ethinyl estradiol-norgestrel combination has been researched along with Insulin Resistance in 10 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 2 (20.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 7 (70.00) | 24.3611 |
2020's | 1 (10.00) | 2.80 |
Authors | Studies |
---|---|
Kim, IK; Olatunji, LA; Seok, YM; Usman, TO | 1 |
Azen, CG; Bergman, RN; Perlman, JA; Roy, S; Watanabe, RM | 1 |
Haymond, MW; Mauras, N; Rini, A; Welch, S | 1 |
Anoka, NA; Lawrence, OA; Olufunmilayo, OM; Olufunto, BO | 1 |
Adeyanju, OA; Areola, ED; Michael, OS; Olatunji, LA; Soladoye, AO | 1 |
Michael, OS; Olatunji, LA | 1 |
Adeyanju, OA; Olatunji, LA; Soetan, OA; Soladoye, AO | 1 |
Michael, OS; Olatunji, LA; Omolekulo, TE | 1 |
Olaniyi, KS; Olatunji, LA | 1 |
Beasley, A; Estes, C; Guerrero, J; Westhoff, C | 1 |
2 trial(s) available for ethinyl estradiol-norgestrel combination and Insulin Resistance
Article | Year |
---|---|
Defects in carbohydrate metabolism in oral contraceptive users without apparent metabolic risk factors.
Topics: Adolescent; Adult; B-Lymphocytes; Carbohydrate Metabolism; Contraceptives, Oral, Hormonal; Cross-Sectional Studies; Dose-Response Relationship, Drug; Ethinyl Estradiol; Ethinyl Estradiol-Norgestrel Combination; Female; Glucose Tolerance Test; Humans; Insulin Resistance; Metabolic Diseases; Norgestrel; Risk Factors | 1994 |
The effect of obesity and low-dose oral contraceptives on carbohydrate and lipid metabolism.
Topics: Adolescent; Adult; Biomarkers; Blood Glucose; Body Mass Index; Carbohydrate Metabolism; Cholesterol, HDL; Cholesterol, LDL; Contraceptives, Oral, Combined; Dose-Response Relationship, Drug; Double-Blind Method; Drug Combinations; Ethinyl Estradiol; Female; Humans; Insulin; Insulin Resistance; Levonorgestrel; Lipid Metabolism; Medication Adherence; Obesity; Patient Dropouts; Triglycerides; Young Adult | 2012 |
8 other study(ies) available for ethinyl estradiol-norgestrel combination and Insulin Resistance
Article | Year |
---|---|
Activation of cardiac renin-angiotensin system and plasminogen activator inhibitor-1 gene expressions in oral contraceptive-induced cardiometabolic disorder.
Topics: Aldosterone; Animals; Ataxia Telangiectasia Mutated Proteins; Cardiovascular Diseases; Contraceptives, Oral, Combined; Corticosterone; Dyslipidemias; Ethinyl Estradiol-Norgestrel Combination; Female; Glucose Intolerance; Heart; Hyperinsulinism; Hypertension; Insulin Resistance; Metabolic Syndrome; Myocardium; Peptidyl-Dipeptidase A; Plasminogen Activator Inhibitor 1; Random Allocation; Rats, Sprague-Dawley; Renin-Angiotensin System; Up-Regulation | 2017 |
Ovarian hyperandrogenism is associated with insulin resistance to both peripheral carbohydrate and whole-body protein metabolism in postpubertal young females: a metabolic study.
Topics: Adolescent; Androgens; Blood Glucose; Body Mass Index; Calorimetry, Indirect; Carbon Isotopes; Deuterium; Ethinyl Estradiol-Norgestrel Combination; Female; Human Growth Hormone; Humans; Hyperandrogenism; Insulin Resistance; Insulin-Like Growth Factor I; Kinetics; Lipid Metabolism; Obesity; Ovary; Oxidation-Reduction; Proteins | 1998 |
Insulin resistance and depressed cardiac G6PD activity induced by glucocorticoid exposure during pregnancy are attenuated by maternal estrogen-progestin therapy.
Topics: Animals; Contraceptives, Oral, Combined; Drug Combinations; Estrogens; Ethinyl Estradiol; Female; Glucocorticoids; Glucosephosphate Dehydrogenase; Insulin Resistance; Levonorgestrel; Maternal-Fetal Exchange; Myocardium; Pregnancy; Progestins; Rats, Wistar | 2020 |
Anti-inflammatory and antithrombotic effects of nicotine exposure in oral contraceptive-induced insulin resistance are glucocorticoid-independent.
Topics: Animals; Anti-Inflammatory Agents; Blood Viscosity; Contraceptives, Oral; Corticosterone; Dose-Response Relationship, Drug; Drug Combinations; Ethinyl Estradiol; Female; Fibrinolytic Agents; Glucocorticoids; Insulin Resistance; Levonorgestrel; Nicotine; Nicotinic Agonists; Rats; Rats, Sprague-Dawley | 2017 |
Ameliorative effect of nicotine exposure on insulin resistance is accompanied by decreased cardiac glycogen synthase kinase-3 and plasminogen activator inhibitor-1 during oral oestrogen-progestin therapy.
Topics: Administration, Oral; Aldosterone; Animals; Biomarkers; Cardiovascular Diseases; Contraceptives, Oral, Combined; Corticosterone; Dose-Response Relationship, Drug; Drug Combinations; Ethinyl Estradiol; Female; Glycogen Synthase Kinase 3; Heart; Insulin; Insulin Resistance; Insulin Secretion; Insulin-Secreting Cells; Levonorgestrel; Myocardium; Nicotine; Nicotinic Agonists; Plasminogen Activator Inhibitor 1; Prediabetic State; Random Allocation; Rats, Wistar | 2018 |
Oral hormonal therapy with ethinylestradiol-levonorgestrel improves insulin resistance, obesity, and glycogen synthase kinase-3 independent of circulating mineralocorticoid in estrogen-deficient rats.
Topics: Administration, Oral; Animals; Body Weight; Drug Combinations; Eating; Estradiol; Estrogens; Ethinyl Estradiol; Fasting; Female; Glycogen Synthase Kinase 3; Insulin Resistance; Insulin-Secreting Cells; Intra-Abdominal Fat; Levonorgestrel; Mineralocorticoids; Obesity; Rats; Rats, Wistar; Triglycerides | 2018 |
Sodium acetate improves disrupted glucoregulation and hepatic triglyceride content in insulin-resistant female rats: involvement of adenosine deaminase and dipeptidyl peptidase-4 activities.
Topics: Adenosine Deaminase; Animals; Dipeptidyl Peptidase 4; Drug Combinations; Ethinyl Estradiol; Female; Glucose; Insulin Resistance; Levonorgestrel; Liver; Rats, Wistar; Sodium Acetate; Triglycerides; Uric Acid; Xanthine Oxidase | 2019 |
Oral ethinylestradiol-levonorgestrel normalizes fructose-induced hepatic lipid accumulation and glycogen depletion in female rats.
Topics: Administration, Oral; Animals; Antioxidants; Blood Glucose; Dose-Response Relationship, Drug; Drug Combinations; Ethinyl Estradiol; Female; Fructose; Glucosephosphate Dehydrogenase; Glycogen; Hyperinsulinism; Insulin Resistance; Levonorgestrel; Lipid Metabolism; Lipid Peroxidation; Liver; Protein Kinases; Rats; Rats, Wistar; Uric Acid | 2019 |