Benign Neoplasms, Brain

ArticleYear
MET Inhibition Elicits PGC1α-Dependent Metabolic Reprogramming in Glioblastoma.
    Cancer research, 2020, 01-01, Volume: 80, Issue:1

    Topics: Animals; Antineoplastic Combined Chemotherapy Protocols; Brain Neoplasms; Carnitine; Cell Line, Tumor; Cell Proliferation; Cell Respiration; Crizotinib; Drug Synergism; Epoxy Compounds; Fatty Acids; Gene Expression Profiling; Glioblastoma; Glycolysis; Guanidines; Humans; Lactams, Macrocyclic; Metabolomics; Mice; Mitochondria; Mitochondrial Dynamics; Oxidative Phosphorylation; Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha; Proteomics; Proto-Oncogene Proteins c-met; Reactive Oxygen Species; Xenograft Model Antitumor Assays

2020
Fatty acid oxidation is required for the respiration and proliferation of malignant glioma cells.
    Neuro-oncology, 2017, Volume: 19, Issue:1

    Topics: Animals; Apoptosis; Brain Neoplasms; Cell Proliferation; Epoxy Compounds; Fatty Acids; Glioma; Glycolysis; Humans; Hypoglycemic Agents; Mice; Mice, Inbred C57BL; Neural Stem Cells; Oxidation-Reduction; Tumor Cells, Cultured; Xenograft Model Antitumor Assays

2017
Inhibition of fatty acid oxidation by etomoxir impairs NADPH production and increases reactive oxygen species resulting in ATP depletion and cell death in human glioblastoma cells.
    Biochimica et biophysica acta, 2011, Volume: 1807, Issue:6

    Topics: Adenosine Triphosphate; Brain Neoplasms; Cell Death; Down-Regulation; Drug Evaluation, Preclinical; Epoxy Compounds; Fatty Acids; Glioblastoma; Humans; Hypoglycemic Agents; Models, Biological; NADP; Oxidation-Reduction; Reactive Oxygen Species; Tumor Cells, Cultured; Up-Regulation

2011